K+ currents regulate the resting membrane potential, proliferation, and contractile responses in ventricular fibroblasts and myofibroblasts.
about
Transient receptor potential (TRP) channels and cardiac fibrosisTanshinone IIA protects against sudden cardiac death induced by lethal arrhythmias via repression of microRNA-1Computational Approaches to Understanding the Role of Fibroblast-Myocyte Interactions in Cardiac ArrhythmogenesisCross talk between cardiac myocytes and fibroblasts: from multiscale investigative approaches to mechanisms and functional consequencesSpiral-wave dynamics in a mathematical model of human ventricular tissue with myocytes and fibroblasts.Electrophysiological and structural remodeling in heart failure modulate arrhythmogenesis. 2D simulation studyTranscription factors MYOCD, SRF, Mesp1 and SMARCD3 enhance the cardio-inducing effect of GATA4, TBX5, and MEF2C during direct cellular reprogrammingDifferential and combined effects of cardiotrophin-1 and TGF-beta1 on cardiac myofibroblast proliferation and contractionComputational modeling of cardiac fibroblasts and fibrosisCalibration procedures for the quantitative determination of membrane potential in human cells using anionic dyes.Cardiac fibroblast: the renaissance cell.Electrotonic myofibroblast-to-myocyte coupling increases propensity to reentrant arrhythmias in two-dimensional cardiac monolayers.Size and ionic currents of unexcitable cells coupled to cardiomyocytes distinctly modulate cardiac action potential shape and pacemaking activity in micropatterned cell pairsSlow Conduction in the Border Zones of Patchy Fibrosis Stabilizes the Drivers for Atrial Fibrillation: Insights from Multi-Scale Human Atrial Modeling.Characterization of multiple ion channels in cultured human cardiac fibroblasts.Disease and region-related cardiac fibroblast potassium current variations and potential functional significance.Hydrogen sulphide suppresses human atrial fibroblast proliferation and transformation to myofibroblasts.Hyaluronan export through plasma membranes depends on concurrent K+ efflux by K(ir) channels.Fibroblast electrical remodeling in heart failure and potential effects on atrial fibrillationMolecular determinants of cardiac fibroblast electrical function and therapeutic implications for atrial fibrillation.The inward rectifier potassium channel Kir2.1 is expressed in mouse neutrophils from bone marrow and liver.Free-standing kinked nanowire transistor probes for targeted intracellular recording in three dimensions.Susceptibility to arrhythmia in the infarcted heart depends on myofibroblast density.Electrophysiological and structural remodeling in heart failure modulate arrhythmogenesis. 1D simulation study.Arrhythmogenic consequences of myofibroblast-myocyte couplingLoading effect of fibroblast-myocyte coupling on resting potential, impulse propagation, and repolarization: insights from a microstructure model.Low intensity 635 nm diode laser irradiation inhibits fibroblast-myofibroblast transition reducing TRPC1 channel expression/activity: New perspectives for tissue fibrosis treatment.Valsartan Upregulates Kir2.1 in Rats Suffering from Myocardial Infarction via Casein Kinase 2.Cardiac fibroblasts: friend or foe?Altered physiological functions and ion currents in atrial fibroblasts from patients with chronic atrial fibrillation.Modelling cardiac fibroblasts: interactions with myocytes and their impact on impulse propagationA review of the literature on cardiac electrical activity between fibroblasts and myocytes.Fibroblast KATP currents modulate myocyte electrophysiology in infarcted hearts.Effects of hydrogen peroxide on voltage-dependent K(+) currents in human cardiac fibroblasts through protein kinase pathwaysTwo distinct channels mediated by m2mAChR and α9nAChR co-exist in type II vestibular hair cells of guinea pig.A protective antiarrhythmic role of ursodeoxycholic acid in an in vitro rat model of the cholestatic fetal heart.Excitation-contraction coupling between human atrial myocytes with fibroblasts and stretch activated channel current: a simulation studyOver-expressed human TREK-1 inhibits CHO cell proliferation via inhibiting PKA and p38 MAPK pathways and subsequently inducing G1 arrest.Fibroblast-myocyte coupling in the heart: Potential relevance for therapeutic interventionsElectrotonic loading of anisotropic cardiac monolayers by unexcitable cells depends on connexin type and expression level
P2860
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P2860
K+ currents regulate the resting membrane potential, proliferation, and contractile responses in ventricular fibroblasts and myofibroblasts.
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年学术文章
@wuu
2005年学术文章
@zh
2005年学术文章
@zh-cn
2005年学术文章
@zh-hans
2005年学术文章
@zh-my
2005年学术文章
@zh-sg
2005年學術文章
@yue
2005年學術文章
@zh-hant
name
K+ currents regulate the resti ...... ibroblasts and myofibroblasts.
@en
K+ currents regulate the resti ...... ibroblasts and myofibroblasts.
@nl
type
label
K+ currents regulate the resti ...... ibroblasts and myofibroblasts.
@en
K+ currents regulate the resti ...... ibroblasts and myofibroblasts.
@nl
prefLabel
K+ currents regulate the resti ...... ibroblasts and myofibroblasts.
@en
K+ currents regulate the resti ...... ibroblasts and myofibroblasts.
@nl
P2093
P2860
P50
P1476
K+ currents regulate the resti ...... fibroblasts and myofibroblasts
@en
P2093
Imaizumi Y
Maccannell KA
Shibukawa Y
P2860
P304
P356
10.1152/AJPHEART.01220.2004
P577
2005-01-14T00:00:00Z