Caveolin-1 is not required for murine intestinal cholesterol transport.
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Caveolin-1 regulates cell polarization and directional migration through Src kinase and Rho GTPasesImpact of the loss of caveolin-1 on lung mass and cholesterol metabolism in mice with and without the lysosomal cholesterol transporter, Niemann-Pick type C1.Cyclodextrin overcomes the transport defect in nearly every organ of NPC1 mice leading to excretion of sequestered cholesterol as bile acidNPC1L1 and cholesterol transportAnnexin A6-induced alterations in cholesterol transport and caveolin export from the Golgi complex.Znf202 affects high density lipoprotein cholesterol levels and promotes hepatosteatosis in hyperlipidemic mice.Genetic demonstration of intestinal NPC1L1 as a major determinant of hepatic cholesterol and blood atherogenic lipoprotein levels.Protein mediators of sterol transport across intestinal brush border membrane.Blockade of cholesterol absorption by ezetimibe reveals a complex homeostatic network in enterocytesCyclodextrin mediates rapid changes in lipid balance in Npc1-/- mice without carrying cholesterol through the bloodstream.Mechanisms for cellular cholesterol transport: defects and human disease.Emerging roles of the intestine in control of cholesterol metabolism.Role of the gut in lipid homeostasisThe role of Niemann-Pick C1 - Like 1 (NPC1L1) in intestinal sterol absorptionIntestinal caveolin-1 is important for dietary fatty acid absorption.Exposure to dietary lipid leads to rapid production of cytosolic lipid droplets near the brush border membrane.Diosgenin stimulation of fecal cholesterol excretion in mice is not NPC1L1 dependent.Transporters as drug targets: discovery and development of NPC1L1 inhibitors.Niemann-pick C1-like 1 (NPC1L1) protein in intestinal and hepatic cholesterol transport.Intestinal epithelial cell caveolin 1 regulates fatty acid and lipoprotein cholesterol plasma levels.Annexins as organizers of cholesterol- and sphingomyelin-enriched membrane microdomains in Niemann-Pick type C disease.Cholesterol-regulated translocation of NPC1L1 to the cell surface facilitates free cholesterol uptake.The absence of caveolin-1 increases proliferation and anchorage- independent growth by a Rac-dependent, Erk-independent mechanism.Weekly cyclodextrin administration normalizes cholesterol metabolism in nearly every organ of the Niemann-Pick type C1 mouse and markedly prolongs life.Genetic inactivation of NPC1L1 protects against sitosterolemia in mice lacking ABCG5/ABCG8.Plant sterols and stanols: effects on mixed micellar composition and LXR (target gene) activation.Regulation of intestinal NPC1L1 expression by dietary fish oil and docosahexaenoic acid.The Impact of Egg Nutrient Composition and Its Consumption on Cholesterol HomeostasisSelective Compensatory Induction of Hepatic HMG-CoA Reductase in Response to Inhibition of Cholesterol Absorption
P2860
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P2860
Caveolin-1 is not required for murine intestinal cholesterol transport.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
@zh-hant
name
Caveolin-1 is not required for murine intestinal cholesterol transport.
@en
Caveolin-1 is not required for murine intestinal cholesterol transport.
@nl
type
label
Caveolin-1 is not required for murine intestinal cholesterol transport.
@en
Caveolin-1 is not required for murine intestinal cholesterol transport.
@nl
prefLabel
Caveolin-1 is not required for murine intestinal cholesterol transport.
@en
Caveolin-1 is not required for murine intestinal cholesterol transport.
@nl
P2093
P2860
P356
P1476
Caveolin-1 is not required for murine intestinal cholesterol transport
@en
P2093
Joyce J Repa
Philip W Shaul
Richard G W Anderson
P2860
P304
28103-28109
P356
10.1074/JBC.M504609200
P407
P577
2005-05-26T00:00:00Z