TLR4 and TLR5 on corneal macrophages regulate Pseudomonas aeruginosa keratitis by signaling through MyD88-dependent and -independent pathways.
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Ocular diseases: immunological and molecular mechanismsImpact of Microbiota on Resistance to Ocular Pseudomonas aeruginosa-Induced KeratitisA Dual Role for Corneal Dendritic Cells in Herpes Simplex Keratitis: Local Suppression of Corneal Damage and Promotion of Systemic Viral Dissemination3D quantitative imaging of unprocessed live tissue reveals epithelial defense against bacterial adhesion and subsequent traversal requires MyD88TLR4 drives the pathogenesis of acquired cholesteatoma by promoting local inflammation and bone destruction.Signaling Mediated by Toll-Like Receptor 5 Sensing of Pseudomonas aeruginosa Flagellin Influences IL-1β and IL-18 Production by Primary Fibroblasts Derived from the Human CorneaAspergillus and Fusarium corneal infections are regulated by Th17 cells and IL-17-producing neutrophils.Persistence of Innate Immune Pathways in Late Stage Human Bacterial and Fungal Keratitis: Results from a Comparative Transcriptome AnalysisRole of TLR5 and flagella in bacillus intraocular infection.Phage display against corneal epithelial cells produced bioactive peptides that inhibit Aspergillus adhesion to the corneasA crucial role of Flagellin in the induction of airway mucus production by Pseudomonas aeruginosa.Extracellular matrix protein lumican promotes clearance and resolution of Pseudomonas aeruginosa keratitis in a mouse modelHost response and bacterial virulence factor expression in Pseudomonas aeruginosa and Streptococcus pneumoniae corneal ulcers.Autophagy enhances NFκB activity in specific tissue macrophages by sequestering A20 to boost antifungal immunity.Topical flagellin-mediated innate defense against Candida albicans keratitis.Interferon-gamma-induced MD-2 protein expression and lipopolysaccharide (LPS) responsiveness in corneal epithelial cells is mediated by Janus tyrosine kinase-2 activation and direct binding of STAT1 protein to the MD-2 promoter.Expression of innate and adaptive immune mediators in human corneal tissue infected with Aspergillus or fusarium.Neutrophil IL-1β processing induced by pneumolysin is mediated by the NLRP3/ASC inflammasome and caspase-1 activation and is dependent on K+ efflux.Expression of IL-8, IL-6 and IL-1β in tears as a main characteristic of the immune response in human microbial keratitis.CD74 deficiency ameliorates Pseudomonas aeruginosa-induced ocular infection.Macrophage Ablation Reduces M2-Like Populations and Jeopardizes Tumor Growth in a MAFIA-Based Glioma Model.Inhibition of Corneal Inflammation by the Resolvin E1.ExoS and ExoT ADP ribosyltransferase activities mediate Pseudomonas aeruginosa keratitis by promoting neutrophil apoptosis and bacterial survival.Emodin ameliorates lipopolysaccharides-induced corneal inflammation in rats.Anti-inflammatory effect of emodin on lipopolysaccharide-induced keratitis in Wistar rats.TLR9 ligand CpG-ODN applied to the injured mouse cornea elicits retinal inflammation.Inhibition of high-mobility group box 1 protein (HMGB1) enhances bacterial clearance and protects against Pseudomonas Aeruginosa pneumonia in cystic fibrosis.Unexpected Roles for Toll-Like Receptor 4 and TRIF in Intraocular Infection with Gram-Positive Bacteria.Vasoactive intestinal peptide downregulates proinflammatory TLRs while upregulating anti-inflammatory TLRs in the infected cornea.CXCL1-deficient mice are highly sensitive to pseudomonas aeruginosa but not herpes simplex virus type 1 corneal infectionMast Cells Participate in Corneal Development in Mice.Cutting edge: IL-1β processing during Pseudomonas aeruginosa infection is mediated by neutrophil serine proteases and is independent of NLRC4 and caspase-1.A translocator-specific export signal establishes the translocator-effector secretion hierarchy that is important for type III secretion system function.Pattern recognition receptors in microbial keratitisTREM-2 promotes host resistance against Pseudomonas aeruginosa infection by suppressing corneal inflammation via a PI3K/Akt signaling pathway.Mammalian target of rapamycin regulates IL-10 and resistance to Pseudomonas aeruginosa corneal infection.TRIF mobilizes unique primary defense against Gram-negative bacteria in intestinal interface.Resident corneal c-fms(+) macrophages and dendritic cells mediate early cellular infiltration in adenovirus keratitis.Lithium chloride promotes host resistance against Pseudomonas aeruginosa keratitis.Host defense at the ocular surface
P2860
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P2860
TLR4 and TLR5 on corneal macrophages regulate Pseudomonas aeruginosa keratitis by signaling through MyD88-dependent and -independent pathways.
description
2010 nî lūn-bûn
@nan
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
2010年论文
@zh
2010年论文
@zh-cn
name
TLR4 and TLR5 on corneal macro ...... ent and -independent pathways.
@ast
TLR4 and TLR5 on corneal macro ...... ent and -independent pathways.
@en
type
label
TLR4 and TLR5 on corneal macro ...... ent and -independent pathways.
@ast
TLR4 and TLR5 on corneal macro ...... ent and -independent pathways.
@en
prefLabel
TLR4 and TLR5 on corneal macro ...... ent and -independent pathways.
@ast
TLR4 and TLR5 on corneal macro ...... ent and -independent pathways.
@en
P2093
P2860
P356
P1476
TLR4 and TLR5 on corneal macro ...... dent and -independent pathways
@en
P2093
Carey L Shive
Charles M Stopford
Eric Pearlman
Mausita Karmakar
Raniyah T Ramadan
Sanhita Roy
Scott Howell
Susan R Williams
Yiping Han
P2860
P304
P356
10.4049/JIMMUNOL.1000874
P407
P50
P577
2010-09-08T00:00:00Z