Expression of many immunologically important genes in Mycobacterium tuberculosis-infected macrophages is independent of both TLR2 and TLR4 but dependent on IFN-alphabeta receptor and STAT1.
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Immune responsive gene 1 (IRG1) promotes endotoxin tolerance by increasing A20 expression in macrophages through reactive oxygen speciesPhosphodiesterase 4 inhibition reduces innate immunity and improves isoniazid clearance of Mycobacterium tuberculosis in the lungs of infected miceThe SecA2 secretion factor of Mycobacterium tuberculosis promotes growth in macrophages and inhibits the host immune responseNOD2, RIP2 and IRF5 play a critical role in the type I interferon response to Mycobacterium tuberculosis.In mice, tuberculosis progression is associated with intensive inflammatory response and the accumulation of Gr-1 cells in the lungs.Induction of ER stress in macrophages of tuberculosis granulomas.Characterization of a novel necrotic granuloma model of latent tuberculosis infection and reactivation in mice.Integrated microRNA-mRNA-analysis of human monocyte derived macrophages upon Mycobacterium avium subsp. hominissuis infection.Large scale comparison of innate responses to viral and bacterial pathogens in mouse and macaque.Arginine usage in mycobacteria-infected macrophages depends on autocrine-paracrine cytokine signalingAntagonistic crosstalk between type I and II interferons and increased host susceptibility to bacterial infectionsCell-autonomous effector mechanisms against mycobacterium tuberculosis.Characterization of chicken spleen transcriptome after infection with Salmonella enterica serovar EnteritidisInteraction of Pattern Recognition Receptors with Mycobacterium TuberculosisRNA profiling in host-pathogen interactions.Mycobacterium tuberculosis triggers host type I IFN signaling to regulate IL-1β production in human macrophagesCytosolic access of Mycobacterium tuberculosis: critical impact of phagosomal acidification control and demonstration of occurrence in vivoInterleukin-10 alters effector functions of multiple genes induced by Borrelia burgdorferi in macrophages to regulate Lyme disease inflammationMycobacterium tuberculosis Hip1 dampens macrophage proinflammatory responses by limiting toll-like receptor 2 activationA Computational Gene Expression Score for Predicting Immune Injury in Renal AllograftsGene Regulatory Network Inference of Immunoresponsive Gene 1 (IRG1) Identifies Interferon Regulatory Factor 1 (IRF1) as Its Transcriptional Regulator in Mammalian MacrophagesKeap1 regulates inflammatory signaling in Mycobacterium avium-infected human macrophagesTLR9 regulates Th1 responses and cooperates with TLR2 in mediating optimal resistance to Mycobacterium tuberculosis.Identification of novel monosodium urate crystal regulated mRNAs by transcript profiling of dissected murine air pouch membranes.Immune response to Mycobacterium tuberculosis and identification of molecular markers of disease.A common rejection module (CRM) for acute rejection across multiple organs identifies novel therapeutics for organ transplantation.Containment of aerogenic Mycobacterium tuberculosis infection in mice does not require MyD88 adaptor function for TLR2, -4 and -9.A role for IL-18 in protective immunity against Mycobacterium tuberculosis.The impact of ISGylation during Mycobacterium tuberculosis infection in mice.Coxiella burnetii, the agent of Q fever, stimulates an atypical M2 activation program in human macrophages.Nucleotide-binding oligomerization domain protein 2-deficient mice control infection with Mycobacterium tuberculosis.RP105 facilitates macrophage activation by Mycobacterium tuberculosis lipoproteins.Sabine Ehrt: searching for mycobacterial stress points. Interview by Hema Bashyam.NOD2 pathway activation by MDP or Mycobacterium tuberculosis infection involves the stable polyubiquitination of Rip2.Type I Interferons in the Pathogenesis of Tuberculosis: Molecular Drivers and Immunological Consequences.IFN-β: A Contentious Player in Host-Pathogen Interaction in Tuberculosis.For when bacterial infections persist: Toll-like receptor-inducible direct antimicrobial pathways in macrophages.Immunoregulatory and antimicrobial activities of myeloid cells.
P2860
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P2860
Expression of many immunologically important genes in Mycobacterium tuberculosis-infected macrophages is independent of both TLR2 and TLR4 but dependent on IFN-alphabeta receptor and STAT1.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
@zh-hant
name
Expression of many immunologic ...... -alphabeta receptor and STAT1.
@en
Expression of many immunologic ...... -alphabeta receptor and STAT1.
@nl
type
label
Expression of many immunologic ...... -alphabeta receptor and STAT1.
@en
Expression of many immunologic ...... -alphabeta receptor and STAT1.
@nl
prefLabel
Expression of many immunologic ...... -alphabeta receptor and STAT1.
@en
Expression of many immunologic ...... -alphabeta receptor and STAT1.
@nl
P2093
P1476
Expression of many immunologic ...... -alphabeta receptor and STAT1.
@en
P2093
Antje Blumenthal
Christopher M Hickey
Sabine Ehrt
Sheetal Gandotra
Shuangping Shi
P304
P356
10.4049/JIMMUNOL.175.5.3318
P407
P50
P577
2005-09-01T00:00:00Z