Impaired Cl- extrusion in layer V pyramidal neurons of chronically injured epileptogenic neocortex.
about
Raising cytosolic Cl- in cerebellar granule cells affects their excitability and vestibulo-ocular learning.NMDA receptor activity downregulates KCC2 resulting in depolarizing GABAA receptor-mediated currentsModulation of neuronal activity by phosphorylation of the K-Cl cotransporter KCC2Adult cortical plasticity following injury: Recapitulation of critical period mechanisms?Novel determinants of the neuronal Cl(-) concentrationChronic Posttraumatic Epilepsy following Neocortical Undercut Lesion in MiceThe RCC1 domain of protein associated with Myc (PAM) interacts with and regulates KCC2Disrupted dentate granule cell chloride regulation enhances synaptic excitability during development of temporal lobe epilepsy.GABA(A) receptor-mediated excitation in dissociated neurons from human hypothalamic hamartomasExcitatory and inhibitory synaptic connectivity to layer V fast-spiking interneurons in the freeze lesion model of cortical microgyria.Slow GABA(A) mediated synaptic transmission in rat visual cortexReorganization of inhibitory synaptic circuits in rodent chronically injured epileptogenic neocortex.Potassium dynamics in the epileptic cortex: new insights on an old topic.Chloride regulation in the pain pathwayPresynaptic inhibitory terminals are functionally abnormal in a rat model of posttraumatic epilepsyBumetanide, an NKCC1 antagonist, does not prevent formation of epileptogenic focus but blocks epileptic focus seizures in immature rat hippocampus.Neuronal chloride accumulation and excitatory GABA underlie aggravation of neonatal epileptiform activities by phenobarbital.Increased excitatory synaptic input to granule cells from hilar and CA3 regions in a rat model of temporal lobe epilepsyComputational modeling reveals dendritic origins of GABA(A)-mediated excitation in CA1 pyramidal neurons.A C-terminal domain in KCC2 confers constitutive K+-Cl- cotransport.Ovariectomy modulation of morphine analgesia of neuropathic pain is associated with the change of K(+)-Cl(-) cotransporter 2 protein level in spinal dorsal horn.Progressive NKCC1-dependent neuronal chloride accumulation during neonatal seizures.Cation-chloride cotransporters in neuronal development, plasticity and diseaseAlcohol abuse promotes changes in non-synaptic epileptiform activity with concomitant expression changes in cotransporters and glial cells.Altered GABA signaling in early life epilepsies.Loss of cation-chloride cotransporter expression in preterm infants with white matter lesions: implications for the pathogenesis of epilepsyBrain injury impairs dentate gyrus inhibitory efficacyAcute and chronic efficacy of bumetanide in an in vitro model of posttraumatic epileptogenesis.Focal cortical lesions induce bidirectional changes in the excitability of fast spiking and non fast spiking cortical interneuronsNeuronal plasticity and thalamocortical sleep and waking oscillationsInterneuronal calcium channel abnormalities in posttraumatic epileptogenic neocortex.Functional alterations in GABAergic fast-spiking interneurons in chronically injured epileptogenic neocortex.Transition from Initial Hypoactivity to Hyperactivity in Cortical Layer V Pyramidal Neurons after Traumatic Brain Injury In Vivo.Developmental patterns in the regulation of chloride homeostasis and GABA(A) receptor signaling by seizures.Chloride accumulation drives volume dynamics underlying cell proliferation and migration.How do seizures stop?Epilepsy following cortical injury: cellular and molecular mechanisms as targets for potential prophylaxis.Compromising KCC2 transporter activity enhances the development of continuous seizure activity.Functional consequences of the disturbances in the GABA-mediated inhibition induced by injuries in the cerebral cortex.Cortical inhibition, pH and cell excitability in epilepsy: what are optimal targets for antiepileptic interventions?
P2860
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P2860
Impaired Cl- extrusion in layer V pyramidal neurons of chronically injured epileptogenic neocortex.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
@zh
2005年學術文章
@zh-hant
name
Impaired Cl- extrusion in laye ...... jured epileptogenic neocortex.
@en
Impaired Cl- extrusion in laye ...... jured epileptogenic neocortex.
@nl
type
label
Impaired Cl- extrusion in laye ...... jured epileptogenic neocortex.
@en
Impaired Cl- extrusion in laye ...... jured epileptogenic neocortex.
@nl
prefLabel
Impaired Cl- extrusion in laye ...... jured epileptogenic neocortex.
@en
Impaired Cl- extrusion in laye ...... jured epileptogenic neocortex.
@nl
P2860
P356
P1476
Impaired Cl- extrusion in laye ...... jured epileptogenic neocortex.
@en
P2093
David A Prince
Xiaoming Jin
P2860
P304
P356
10.1152/JN.00728.2004
P407
P577
2005-04-01T00:00:00Z