Development of peritoneal adhesions in macrophage depleted mice.
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Obstacles and opportunities for understanding macrophage polarization.Macrophage Ablation Reduces M2-Like Populations and Jeopardizes Tumor Growth in a MAFIA-Based Glioma Model.ExoS and ExoT ADP ribosyltransferase activities mediate Pseudomonas aeruginosa keratitis by promoting neutrophil apoptosis and bacterial survival.Intestinal macrophages arising from CCR2(+) monocytes control pathogen infection by activating innate lymphoid cells.TLR4 and TLR5 on corneal macrophages regulate Pseudomonas aeruginosa keratitis by signaling through MyD88-dependent and -independent pathways.Macrophages: Their Emerging Roles in Bone.Murine ovarian cancer vascular leukocytes require arginase-1 activity for T cell suppression.Origin of fundus hyperautofluorescent spots and their role in retinal degeneration in a mouse model of Goldmann-Favre syndromeOvarian tumor-induced T cell suppression is alleviated by vascular leukocyte depletion.Bone marrow chimeras and c-fms conditional ablation (Mafia) mice reveal an essential role for resident myeloid cells in lipopolysaccharide/TLR4-induced corneal inflammation.Prevention of intraabdominal adhesions by local and systemic administration of immunosuppressive drugs.Macrophage and cancer cell cross-talk via CCR2 and CX3CR1 is a fundamental mechanism driving lung cancer.Macrophages and skeletal health.Colony Stimulating Factor-1 Receptor Expressing Cells Infiltrating the Cornea Control Corneal Nerve Degeneration in Response to HSV-1 Infection.Pharmacological HIF-inhibition attenuates postoperative adhesion formationPromoter Specificity and Efficacy in Conditional and Inducible Transgenic Targeting of Lung Macrophages.Macrophages promote osteoblastic differentiation in-vivo: implications in fracture repair and bone homeostasis.Effects of macrophage-dependent peroxisome proliferator-activated receptor γ signalling on adhesion formation after abdominal surgery in an experimental model.Conditional Macrophage Depletion Increases Inflammation and Does Not Inhibit the Development of Osteoarthritis in Obese Macrophage Fas-Induced Apoptosis-Transgenic Mice.
P2860
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P2860
Development of peritoneal adhesions in macrophage depleted mice.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
@zh
2005年學術文章
@zh-hant
name
Development of peritoneal adhesions in macrophage depleted mice.
@en
Development of peritoneal adhesions in macrophage depleted mice.
@nl
type
label
Development of peritoneal adhesions in macrophage depleted mice.
@en
Development of peritoneal adhesions in macrophage depleted mice.
@nl
prefLabel
Development of peritoneal adhesions in macrophage depleted mice.
@en
Development of peritoneal adhesions in macrophage depleted mice.
@nl
P2093
P1476
Development of peritoneal adhesions in macrophage depleted mice
@en
P2093
Alan M Kaplan
Don A Cohen
Rita Avdiushko
Sandra H Burnett
P304
P356
10.1016/J.JSS.2005.08.026
P577
2005-11-14T00:00:00Z