Glucose Catabolism in Liver Tumors Induced by c-MYC Can Be Sustained by Various PKM1/PKM2 Ratios and Pyruvate Kinase Activities.
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miR372 Promotes Progression of Liver Cancer Cells by Upregulating erbB-2 through Enhancement of YB-1.p300/CBP as a Key Nutritional Sensor for Hepatic Energy Homeostasis and Liver Fibrosis.PKM2 functions as a potential oncogene and is a crucial target of miR-148a and miR-326 in thyroid tumorigenesis.Pim1 promotes cell proliferation and regulates glycolysis via interaction with MYC in ovarian cancer
P2860
Glucose Catabolism in Liver Tumors Induced by c-MYC Can Be Sustained by Various PKM1/PKM2 Ratios and Pyruvate Kinase Activities.
description
2017 nî lūn-bûn
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2017年の論文
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2017年学术文章
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2017年学术文章
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2017年学术文章
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2017年学术文章
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2017年学术文章
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2017年学术文章
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name
Glucose Catabolism in Liver Tu ...... nd Pyruvate Kinase Activities.
@en
Glucose Catabolism in Liver Tu ...... nd Pyruvate Kinase Activities.
@nl
type
label
Glucose Catabolism in Liver Tu ...... nd Pyruvate Kinase Activities.
@en
Glucose Catabolism in Liver Tu ...... nd Pyruvate Kinase Activities.
@nl
prefLabel
Glucose Catabolism in Liver Tu ...... nd Pyruvate Kinase Activities.
@en
Glucose Catabolism in Liver Tu ...... nd Pyruvate Kinase Activities.
@nl
P2093
P2860
P1433
P1476
Glucose Catabolism in Liver Tu ...... nd Pyruvate Kinase Activities.
@en
P2093
Andrés Méndez-Lucas
Chencheng Xie
Darjus F Tschaharganeh
Diego F Calvisi
Jiaoyuan Jia
Jingxiao Wang
Mariia Yuneva
Paul C Driscoll
P2860
P304
P356
10.1158/0008-5472.CAN-17-0498
P407
P577
2017-06-19T00:00:00Z