Differential regulation of two types of intracellular calcium release channels during end-stage heart failure - Wikidata - wikimedia - TriplyDB

Differential regulation of two types of intracellular calcium release channels during end-stage heart failure

Differential regulation of two types of intracellular calcium release channels during end-stage heart failure

http://www.wikidata.org/entity/Q34203786

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Stanniocalcin-1 is a naturally occurring L-channel inhibitor in cardiomyocytes: relevance to human heart failure Increased InsP3Rs in the junctional sarcoplasmic reticulum augment Ca2+ transients and arrhythmias associated with cardiac hypertrophy Excitation-contraction coupling of human induced pluripotent stem cell-derived cardiomyocytes Stroma cell-derived factor-1α signaling enhances calcium transients and beating frequency in rat neonatal cardiomyocytes Maternal Nutrient Restriction Alters Ca2+ Handling Properties and Contractile Function of Isolated Left Ventricle Bundles in Male But Not Female Juvenile Rats Apelin increases contractility in failing cardiac muscle cADP-ribose/ryanodine channel/Ca2+-release signal transduction pathway in mesangial cells Reduced Ca2+ spark activity after subarachnoid hemorrhage disables BK channel control of cerebral artery tone.No contribution of IP3-R(2) to disease phenotype in models of dilated cardiomyopathy or pressure overload hypertrophy.Electrophysiological remodeling in heart failure Rate-dependent action potential alternans in human heart failure implicates abnormal intracellular calcium handling.Three-dimensional structure of the type 1 inositol 1,4,5-trisphosphate receptor at 24 A resolution.The IP3 receptor regulates cardiac hypertrophy in response to select stimuli.Chromogranin B regulates calcium signaling, nuclear factor kappaB activity, and brain natriuretic peptide production in cardiomyocytes Increased expression of fatty-acid and calcium metabolism genes in failing human heart.CaMKII inhibition rescues proarrhythmic phenotypes in the model of human ankyrin-B syndrome.Mitochondrial calcium overload is a key determinant in heart failure.Dysfunction in the βII spectrin-dependent cytoskeleton underlies human arrhythmia.Molecular determinants of altered contractility in heart failure.Calcium/calmodulin-dependent protein kinase II (CaMKII) inhibition ameliorates arrhythmias elicited by junctin ablation under stress conditions Myocardial region (right or left ventricle) and aetiology of heart failure can influence the inotropic effect of ouabain in failing human myocardium.Metabolomic approach to profile functional and metabolic changes in heart failure.Cytosolic and nuclear calcium signaling in atrial myocytes: IP3-mediated calcium release and the role of mitochondria.Enhanced ryanodine receptor-mediated calcium leak determines reduced sarcoplasmic reticulum calcium content in chronic canine heart failure.Modulation of inositol 1,4,5-trisphosphate receptor type 2 channel activity by Ca2+/calmodulin-dependent protein kinase II (CaMKII)-mediated phosphorylation Nuclear Ca2+ sparks and waves mediated by inositol 1,4,5-trisphosphate receptors in neonatal rat cardiomyocytes.The role of the paracrine/autocrine mediator endothelin-1 in regulation of cardiac contractility and growth.Arrhythmogenic ion-channel remodeling in the heart: heart failure, myocardial infarction, and atrial fibrillation.Altered sarcoplasmic reticulum calcium cycling--targets for heart failure therapy.Regulation of the ankyrin-B-based targeting pathway following myocardial infarction Emerging roles of inositol 1,4,5-trisphosphate signaling in cardiac myocytes.Requirement for Ca2+/calmodulin-dependent kinase II in the transition from pressure overload-induced cardiac hypertrophy to heart failure in mice.Nuclear and mitochondrial signalling Akts in cardiomyocytes Calcium uptake and release through sarcoplasmic reticulum in the inferior oblique muscles of patients with inferior oblique overaction.Ryanodine receptor-mediated arrhythmias and sudden cardiac death.Akt mediated mitochondrial protection in the heart: metabolic and survival pathways to the rescue.Inositol 1, 4, 5-trisphosphate receptors and human left ventricular myocytes Calcium/calmodulin-dependent kinase II regulation of cardiac ion channels.Ca handling during excitation-contraction coupling in heart failure.Endothelial dysfunction as a nexus for endothelial cell-cardiomyocyte miscommunication.

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P2860

Differential regulation of two types of intracellular calcium release channels during end-stage heart failure

http://www.wikidata.org/entity/Q34203786

description

1995 nî lūn-bûn

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1995 թուականի Փետրուարին հրատարակուած գիտական յօդուած

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1995 թվականի փետրվարին հրատարակված գիտական հոդված

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1995年の論文

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1995年論文

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1995年論文

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1995年论文

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Differential regulation of two ...... during end-stage heart failure

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Differential regulation of two ...... during end-stage heart failure

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Differential regulation of two ...... during end-stage heart failure

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Differential regulation of two ...... during end-stage heart failure

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Differential regulation of two ...... during end-stage heart failure

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Differential regulation of two ...... during end-stage heart failure

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Differential regulation of two ...... during end-stage heart failure

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Journal of Clinical Investigation

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Differential regulation of two ...... during end-stage heart failure

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A R Marks

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B S Fyfe

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P2860

Inositol trisphosphate and calcium signalling

Calcium-induced release of calcium from the cardiac sarcoplasmic reticulum

Regulation of cardiac gene expression during myocardial growth and hypertrophy: molecular studies of an adaptive physiologic response

Molecular cloning of cDNA encoding the Ca2+ release channel (ryanodine receptor) of rabbit cardiac muscle sarcoplasmic reticulum

Diltiazem increases late-onset congestive heart failure in postinfarction patients with early reduction in ejection fraction. The Adverse Experience Committee; and the Multicenter Diltiazem Postinfarction Research Group

Autocrine release of angiotensin II mediates stretch-induced hypertrophy of cardiac myocytes in vitro

The subtypes of the mouse inositol 1,4,5-trisphosphate receptor are expressed in a tissue-specific and developmentally specific manner

Inositol phosphates and cell signalling

Calcium release from cardiac sarcoplasmic reticulum induced by photorelease of calcium or Ins(1,4,5)P3.

Congestive heart failure. New frontiers

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10.1172/JCI117739

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