Overexpressed TGF-β in subchondral bone leads to mandibular condyle degradation. - Wikidata - wikimedia - TriplyDB

Overexpressed TGF-β in subchondral bone leads to mandibular condyle degradation.

Overexpressed TGF-β in subchondral bone leads to mandibular condyle degradation.

http://www.wikidata.org/entity/Q46845888

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Conditional removal of the canonical TGF-β1 signaling delays condylar cartilage degeneration induced by a partial discectomy in mice Gene Mutations Associated with Temporomandibular Joint Disorders: A Systematic Review.Injecting vascular endothelial growth factor into the temporomandibular joint induces osteoarthritis in mice.Systemic administration of strontium or NBD peptide ameliorates early stage cartilage degradation of mouse mandibular condyles Conditional Deletion of Fgfr3 in Chondrocytes leads to Osteoarthritis-like Defects in Temporomandibular Joint of Adult Mice.Overexpression of Dlx2 leads to postnatal condyle degradation.Current understanding of pathogenesis and treatment of TMJ osteoarthritis.Mouse genetic models for temporomandibular joint development and disorders The changing role of TGFβ in healthy, ageing and osteoarthritic joints.Mesenchymal Stem Cells for Cartilage Regeneration of TMJ Osteoarthritis.Osteochondral Interface Stiffening in Mandibular Condylar Osteoarthritis.Impaired periodontium and temporomandibular joints in tumour necrosis factor-α transgenic mice.Elevated marrow inflammatory cells and osteoclasts in subchondral osteosclerosis in human knee osteoarthritis.Aberrant activation of latent transforming growth factor-β initiates the onset of temporomandibular joint osteoarthritis

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P2860

Overexpressed TGF-β in subchondral bone leads to mandibular condyle degradation.

http://www.wikidata.org/entity/Q46845888

description

2013 nî lūn-bûn

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2013年の論文

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2013年学术文章

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2013年学术文章

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2013年学术文章

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2013年学术文章

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2013年学术文章

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2013年學術文章

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2013年學術文章

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2013年學術文章

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name

Overexpressed TGF-β in subchondral bone leads to mandibular condyle degradation.

@en

Overexpressed TGF-β in subchondral bone leads to mandibular condyle degradation.

@nl

type

label

Overexpressed TGF-β in subchondral bone leads to mandibular condyle degradation.

@en

Overexpressed TGF-β in subchondral bone leads to mandibular condyle degradation.

@nl

prefLabel

Overexpressed TGF-β in subchondral bone leads to mandibular condyle degradation.

@en

Overexpressed TGF-β in subchondral bone leads to mandibular condyle degradation.

@nl

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Journal of Dental Research

P1476

Overexpressed TGF-β in subchondral bone leads to mandibular condyle degradation.

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P2093

B Yu

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G Zhen

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K Jiao

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P2860

Inhibition of TGF-β signaling in mesenchymal stem cells of subchondral bone attenuates osteoarthritis

Progression of cartilage degradation, bone resorption and pain in rat temporomandibular joint osteoarthritis induced by injection of iodoacetate.

Smad3-dependent nuclear translocation of beta-catenin is required for TGF-beta1-induced proliferation of bone marrow-derived adult human mesenchymal stem cells.

Role of subchondral bone during early-stage experimental TMJ osteoarthritis.

Transforming growth factor-beta1 to the bone.

TGF-beta/Smad3 signals repress chondrocyte hypertrophic differentiation and are required for maintaining articular cartilage

Prevalence of clinical and radiographic signs of osteoarthrosis of the temporomandibular joint in an older persons community.

TGF-beta and osteoarthritis.

TGF-beta1-induced migration of bone mesenchymal stem cells couples bone resorption with formation.

Strontium ranelate reduces cartilage degeneration and subchondral bone remodeling in rat osteoarthritis model

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