A potassium channel-MiRP complex controls neurosensory function in Caenorhabditis elegans.
about
Oxidation of K(+) Channels in Aging and NeurodegenerationTargeted deletion of kcne2 impairs ventricular repolarization via disruption of I(K,slow1) and I(to,f)Interdependent roles for accessory KChIP2, KChIP3, and KChIP4 subunits in the generation of Kv4-encoded IA channels in cortical pyramidal neuronsAn evolutionarily conserved mode of modulation of Shaw-like K⁺ channels.Mutant analysis of the Shal (Kv4) voltage-gated fast transient K+ channel in Caenorhabditis elegansAuto-phosphorylation of a voltage-gated K+ channel controls non-associative learning.KCNQ-like potassium channels in Caenorhabditis elegans. Conserved properties and modulation.Gene expression of Caenorhabditis elegans neurons carries information on their synaptic connectivity.The membrane protein MiRP3 regulates Kv4.2 channels in a KChIP-dependent manner.Solution NMR of MPS-1 reveals a random coil cytosolic domain structureGuanine nucleotide exchange factor OSG-1 confers functional aging via dysregulated Rho signaling in Caenorhabditis elegans neuronsKChIP-like auxiliary subunits of Kv4 channels regulate excitability of muscle cells and control male turning behavior during mating in Caenorhabditis elegansLife span and stress resistance of Caenorhabditis elegans are differentially affected by glutathione transferases metabolizing 4-hydroxynon-2-enal.KCNE Regulation of K(+) Channel Trafficking - a Sisyphean Task?Molecular mechanisms underlying KVS-1-MPS-1 complex assembly.Cumulative activation of voltage-dependent KVS-1 potassium channels.The long processes of short interfering RNAs--RNA interference and its implications in neuronal cells.Trafficking of the IKs -complex in MDCK cells: site of subunit assembly and determinants of polarized localization.KCNE1 subunits require co-assembly with K+ channels for efficient trafficking and cell surface expression.KCNE3 truncation mutants reveal a bipartite modulation of KCNQ1 K+ channels.Oxidation of a potassium channel causes progressive sensory function loss during aging.A family of K+ channel ancillary subunits regulate taste sensitivity in Caenorhabditis elegans.Akinesia and freezing caused by Na+ leak-current channel (NALCN) deficiency corrected by pharmacological inhibition of K+ channels and gap junctions.A new mode of regulation of N-type inactivation in a Caenorhabditis elegans voltage-gated potassium channel.Single-walled carbon nanotubes are a new class of ion channel blockers.An arrhythmia susceptibility gene in Caenorhabditis elegans.Identification of small-molecule ion channel modulators in C. elegans channelopathy models
P2860
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P2860
A potassium channel-MiRP complex controls neurosensory function in Caenorhabditis elegans.
description
2003 nî lūn-bûn
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2003年の論文
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name
A potassium channel-MiRP compl ...... ion in Caenorhabditis elegans.
@en
A potassium channel-MiRP compl ...... ion in Caenorhabditis elegans.
@nl
type
label
A potassium channel-MiRP compl ...... ion in Caenorhabditis elegans.
@en
A potassium channel-MiRP compl ...... ion in Caenorhabditis elegans.
@nl
prefLabel
A potassium channel-MiRP compl ...... ion in Caenorhabditis elegans.
@en
A potassium channel-MiRP compl ...... ion in Caenorhabditis elegans.
@nl
P2093
P2860
P356
P1476
A potassium channel-MiRP compl ...... ion in Caenorhabditis elegans.
@en
P2093
Federico Sesti
Laura Bianchi
Suk-Mei Kwok
P2860
P304
12415-12424
P356
10.1074/JBC.M212788200
P407
P577
2003-01-17T00:00:00Z