Thrombospondins 1 and 2 are necessary for synaptic plasticity and functional recovery after stroke. - Wikidata - wikimedia - TriplyDB

Thrombospondins 1 and 2 are necessary for synaptic plasticity and functional recovery after stroke.

Thrombospondins 1 and 2 are necessary for synaptic plasticity and functional recovery after stroke.

http://www.wikidata.org/entity/Q46438887

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Gabapentin receptor alpha2delta-1 is a neuronal thrombospondin receptor responsible for excitatory CNS synaptogenesis Oxygen levels and the regulation of cell adhesion in the nervous system: a control point for morphogenesis in development, disease and evolution?Cell-based therapy for ischemic stroke Glial cells in (patho)physiology Astrocytes Control Synapse Formation, Function, and Elimination Thrombospondins as key regulators of synaptogenesis in the central nervous system Cortical astrocytes rewire somatosensory cortical circuits for peripheral neuropathic pain CD47 knockout mice exhibit improved recovery from spinal cord injury.The role of astrocyte-secreted matricellular proteins in central nervous system development and function Thrombospondins 1 and 2 are important for afferent synapse formation and function in the inner ear.An X-linked channelopathy with cardiomegaly due to a CLIC2 mutation enhancing ryanodine receptor channel activity.Functional identification of neuroprotective molecules.A role for thrombospondin-1 deficits in astrocyte-mediated spine and synaptic pathology in Down's syndrome.Reactive astrocytes function as phagocytes after brain ischemia via ABCA1-mediated pathway.Quantitative mass spectrometry-based proteomics reveals the dynamic range of primary mouse astrocyte protein secretion Astrocyte response to motor neuron injury promotes structural synaptic plasticity via STAT3-regulated TSP-1 expression.Induction of neuro-protective/regenerative genes in stem cells infiltrating post-ischemic brain tissue.Neuronal production of lipocalin-2 as a help-me signal for glial activation Gabapentin attenuates hyperexcitability in the freeze-lesion model of developmental cortical malformation.Role of astrocytes in manganese mediated neurotoxicity.Stem cells and stroke: opportunities, challenges and strategies Human neural stem cells enhance structural plasticity and axonal transport in the ischaemic brain Control of excitatory CNS synaptogenesis by astrocyte-secreted proteins Hevin and SPARC Decreased astrocytic thrombospondin-1 secretion after chronic ammonia treatment reduces the level of synaptic proteins: in vitro and in vivo studies The thrombospondins.Albumin induces excitatory synaptogenesis through astrocytic TGF-β/ALK5 signaling in a model of acquired epilepsy following blood-brain barrier dysfunction Genetic variant of BDNF (Val66Met) polymorphism attenuates stroke-induced angiogenic responses by enhancing anti-angiogenic mediator CD36 expression Neurological diseases as primary gliopathies: a reassessment of neurocentrism Astrocytes and disease: a neurodevelopmental perspective Astrocyte roles in traumatic brain injury.Astrocyte matricellular proteins that control excitatory synaptogenesis are regulated by inflammatory cytokines and correlate with paralysis severity during experimental autoimmune encephalomyelitis.The role of the neuro-astro-vascular unit in the etiology of ataxia telangiectasia.Human Umbilical Tissue-Derived Cells Promote Synapse Formation and Neurite Outgrowth via Thrombospondin Family Proteins Astrocyte-derived thrombospondins mediate the development of hippocampal presynaptic plasticity in vitro Adolescent Intermittent Alcohol Exposure: Dysregulation of Thrombospondins and Synapse Formation are Associated with Decreased Neuronal Density in the Adult Hippocampus.Cellular connections, microenvironment and brain angiogenesis in diabetes: Lost communication signals in the post-stroke period.Biodistribution of neural stem cells after intravascular therapy for hypoxic-ischemia Protective astrogenesis from the SVZ niche after injury is controlled by Notch modulator Thbs4.Thrombospondin-1 might be a therapeutic target to suppress RB cells by regulating the DNA double-strand breaks repair.Astrocyte-secreted thrombospondin-1 modulates synapse and spine defects in the fragile X mouse model.

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P2860

Thrombospondins 1 and 2 are necessary for synaptic plasticity and functional recovery after stroke.

http://www.wikidata.org/entity/Q46438887

description

2008 nî lūn-bûn

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2008年の論文

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2008年学术文章

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2008年学术文章

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2008年学术文章

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2008年学术文章

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2008年学术文章

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2008年學術文章

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2008年學術文章

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name

Thrombospondins 1 and 2 are ne ...... ctional recovery after stroke.

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Thrombospondins 1 and 2 are ne ...... ctional recovery after stroke.

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Thrombospondins 1 and 2 are ne ...... ctional recovery after stroke.

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Thrombospondins 1 and 2 are ne ...... ctional recovery after stroke.

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prefLabel

Thrombospondins 1 and 2 are ne ...... ctional recovery after stroke.

@en

Thrombospondins 1 and 2 are ne ...... ctional recovery after stroke.

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Journal of Cerebral Blood Flow & Metabolism

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Thrombospondins 1 and 2 are ne ...... ctional recovery after stroke.

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P2093

Ben A Barres

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Benjamin Wildman-Tobriner

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Gary K Steinberg

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Guo-hua Sun

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Jason Liauw

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Matthew Choi

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Matthew Percy

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Michael Choi

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Raphael G Guzman

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Stanley Hoang

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P2860

Interactions of thrombospondin with extracellular matrix proteins: selective binding to type V collagen

A new mathematical model for relative quantification in real-time RT-PCR

Fezl regulates the differentiation and axon targeting of layer 5 subcortical projection neurons in cerebral cortex

Differential regulation of thrombospondin-1 and thrombospondin-2 after focal cerebral ischemia/reperfusion

Thrombospondins are astrocyte-secreted proteins that promote CNS synaptogenesis

A gene expression atlas of the central nervous system based on bacterial artificial chromosomes

A PCR primer bank for quantitative gene expression analysis.

Plasticity of cortical projections after stroke.

Overexpression of thrombospondin-1 decreases angiogenesis and inhibits the growth of human cutaneous squamous cell carcinomas.

The lack of thrombospondin-1 (TSP1) dictates the course of wound healing in double-TSP1/TSP2-null mice

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1722-1732

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10.1038/JCBFM.2008.65

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10

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28

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18594557

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