Osteoclast size is controlled by Fra-2 through LIF/LIF-receptor signalling and hypoxia. - Wikidata - wikimedia - TriplyDB

Osteoclast size is controlled by Fra-2 through LIF/LIF-receptor signalling and hypoxia.

Osteoclast size is controlled by Fra-2 through LIF/LIF-receptor signalling and hypoxia.

http://www.wikidata.org/entity/Q46544733

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Hypoxia and TGF-beta drive breast cancer bone metastases through parallel signaling pathways in tumor cells and the bone microenvironment.Hypoxic regulation of osteoclast differentiation and bone resorption activity Sinomenine suppresses osteoclast formation and Mycobacterium tuberculosis H37Ra-induced bone loss by modulating RANKL signaling pathways Selective genomic targeting by FRA-2/FOSL2 transcription factor: regulation of the Rgs4 gene is mediated by a variant activator protein 1 (AP-1) promoter sequence/CREB-binding protein (CBP) mechanism LIF-dependent signaling: new pieces in the Lego c-Jun in Schwann cells promotes axonal regeneration and motoneuron survival via paracrine signaling.Retinoid X receptors orchestrate osteoclast differentiation and postnatal bone remodeling.Spatial and temporal analysis of gene expression during growth and fusion of the mouse facial prominences.A central role for hypoxic signaling in cartilage, bone, and hematopoiesis.The purinergic receptor P2X5 regulates inflammasome activity and hyper-multinucleation of murine osteoclasts.Blimp1-mediated repression of negative regulators is required for osteoclast differentiation.Transforming growth factor-{beta} coordinately induces suppressor of cytokine signaling 3 and leukemia inhibitory factor to suppress osteoclast apoptosis.Directed differentiation of hematopoietic precursors and functional osteoclasts from human ES and iPS cells A mathematical model of bone remodeling dynamics for normal bone cell populations and myeloma bone disease.Vaginally administered PEGylated LIF antagonist blocked embryo implantation and eliminated non-target effects on bone in mice.Hypoxia inhibits osteogenesis in human mesenchymal stem cells through direct regulation of RUNX2 by TWIST Important roles of PI3Kgamma in osteoclastogenesis and bone homeostasis.The loss of Cbl-phosphatidylinositol 3-kinase interaction perturbs RANKL-mediated signaling, inhibiting bone resorption and promoting osteoclast survival.Hypoxia-inducible factor regulates osteoclast-mediated bone resorption: role of angiopoietin-like 4 Developing functional musculoskeletal tissues through hypoxia and lysyl oxidase-induced collagen cross-linking Fra-2/AP-1 controls bone formation by regulating osteoblast differentiation and collagen production.Associated bone mineral density and obstructive sleep apnea in chronic obstructive pulmonary disease c-Jun gene-modified Schwann cells: upregulating multiple neurotrophic factors and promoting neurite outgrowth Nmp4/CIZ suppresses the response of bone to anabolic parathyroid hormone by regulating both osteoblasts and osteoclasts.FOSL2 promotes leptin gene expression in human and mouse adipocytes.Benefits of hypoxic culture on bone marrow multipotent stromal cells.E proteins regulate osteoclast maturation and survival.MMP-9 facilitates selective proteolysis of the histone H3 tail at genes necessary for proficient osteoclastogenesis.The AP-1 transcription factor component Fosl2 potentiates the rate of myocardial differentiation from the zebrafish second heart field.Unique Distal Enhancers Linked to the Mouse Tnfsf11 Gene Direct Tissue-Specific and Inflammation-Induced Expression of RANKL.Nmp4/CIZ suppresses the parathyroid hormone anabolic window by restricting mesenchymal stem cell and osteoprogenitor frequency Understanding and targeting osteoclastic activity in prostate cancer bone metastases.Tet2 facilitates the derepression of myeloid target genes during CEBPα-induced transdifferentiation of pre-B cells.Autosomal dominant STAT3 deficiency and hyper-IgE syndrome: molecular, cellular, and clinical features from a French national survey Role of leukemia inhibitory factor in nasopharyngeal carcinogenesis.Mechanism of Regulation of Adipocyte Numbers in Adult Organisms Through Differentiation and Apoptosis Homeostasis.Transforming growth factor beta 1 induces CXCL16 and leukemia inhibitory factor expression in osteoclasts to modulate migration of osteoblast progenitors.β-catenin activity in late hypertrophic chondrocytes locally orchestrates osteoblastogenesis and osteoclastogenesis.The FOS transcription factor family differentially controls trophoblast migration and invasion.Fra-2/AP-1 controls adipocyte differentiation and survival by regulating PPARγ and hypoxia.

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P2860

Osteoclast size is controlled by Fra-2 through LIF/LIF-receptor signalling and hypoxia.

http://www.wikidata.org/entity/Q46544733

description

2008 nî lūn-bûn

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2008年の論文

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2008年学术文章

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2008年学术文章

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2008年学术文章

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2008年學術文章

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2008年學術文章

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name

Osteoclast size is controlled by Fra-2 through LIF/LIF-receptor signalling and hypoxia.

@en

Osteoclast size is controlled by Fra-2 through LIF/LIF-receptor signalling and hypoxia.

@nl

type

label

Osteoclast size is controlled by Fra-2 through LIF/LIF-receptor signalling and hypoxia.

@en

Osteoclast size is controlled by Fra-2 through LIF/LIF-receptor signalling and hypoxia.

@nl

prefLabel

Osteoclast size is controlled by Fra-2 through LIF/LIF-receptor signalling and hypoxia.

@en

Osteoclast size is controlled by Fra-2 through LIF/LIF-receptor signalling and hypoxia.

@nl

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Osteoclast size is controlled by Fra-2 through LIF/LIF-receptor signalling and hypoxia.

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Aline Bozec

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Arndt F Schilling

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Astrid Hoebertz

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Colin L Stewart

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Harald Scheuch

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Latifa Bakiri

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Matthias Priemel

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Michael Amling

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Vukoslav Komnenovic

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10.1038/NATURE07019

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