A novel peptide CXCR ligand derived from extracellular matrix degradation during airway inflammation. - Wikidata - wikimedia - TriplyDB

A novel peptide CXCR ligand derived from extracellular matrix degradation during airway inflammation.

A novel peptide CXCR ligand derived from extracellular matrix degradation during airway inflammation.

http://www.wikidata.org/entity/Q46941338

about

Neutrophil elastase cleaves the murine hemidesmosomal protein BP180/type XVII collagen and generates degradation products that modulate experimental bullous pemphigoid Pathogenic triad in COPD: oxidative stress, protease-antiprotease imbalance, and inflammation α-1 Antitrypsin regulates human neutrophil chemotaxis induced by soluble immune complexes and IL-8 Gliadin induces an increase in intestinal permeability and zonulin release by binding to the chemokine receptor CXCR3 Fragments of extracellular matrix as mediators of inflammation The chemokine superfamily revisited Always cleave up your mess: targeting collagen degradation to treat tissue fibrosis Remodelling the extracellular matrix in development and disease Multiple sclerosis: molecular mechanisms and therapeutic opportunities Neutrophils: Between host defence, immune modulation, and tissue injury Inflammatory cells do not decrease the ultimate tensile strength of intact tendons in vivo and in vitro: protective role of mechanical loading Systemic disease during Streptococcus pneumoniae acute lung infection requires 12-lipoxygenase-dependent inflammation Nebulized hypertonic saline decreases IL-8 in sputum of patients with cystic fibrosis Rtp801, a suppressor of mTOR signaling, is an essential mediator of cigarette smoke-induced pulmonary injury and emphysema Epilysin (MMP-28) restrains early macrophage recruitment in Pseudomonas aeruginosa pneumonia Estrogen modulates TNF-alpha-induced inflammatory responses in rat aortic smooth muscle cells through estrogen receptor-beta activation Alternatively activated macrophages and collagen remodeling characterize the postpartum involuting mammary gland across species A thrombospondin-1 antagonist of transforming growth factor-beta activation blocks cardiomyopathy in rats with diabetes and elevated angiotensin II Genetic regulation of expression of leukotriene A4 hydrolase How dying cells alert the immune system to danger The extracellular matrix: a dynamic niche in cancer progression Matrix metalloproteinases and the regulation of tissue remodelling Matrix metalloproteinases: regulators of the tumor microenvironment Matrikines are key regulators in modulating the amplitude of lung inflammation in acute pulmonary infection.Role of leukotriene A4 hydrolase aminopeptidase in the pathogenesis of emphysema.A critical role for LTA4H in limiting chronic pulmonary neutrophilic inflammation Discoidin domain receptor 2 regulates neutrophil chemotaxis in 3D collagen matrices.Extracellular matrix-derived tripeptide proline-glycine-proline inhibits keratinocyte proliferation and migration.Localized bacterial infection induces systemic activation of neutrophils through Cxcr2 signaling in zebrafish.N-Acetylated Proline-Glycine-Proline Accelerates Cutaneous Wound Healing and Neovascularization by Human Endothelial Progenitor Cells Bronchiolar chemokine expression is different after single versus repeated cigarette smoke exposure Targeting prolyl endopeptidase with valproic acid as a potential modulator of neutrophilic inflammation Toll-Like Receptor 4 Engagement Mediates Prolyl Endopeptidase Release from Airway Epithelia via Exosomes Pulmonary matrix metalloproteinase-9 activity in mechanically ventilated children with respiratory syncytial virus.Collagen-derived N-acetylated proline-glycine-proline upregulates the expression of pro-inflammatory cytokines and extracellular matrix proteases in nucleus pulposus cells via the NF-κB and MAPK signaling pathways.A self-propagating matrix metalloprotease-9 (MMP-9) dependent cycle of chronic neutrophilic inflammation.Prolyl oligopeptidase is inhibited in relapsing-remitting multiple sclerosis.Proline-Glycine-Proline (PGP) and High Mobility Group Box Protein-1 (HMGB1): Potential Mediators of Cystic Fibrosis Airway Inflammation.Collagen I matrix turnover is regulated by fibronectin polymerization.Uric acid promotes an acute inflammatory response to sterile cell death in mice

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P2860

A novel peptide CXCR ligand derived from extracellular matrix degradation during airway inflammation.

http://www.wikidata.org/entity/Q46941338

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2006 nî lūn-bûn

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A novel peptide CXCR ligand de ...... on during airway inflammation.

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A novel peptide CXCR ligand de ...... on during airway inflammation.

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A novel peptide CXCR ligand de ...... on during airway inflammation.

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Aletta D Kraneveld

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Anneke H van Houwelingen

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Brett D Noerager

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F Shawn Galin

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Frans P Nijkamp

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Gert Folkerts

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Nathaniel M Weathington

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Patricia L Jackson

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10.1038/NM1361

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J. Edwin Blalock

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