IP3 receptor-dependent Ca2+ release modulates excitation-contraction coupling in rabbit ventricular myocytes.
about
Wide long lasting perinuclear Ca2+ release events generated by an interaction between ryanodine and IP3 receptors in canine Purkinje cellsCalcium and IP3 dynamics in cardiac myocytes: experimental and computational perspectives and approachesCaMKII regulation of cardiac ryanodine receptors and inositol triphosphate receptors.Calcium release from intra-axonal endoplasmic reticulum leads to axon degeneration through mitochondrial dysfunctionCaveolae, ion channels and cardiac arrhythmias.Intracellular calcium transients evoked by pulsed infrared radiation in neonatal cardiomyocytes.Isoform- and tissue-specific regulation of the Ca(2+)-sensitive transcription factor NFAT in cardiac myocytes and heart failure.Developmental aspects of cardiac Ca(2+) signaling: interplay between RyR- and IP(3)R-gated Ca(2+) storesThe IP3 receptor regulates cardiac hypertrophy in response to select stimuli.Activation of NFATc1 is directly mediated by IP3 in adult cardiac myocytesRole of endothelin in the induction of cardiac hypertrophy in vitro.Role of inositol 1,4,5-trisphosphate in the regulation of ventricular Ca(2+) signaling in intact mouse heart.SKF83959, an agonist of phosphatidylinositol-linked D(1)-like receptors, promotes ERK1/2 activation and cell migration in cultured rat astrocytes.Anacardium occidentale Linn. (Anacardiaceae) stem bark extract induces hypotensive and cardio-inhibitory effects in experimental animal models.The type 2 inositol 1,4,5-trisphosphate receptor, emerging functions for an intriguing Ca²⁺-release channel.Cytosolic and nuclear calcium signaling in atrial myocytes: IP3-mediated calcium release and the role of mitochondria.Inhibition of cAMP-Dependent PKA Activates β2-Adrenergic Receptor Stimulation of Cytosolic Phospholipase A2 via Raf-1/MEK/ERK and IP3-Dependent Ca2+ Signaling in Atrial Myocytes.Modulation of inositol 1,4,5-trisphosphate receptor type 2 channel activity by Ca2+/calmodulin-dependent protein kinase II (CaMKII)-mediated phosphorylationType 1 inositol (1,4,5)-trisphosphate receptor activates ryanodine receptor 1 to mediate calcium spark signaling in adult mammalian skeletal muscleThe role of the paracrine/autocrine mediator endothelin-1 in regulation of cardiac contractility and growth.The cardiac IP3 receptor: uncovering the role of "the other" calcium-release channel.Emerging roles of inositol 1,4,5-trisphosphate signaling in cardiac myocytes.Akt mediated mitochondrial protection in the heart: metabolic and survival pathways to the rescue.Inositol 1, 4, 5-trisphosphate receptors and human left ventricular myocytesCalcium/calmodulin-dependent kinase II regulation of cardiac ion channels.Inhibition of thromboxane A2-induced arrhythmias and intracellular calcium changes in cardiac myocytes by blockade of the inositol trisphosphate pathway.Role of the excessive amounts of circulating catecholamines and glucocorticoids in stress-induced heart disease.Calcineurin-NFATc regulates type 2 inositol 1,4,5-trisphosphate receptor (InsP3R2) expression during cardiac remodeling.Trimeric intracellular cation channels and sarcoplasmic/endoplasmic reticulum calcium homeostasisMultiple H+ sensors mediate the extracellular acidification-induced [Ca2+]i elevation in cultured rat ventricular cardiomyocytes.How cardiomyocytes sense pathophysiological stresses for cardiac remodeling.Cardiac inositol 1,4,5-trisphosphate receptors.Ca2+ removal mechanisms in mouse embryonic stem cell-derived cardiomyocytes.Endothelin-1 promotes hypertrophic remodelling of cardiac myocytes by activating sustained signalling and transcription downstream of endothelin type A receptors.Spatially defined InsP3-mediated signaling in embryonic stem cell-derived cardiomyocytes.Organelle membrane derived patches: reshaping classical methods for new targetsInositol 1,4,5-trisphosphate receptors: "exciting" players in cardiac excitation-contraction coupling?Ca²+ spark-dependent and -independent sarcoplasmic reticulum Ca²+ leak in normal and failing rabbit ventricular myocytes.Inositol-1,4,5-trisphosphate induced Ca2+ release and excitation-contraction coupling in atrial myocytes from normal and failing hearts.Why, where, and when do cardiac myocytes express inositol 1,4,5-trisphosphate receptors?
P2860
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P2860
IP3 receptor-dependent Ca2+ release modulates excitation-contraction coupling in rabbit ventricular myocytes.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年学术文章
@wuu
2007年学术文章
@zh
2007年学术文章
@zh-cn
2007年学术文章
@zh-hans
2007年学术文章
@zh-my
2007年学术文章
@zh-sg
2007年學術文章
@yue
2007年學術文章
@zh-hant
name
IP3 receptor-dependent Ca2+ re ...... n rabbit ventricular myocytes.
@en
IP3 receptor-dependent Ca2+ re ...... n rabbit ventricular myocytes.
@nl
type
label
IP3 receptor-dependent Ca2+ re ...... n rabbit ventricular myocytes.
@en
IP3 receptor-dependent Ca2+ re ...... n rabbit ventricular myocytes.
@nl
prefLabel
IP3 receptor-dependent Ca2+ re ...... n rabbit ventricular myocytes.
@en
IP3 receptor-dependent Ca2+ re ...... n rabbit ventricular myocytes.
@nl
P2093
P2860
P1476
IP3 receptor-dependent Ca2+ re ...... n rabbit ventricular myocytes.
@en
P2093
Aleksey V Zima
Gregory A Mignery
Joshua T Maxwell
Lothar A Blatter
Sabine Huke
Timothy L Domeier
P2860
P304
P356
10.1152/AJPHEART.01155.2007
P577
2007-11-30T00:00:00Z