Cocaine craving during protracted withdrawal requires PKCε priming within vmPFC.
about
Synaptic mechanisms underlying persistent cocaine cravingCocaine Self-Administration Elevates GluN2B within dmPFC Mediating Heightened Cue-Elicited Operant RespondingModeling the development of drug addiction in male and female animals.Kinase interest you in treating incubated cocaine-craving? A hypothetical model for treatment intervention during protracted withdrawal from cocaine.Protein kinase Cɛ activity regulates mGluR5 surface expression in the rat nucleus accumbens.
P2860
Cocaine craving during protracted withdrawal requires PKCε priming within vmPFC.
description
2016 nî lūn-bûn
@nan
2016年の論文
@ja
2016年学术文章
@wuu
2016年学术文章
@zh
2016年学术文章
@zh-cn
2016年学术文章
@zh-hans
2016年学术文章
@zh-my
2016年学术文章
@zh-sg
2016年學術文章
@yue
2016年學術文章
@zh-hant
name
Cocaine craving during protracted withdrawal requires PKCε priming within vmPFC.
@en
Cocaine craving during protracted withdrawal requires PKCε priming within vmPFC.
@nl
type
label
Cocaine craving during protracted withdrawal requires PKCε priming within vmPFC.
@en
Cocaine craving during protracted withdrawal requires PKCε priming within vmPFC.
@nl
prefLabel
Cocaine craving during protracted withdrawal requires PKCε priming within vmPFC.
@en
Cocaine craving during protracted withdrawal requires PKCε priming within vmPFC.
@nl
P2093
P2860
P356
P1433
P1476
Cocaine craving during protracted withdrawal requires PKCε priming within vmPFC
@en
P2093
Arianne D Sacramento
Bailey W Miller
Christina B Shin
Hannah E Silva
Karen K Szumlinski
Melissa G Wroten
Osnat Ben-Shahar
Tod E Kippin
P2860
P304
P356
10.1111/ADB.12354
P577
2016-01-14T00:00:00Z