about
Nox2-derived superoxide contributes to cerebral vascular dysfunction in diet-induced obesity.Angiotensin 1-7 reduces mortality and rupture of intracranial aneurysms in mice.Chronic aldosterone administration causes Nox2-mediated increases in reactive oxygen species production and endothelial dysfunction in the cerebral circulation.Protective vascular and cardiac effects of inducible nitric oxide synthase in mice with hyperhomocysteinemia.Role of peroxisome proliferator-activated receptor-γ in vascular muscle in the cerebral circulation.Genetic interference with peroxisome proliferator-activated receptor γ in smooth muscle enhances myogenic tone in the cerebrovasculature via A Rho kinase-dependent mechanism.Cullin-3 regulates vascular smooth muscle function and arterial blood pressure via PPARγ and RhoA/Rho-kinase.Deficiency of superoxide dismutase promotes cerebral vascular hypertrophy and vascular dysfunction in hyperhomocysteinemiaProtective Role for Tissue Inhibitor of Metalloproteinase-4, a Novel Peroxisome Proliferator-Activated Receptor-γ Target Gene, in Smooth Muscle in Deoxycorticosterone Acetate-Salt Hypertension.Interference with PPARγ in endothelium accelerates angiotensin II-induced endothelial dysfunction.Context-dependent effects of SOCS3 in angiotensin II-induced vascular dysfunction and hypertension in mice: mechanisms and role of bone marrow-derived cellsInterleukin-10 protects against aging-induced endothelial dysfunction.ASIC1A in neurons is critical for fear-related behaviors.Cerebral Vascular Disease and Neurovascular Injury in Ischemic Stroke.Heterogeneous Impact of ROCK2 on Carotid and Cerebrovascular FunctionIncreased Notch3 Activity Mediates Pathological Changes in Structure of Cerebral Arteries.Genetic Interference With Endothelial PPAR-γ (Peroxisome Proliferator-Activated Receptor-γ) Augments Effects of Angiotensin II While Impairing Responses to Angiotensin 1-7.Disease Highlights the Cellular Diversity of Neurovascular Units: Sign in Stranger.Changes in Cerebral Arteries and Parenchymal Arterioles With Aging: Role of Rho Kinase 2 and Impact of Genetic Background.Endothelial PPARγ (Peroxisome Proliferator-Activated Receptor-γ) Is Essential for Preventing Endothelial Dysfunction With Aging.Watching Small Vessel Disease GrowContributions of Aging to Cerebral Small Vessel DiseaseAcid-Sensing Ion Channels: Novel Mediators of Cerebral Vascular Responses
P50
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P50
description
investigador
@es
researcher
@en
name
Frank M Faraci
@en
type
label
Frank M Faraci
@en
prefLabel
Frank M Faraci
@en
P31
P496
0000-0002-0203-1690