Increased Notch3 Activity Mediates Pathological Changes in Structure of Cerebral Arteries.

about

Cerebral Vascular Disease and Neurovascular Injury in Ischemic Stroke.Therapeutic antibody targeting of Notch3 signaling prevents mural cell loss in CADASIL.

P2860

Q39116577-3C965EBC-8F3D-4873-B365-471D6C3358A0 Q48143289-F7EBF2C4-CED3-484E-8D79-1C54A6734E7C

P2860

Increased Notch3 Activity Mediates Pathological Changes in Structure of Cerebral Arteries.

Item

http://www.wikidata.org/entity/Q47155731

description

2016 nî lūn-bûn

@nan

2016年の論文

@ja

2016年学术文章

@wuu

2016年学术文章

@zh

2016年学术文章

@zh-cn

2016年学术文章

@zh-hans

2016年学术文章

@zh-my

2016年学术文章

@zh-sg

2016年學術文章

@yue

2016年學術文章

@zh-hant

name

Increased Notch3 Activity Medi ...... tructure of Cerebral Arteries.

@en

Increased Notch3 Activity Medi ...... tructure of Cerebral Arteries.

@nl

type

Item

label

Increased Notch3 Activity Medi ...... tructure of Cerebral Arteries.

@en

Increased Notch3 Activity Medi ...... tructure of Cerebral Arteries.

@nl

prefLabel

Increased Notch3 Activity Medi ...... tructure of Cerebral Arteries.

@en

Increased Notch3 Activity Medi ...... tructure of Cerebral Arteries.

@nl

P1476

Increased Notch3 Activity Mediates Pathological Changes in Structure of Cerebral Arteries

@en

P2093

Celine Baron-Menguy

http://www.w3.org/2001/XMLSchema#string

Lamia Ghezali

http://www.w3.org/2001/XMLSchema#string

Valérie Domenga-Denier

http://www.w3.org/2001/XMLSchema#string

P2860

The structural factor of hypertension: large and small artery alterations

Mechanisms of the inward remodeling process in resistance vessels: is the actin cytoskeleton involved?

Strong clustering and stereotyped nature of Notch3 mutations in CADASIL patients

Cerebral small vessel disease: from pathogenesis and clinical characteristics to therapeutic challenges

Transcriptome analysis for Notch3 target genes identifies Grip2 as a novel regulator of myogenic response in the cerebrovasculature

The canonical Notch signaling pathway: unfolding the activation mechanism.

Distinct phenotypic and functional features of CADASIL mutations in the Notch3 ligand binding domain

Cerebrovascular dysfunction and microcirculation rarefaction precede white matter lesions in a mouse genetic model of cerebral ischemic small vessel disease

Prolonged vasoconstriction of resistance arteries involves vascular smooth muscle actin polymerization leading to inward remodelling

Cadasil.

P304

60-70

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1161/HYPERTENSIONAHA.116.08015

http://www.w3.org/2001/XMLSchema#string

P407

English

P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P50

Anne Joutel

Frank M Faraci

P577

2016-11-07T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

27821617

http://www.w3.org/2001/XMLSchema#string

P932

5145742

http://www.w3.org/2001/XMLSchema#string

Increased Notch3 Activity Mediates Pathological Changes in Structure of Cerebral Arteries.

about

P2860

P2860

Increased Notch3 Activity Mediates Pathological Changes in Structure of Cerebral Arteries.

description

name

type

label

prefLabel

P1433

P1476

P2093

P2860

P304

P31

P356

P407

P433

P478

P50

P577

P698

P932

P356

P1433

P1476

P2093

P2860

P304

P31

P356

P407

P433

P478

P50

P577

P698

P932