Inhibition of 53BP1 favors homology-dependent DNA repair and increases CRISPR-Cas9 genome-editing efficiency.

about

Clustered regularly interspaced short palindromic repeats (CRISPR)/CRISPR-associated protein 9 with improved proof-reading enhances homology-directed repair.High-throughput antibody engineering in mammalian cells by CRISPR/Cas9-mediated homology-directed mutagenesis

P2860

Q55449385-AC0DD5BC-9CF8-401E-B7F9-201D3924AC51 Q56888745-F6B844F9-565E-45A6-92C6-F81B2424E0AE

P2860

Inhibition of 53BP1 favors homology-dependent DNA repair and increases CRISPR-Cas9 genome-editing efficiency.

Item

http://www.wikidata.org/entity/Q46257646

description

2017 nî lūn-bûn

@nan

2017年の論文

@ja

2017年学术文章

@wuu

2017年学术文章

@zh

2017年学术文章

@zh-cn

2017年学术文章

@zh-hans

2017年学术文章

@zh-my

2017年学术文章

@zh-sg

2017年學術文章

@yue

2017年學術文章

@zh-hant

name

Inhibition of 53BP1 favors hom ...... as9 genome-editing efficiency.

@en

Inhibition of 53BP1 favors hom ...... as9 genome-editing efficiency.

@nl

type

Item

label

Inhibition of 53BP1 favors hom ...... as9 genome-editing efficiency.

@en

Inhibition of 53BP1 favors hom ...... as9 genome-editing efficiency.

@nl

prefLabel

Inhibition of 53BP1 favors hom ...... as9 genome-editing efficiency.

@en

Inhibition of 53BP1 favors hom ...... as9 genome-editing efficiency.

@nl

P1476

Inhibition of 53BP1 favors hom ...... as9 genome-editing efficiency.

@en

P2093

Alexandre Orthwein

http://www.w3.org/2001/XMLSchema#string

Amélie Fradet-Turcotte

http://www.w3.org/2001/XMLSchema#string

Andreas Ernst

http://www.w3.org/2001/XMLSchema#string

Andrew Vorobyov

http://www.w3.org/2001/XMLSchema#string

Danielle Krasner

http://www.w3.org/2001/XMLSchema#string

Eduardo Seclen

http://www.w3.org/2001/XMLSchema#string

Frank Sicheri

http://www.w3.org/2001/XMLSchema#string

Leo C K Wan

http://www.w3.org/2001/XMLSchema#string

Marella D Canny

http://www.w3.org/2001/XMLSchema#string

Meagan Munro

http://www.w3.org/2001/XMLSchema#string

P2860

Alternative-NHEJ is a mechanistically distinct pathway of mammalian chromosome break repair

The MMS22L-TONSL complex mediates recovery from replication stress and homologous recombination

Structural basis for the methylation state-specific recognition of histone H4-K20 by 53BP1 and Crb2 in DNA repair

Origins of Programmable Nucleases for Genome Engineering

A Strategy for Modulation of Enzymes in the Ubiquitin System

A cell cycle-dependent regulatory circuit composed of 53BP1-RIF1 and BRCA1-CtIP controls DNA repair pathway choice.

Noncanonical views of homology-directed DNA repair

Genome editing at the crossroads of delivery, specificity, and fidelity

Microhomology-Mediated End Joining: A Back-up Survival Mechanism or Dedicated Pathway?

Therapeutic genome editing: prospects and challenges

P2888

nbt.4021

P304

95-102

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1038/NBT.4021

http://www.w3.org/2001/XMLSchema#string

P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P50

P577

2017-11-27T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

29176614

http://www.w3.org/2001/XMLSchema#string

P921

CRISPR

P932

5762392

http://www.w3.org/2001/XMLSchema#string

Inhibition of 53BP1 favors homology-dependent DNA repair and increases CRISPR-Cas9 genome-editing efficiency.

about

P2860

P2860

Inhibition of 53BP1 favors homology-dependent DNA repair and increases CRISPR-Cas9 genome-editing efficiency.

description

name

type

label

prefLabel

P1433

P1476

P2093

P2860

P2888

P304

P31

P356

P433

P478

P50

P577

P698

P921

P932

P356

P1433

P1476

P2093

P2860

P2888

P304

P31

P356

P433

P478

P50

P577

P698

P921

P932