Chloride transporter KCC2-dependent neuroprotection depends on the N-terminal protein domain.
about
Prenatal Hypoxia-Ischemia Induces Abnormalities in CA3 Microstructure, Potassium Chloride Co-Transporter 2 Expression and Inhibitory Tone.Glycine transporter 1 is a target for the treatment of epilepsy.Imaging and serum biomarkers reflecting the functional efficacy of extended erythropoietin treatment in rats following infantile traumatic brain injury.A Novel RNA Editing Sensor Tool and a Specific Agonist Determine Neuronal Protein Expression of RNA-Edited Glycine Receptors and Identify a Genomic APOBEC1 Dimorphism as a New Genetic Risk Factor of Epilepsy.Dose-dependent reversal of KCC2 hypofunction and phenobarbital-resistant neonatal seizures by ANA12
P2860
Chloride transporter KCC2-dependent neuroprotection depends on the N-terminal protein domain.
description
2015 nî lūn-bûn
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2015年の論文
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2015年学术文章
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2015年学术文章
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2015年学术文章
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2015年学术文章
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name
Chloride transporter KCC2-depe ...... the N-terminal protein domain.
@en
Chloride transporter KCC2-depe ...... the N-terminal protein domain.
@nl
type
label
Chloride transporter KCC2-depe ...... the N-terminal protein domain.
@en
Chloride transporter KCC2-depe ...... the N-terminal protein domain.
@nl
prefLabel
Chloride transporter KCC2-depe ...... the N-terminal protein domain.
@en
Chloride transporter KCC2-depe ...... the N-terminal protein domain.
@nl
P2860
P356
P1476
Chloride transporter KCC2-depe ...... the N-terminal protein domain
@en
P2093
A Winkelmann
P2860
P356
10.1038/CDDIS.2015.127
P577
2015-06-04T00:00:00Z