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Inactivation of a novel FGF23 regulator, FAM20C, leads to hypophosphatemic rickets in miceThe amazing osteocyteFGF23-mediated regulation of systemic phosphate homeostasis: is Klotho an essential player?FGF23 and Phosphate Wasting DisordersSkeletal secretion of FGF-23 regulates phosphate and vitamin D metabolismRegulation and function of the FGF23/klotho endocrine pathwaysCalcium regulation and bone mineral metabolism in elderly patients with chronic kidney diseaseFibroblast Growth Factor 23: A New Dimension to Diseases of Calcium-Phosphorus MetabolismRole of FGF23 in vitamin D and phosphate metabolism: implications in chronic kidney diseaseEndocrine crosstalk between muscle and boneGenetic disorders of phosphate regulationOsteocytes: master orchestrators of boneProtective roles of DMP1 in high phosphate homeostasisExcessive Osteocytic Fgf23 Secretion Contributes to Pyrophosphate Accumulation and Mineralization Defect in Hyp MiceDysregulated gene expression in the primary osteoblasts and osteocytes isolated from hypophosphatemic Hyp miceVascular calcification: When should we interfere in chronic kidney disease patients and how?From "Kidneys Govern Bones" to Chronic Kidney Disease, Diabetes Mellitus, and Metabolic Bone Disorder: A Crosstalk between Traditional Chinese Medicine and Modern ScienceCardiovascular biomarkers in chronic kidney disease: state of current research and clinical applicabilityFibroblast growth factors in cardiovascular disease: The emerging role of FGF21Phosphate homeostasis and its role in bone healthRecent advances in the renal-skeletal-gut axis that controls phosphate homeostasisGenetic evidence of serum phosphate-independent functions of FGF-23 on boneOsteocyte-specific deletion of Fgfr1 suppresses FGF23The parathyroid is a target organ for FGF23 in ratsFGF23 deficiency leads to mixed hearing loss and middle ear malformation in mice.Novel bone endocrine networks integrating mineral and energy metabolismIncreased bone volume and correction of HYP mouse hypophosphatemia in the Klotho/HYP mouse.Interactions of sclerostin with FGF23, soluble klotho and vitamin D in renal transplantationSpecific ablation of mouse Fam20C in cells expressing type I collagen leads to skeletal defects and hypophosphatemia.Bone mineral density and serum biochemical predictors of bone loss in patients with CKD on dialysis.FGF23 decreases renal NaPi-2a and NaPi-2c expression and induces hypophosphatemia in vivo predominantly via FGF receptor 1.Hypophosphatemic rickets: revealing novel control points for phosphate homeostasis.The receptor-dependent actions of 1,25-dihydroxyvitamin D are required for normal growth plate maturation in NPt2a knockout mice.Altered bone development and an increase in FGF-23 expression in Enpp1(-/-) mice.Extracellular matrix mineralization promotes E11/gp38 glycoprotein expression and drives osteocytic differentiationThe specific role of FAM20C in amelogenesisOsteocyte biology and space flight.A comparative transcriptome analysis identifying FGF23 regulated genes in the kidney of a mouse CKD model.CCL7 is a protective factor secreted by mechanically loaded osteocytesAltered renal FGF23-mediated activity involving MAPK and Wnt: effects of the Hyp mutation
P2860
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P2860
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年学术文章
@wuu
2006年学术文章
@zh
2006年学术文章
@zh-cn
2006年学术文章
@zh-hans
2006年学术文章
@zh-my
2006年学术文章
@zh-sg
2006年學術文章
@yue
2006年學術文章
@zh-hant
name
Pathogenic role of Fgf23 in Hyp mice.
@en
Pathogenic role of Fgf23 in Hyp mice.
@nl
type
label
Pathogenic role of Fgf23 in Hyp mice.
@en
Pathogenic role of Fgf23 in Hyp mice.
@nl
prefLabel
Pathogenic role of Fgf23 in Hyp mice.
@en
Pathogenic role of Fgf23 in Hyp mice.
@nl
P2093
P1476
Pathogenic role of Fgf23 in Hyp mice
@en
P2093
David W Rowe
Jianping Zhou
Shiguang Liu
P304
P356
10.1152/AJPENDO.00008.2006
P577
2006-01-31T00:00:00Z