K(ATP) activation prevents progression of cardiac hypertrophy to failure induced by pressure overload via protecting endothelial function.
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The novel ATP-sensitive potassium channel opener iptakalim prevents insulin resistance associated with hypertension via restoring endothelial function.Natakalim improves post-infarction left ventricular remodeling by restoring the coordinated balance between endothelial function and cardiac hypertrophy.Effects of traditional Chinese medicine Xin-Ji-Er-Kang formula on 2K1C hypertensive rats: role of oxidative stress and endothelial dysfunctionThe effects of 17-methoxyl-7-hydroxy-benzene-furanchalcone on the pressure overload-induced progression of cardiac hypertrophy to cardiac failure.ATP-Sensitive Potassium Channel Currents in Eccentrically Hypertrophied Cardiac Myocytes of Volume-Overloaded Rats.ATP-sensitive potassium (KATP) channel activation decreases intraocular pressure in the anterior chamber of the eyeSimvastatin modulates remodeling of Kv4.3 expression in rat hypertrophied cardiomyocytes.Effect of the angiotensin II receptor blocker valsartan on cardiac hypertrophy and myocardial histone deacetylase expression in rats with aortic constriction.How full is our antihypertensives pipeline?Mutational analysis of the Kir6.1 gene in Chinese hypertensive patients treated with the novel ATP-sensitive potassium channel opener iptakalim.ATP-sensitive potassium (K(ATP)) channel openers diazoxide and nicorandil lower intraocular pressure in vivoThe role of ATP-sensitive potassium channels in cellular function and protection in the cardiovascular systemCinaciguat prevents the development of pathologic hypertrophy in a rat model of left ventricular pressure overload.Pharmacological evidence: a new therapeutic approach to the treatment of chronic heart failure through SUR2B/Kir6.1 channel in endothelial cells.Silencing salusin-β attenuates cardiovascular remodeling and hypertension in spontaneously hypertensive rats.Chemerin/ChemR23 signaling axis is involved in the endothelial protection by K(ATP) channel opener iptakalim.Association of the antihypertensive response of iptakalim with KCNJ11 (Kir6.2 gene) polymorphisms in Chinese Han hypertensive patients.ATP sensitive potassium channel openers: A new class of ocular hypotensive agents.Epigenetic Therapy for the Treatment of Hypertension-Induced Cardiac Hypertrophy and Fibrosis.Analysis on mechanism of ATP-sensitive K+ channel opener natakalim improving congestive heart failure after myocardial infarctionIptakalim: A novel multi-utility potassium channel opener.Molecular and functional characterization of the endothelial ATP-sensitive potassium channel.Iptakalim ameliorates monocrotaline-induced pulmonary arterial hypertension in rats.Artificial microRNA interference targeting AT(1a) receptors in paraventricular nucleus attenuates hypertension in rats.Effects of oligomeric grape seed proanthocyanidins on heart, aorta, kidney in DOCA-salt mice: role of oxidative stress.Protective effects of tannic acid on pressure overload-induced cardiac hypertrophy and underlying mechanisms in rats.Iptakalim enhances adult mouse hippocampal neurogenesis via opening Kir6.1-composed K-ATP channels expressed in neural stem cells.In vivo and in vitro protective effects of pentamethylquercetin on cardiac hypertrophy.Protective effect of hydrogen-rich saline on pressure overload-induced cardiac hypertrophyin rats: possible role of JAK-STAT signaling.Single-dose pharmacokinetics and safety of iptakalim hydrochloride in Chinese healthy volunteers.The CD147/MMP-2 signaling pathway may regulate early stage cardiac remodelling in spontaneously hypertensive rats.Serum Amyloid P-Component Prevents Cardiac Remodeling in Hypertensive Heart Disease.Grape seed proanthocyanidins prevent DOCA-salt hypertension-induced renal injury and its mechanisms in rats.Oligomerized grape seed proanthocyanidins ameliorates isoproterenol-induced cardiac remodeling in rats: role of oxidative stress.
P2860
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P2860
K(ATP) activation prevents progression of cardiac hypertrophy to failure induced by pressure overload via protecting endothelial function.
description
2009 nî lūn-bûn
@nan
2009年の論文
@ja
2009年学术文章
@wuu
2009年学术文章
@zh
2009年学术文章
@zh-cn
2009年学术文章
@zh-hans
2009年学术文章
@zh-my
2009年学术文章
@zh-sg
2009年學術文章
@yue
2009年學術文章
@zh-hant
name
K(ATP) activation prevents pro ...... otecting endothelial function.
@en
type
label
K(ATP) activation prevents pro ...... otecting endothelial function.
@en
prefLabel
K(ATP) activation prevents pro ...... otecting endothelial function.
@en
P2093
P2860
P356
P1476
K(ATP) activation prevents pro ...... otecting endothelial function.
@en
P2093
Chao-Liang Long
Ru-Huan Wang
P2860
P304
P356
10.1093/CVR/CVP099
P577
2009-03-20T00:00:00Z