IL-18 is a key proximal mediator of contact hypersensitivity and allergen-induced Langerhans cell migration in murine epidermis.
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IL-18 and Cutaneous Inflammatory DiseasesMechanisms of chemical-induced innate immunity in allergic contact dermatitis.Preliminary performance data of the RHE/IL-18 assay performed on SkinEthic™ RHE for the identification of contact sensitizers.The role of the IL-12 cytokine family in directing T-cell responses in oral candidosis.IL-27 Regulates IL-18 binding protein in skin resident cellsTopical pimecrolimus inhibits high-dose UVB irradiation-induced epidermal Langerhans cell migration, via regulation of TNF-α and E-cadherin.Effects of interleukin-18 on cardiac fibroblast function and gene expressionIL-1 receptor signaling is required at multiple stages of sensitization and elicitation of the contact hypersensitivity responseAllergic contact dermatitis: epidemiology, molecular mechanisms, in vitro methods and regulatory aspects. Current knowledge assembled at an international workshop at BfR, Germany.Concurrent exposure to a dectin-1 agonist suppresses the Th2 response to epicutaneously introduced antigen in mice.Contrasting roles of the IL-1 and IL-18 receptors in MyD88-dependent contact hypersensitivity.Immunological mechanisms of contact hypersensitivity in mice.Dendritic cells and the assessment in vitro of skin sensitizing potential.Non-animal test methods for predicting skin sensitization potentials.Immunoregulation of skin sensitization and regulatory T cells.Bioactivation of drugs in the skin: relationship to cutaneous adverse drug reactions.In vitro approaches for detection of chemical sensitization.Reporter cell lines for skin sensitization testing.Chemical allergy: translating biology into hazard characterization.Immunology and genetics of tumour necrosis factor in allergic contact dermatitis.The NCTC 2544 IL-18 assay for the in vitro identification of contact allergens.Understanding chemical allergen potency: role of NLRP12 and Blimp-1 in the induction of IL-18 in human keratinocytes.Lack of the purinergic receptor P2X(7) results in resistance to contact hypersensitivityGingiva Equivalents Secrete Negligible Amounts of Key Chemokines Involved in Langerhans Cell Migration Compared to Skin Equivalents.Caspase-1 inhibition alleviates acute renal injury in rats with severe acute pancreatitisLangerhans cells require MyD88-dependent signals for Candida albicans response but not for contact hypersensitivity or migrationAn ITAM-Syk-CARD9 signalling axis triggers contact hypersensitivity by stimulating IL-1 production in dendritic cells.Development of a multiparametric in vitro model of skin sensitization.The balance between pro- and anti-inflammatory cytokines is crucial in human allergic contact dermatitis pathogenesis: the role of IL-1 family members.
P2860
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P2860
IL-18 is a key proximal mediator of contact hypersensitivity and allergen-induced Langerhans cell migration in murine epidermis.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年学术文章
@wuu
2007年学术文章
@zh
2007年学术文章
@zh-cn
2007年学术文章
@zh-hans
2007年学术文章
@zh-my
2007年学术文章
@zh-sg
2007年學術文章
@yue
2007年學術文章
@zh-hant
name
IL-18 is a key proximal mediat ...... migration in murine epidermis.
@en
IL-18 is a key proximal mediat ...... migration in murine epidermis.
@nl
type
label
IL-18 is a key proximal mediat ...... migration in murine epidermis.
@en
IL-18 is a key proximal mediat ...... migration in murine epidermis.
@nl
prefLabel
IL-18 is a key proximal mediat ...... migration in murine epidermis.
@en
IL-18 is a key proximal mediat ...... migration in murine epidermis.
@nl
P2093
P2860
P356
P1476
IL-18 is a key proximal mediat ...... migration in murine epidermis.
@en
P2093
Christos Antonopoulos
Foo Y Liew
Ian Kimber
John B Mee
Marie Cumberbatch
Rebecca J Dearman
Richard W Groves
Xiao-Qing Wei
P2860
P304
P356
10.1189/JLB.0604352
P577
2007-11-05T00:00:00Z