Time course of post-traumatic mitochondrial oxidative damage and dysfunction in a mouse model of focal traumatic brain injury: implications for neuroprotective therapy.
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Inhibition of cathepsin S produces neuroprotective effects after traumatic brain injury in miceMapping of phospholipids by MALDI imaging (MALDI-MSI): realities and expectationsMass-spectrometry based oxidative lipidomics and lipid imaging: applications in traumatic brain injuryTherapeutic window analysis of the neuroprotective effects of cyclosporine A after traumatic brain injuryNitric oxide and peroxynitrite in health and diseaseRole of Glia in Memory Deficits Following Traumatic Brain Injury: Biomarkers of Glia DysfunctionTraumatic Brain Injury pathophysiology and treatments: early, intermediate, and late phases post-injuryTraumatic brain injury and NADPH oxidase: a deep relationshipEmerging therapies in traumatic brain injuryN-Acetylaspartate reductions in brain injury: impact on post-injury neuroenergetics, lipid synthesis, and protein acetylationBench-to-bedside review: Mitochondrial injury, oxidative stress and apoptosis--there is nothing more practical than a good theoryClinical characteristics and pathophysiological mechanisms of focal and diffuse traumatic brain injuryMitochondrial injury after mechanical stretch of cortical neurons in vitro: biomarkers of apoptosis and selective peroxidation of anionic phospholipidsThioredoxin-Mimetic-Peptides Protect Cognitive Function after Mild Traumatic Brain Injury (mTBI)Syndromics: a bioinformatics approach for neurotrauma researchImaging mass spectrometry reveals loss of polyunsaturated cardiolipins in the cortical contusion, hippocampus, and thalamus after traumatic brain injuryNew insights into mitochondrial structure during cell death.Dietary fructose aggravates the pathobiology of traumatic brain injury by influencing energy homeostasis and plasticity.Metabolic acetate therapy for the treatment of traumatic brain injury.Age-related changes in mitochondrial respiration and oxidative damage in the cerebral cortex of the Fischer 344 rat.CLOCK and BMAL1 regulate MyoD and are necessary for maintenance of skeletal muscle phenotype and functionEarly mitochondrial dysfunction after cortical contusion injuryTemporal and spatial dynamics of peroxynitrite-induced oxidative damage after spinal cord contusion injury.Temporal and spatial dynamics of nrf2-antioxidant response elements mediated gene targets in cortex and hippocampus after controlled cortical impact traumatic brain injury in mice.Modern approaches to pediatric brain injury therapy.Gp91phox (NOX2) in classically activated microglia exacerbates traumatic brain injury.Antioxidant therapies in traumatic brain and spinal cord injuryLipid peroxidation-derived reactive aldehydes directly and differentially impair spinal cord and brain mitochondrial function.Suppression of oxidative stress and 5-lipoxygenase activation by edaravone improves depressive-like behavior after concussion.Attenuation of brain nitrostative and oxidative damage by brain cooling during experimental traumatic brain injury.The collective therapeutic potential of cerebral ketone metabolism in traumatic brain injuryMitochondrial protection after traumatic brain injury by scavenging lipid peroxyl radicals.Pharmacological inhibition of lipid peroxidation attenuates calpain-mediated cytoskeletal degradation after traumatic brain injury.α-Synuclein levels are elevated in cerebrospinal fluid following traumatic brain injury in infants and children: the effect of therapeutic hypothermia.A selective role for ARMS/Kidins220 scaffold protein in spatial memory and trophic support of entorhinal and frontal cortical neuronsNrf2-ARE activator carnosic acid decreases mitochondrial dysfunction, oxidative damage and neuronal cytoskeletal degradation following traumatic brain injury in mice.Cyclosporin A preserves mitochondrial function after traumatic brain injury in the immature rat and piglet.Mitochondria-associated microRNAs in rat hippocampus following traumatic brain injury.Life-time expression of the proteins peroxiredoxin, beta-synuclein, PARK7/DJ-1, and stathmin in the primary visual and primary somatosensory cortices in rats.Post-injury administration of the mitochondrial permeability transition pore inhibitor, NIM811, is neuroprotective and improves cognition after traumatic brain injury in rats
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P2860
Time course of post-traumatic mitochondrial oxidative damage and dysfunction in a mouse model of focal traumatic brain injury: implications for neuroprotective therapy.
description
2006 nî lūn-bûn
@nan
2006年の論文
@ja
2006年学术文章
@wuu
2006年学术文章
@zh
2006年学术文章
@zh-cn
2006年学术文章
@zh-hans
2006年学术文章
@zh-my
2006年学术文章
@zh-sg
2006年學術文章
@yue
2006年學術文章
@zh-hant
name
Time course of post-traumatic ...... s for neuroprotective therapy.
@en
Time course of post-traumatic ...... s for neuroprotective therapy.
@nl
type
label
Time course of post-traumatic ...... s for neuroprotective therapy.
@en
Time course of post-traumatic ...... s for neuroprotective therapy.
@nl
prefLabel
Time course of post-traumatic ...... s for neuroprotective therapy.
@en
Time course of post-traumatic ...... s for neuroprotective therapy.
@nl
P2093
P2860
P356
P1476
Time course of post-traumatic ...... s for neuroprotective therapy.
@en
P2093
Edward D Hall
Indrapal N Singh
Lamin H Mbye
Patrick G Sullivan
P2860
P304
P356
10.1038/SJ.JCBFM.9600297
P577
2006-03-15T00:00:00Z