Differential expression of RANKL and osteoprotegerin in gingival crevicular fluid of patients with periodontitis. - Wikidata - wikimedia - TriplyDB

Differential expression of RANKL and osteoprotegerin in gingival crevicular fluid of patients with periodontitis.

Differential expression of RANKL and osteoprotegerin in gingival crevicular fluid of patients with periodontitis.

http://www.wikidata.org/entity/Q47243804

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Biological response at the cellular level within the periodontal ligament on application of orthodontic force - An update Periodontal and inflammatory bowel diseases: Is there evidence of complex pathogenic interactions?The Cytolethal Distending Toxin Contributes to Microbial Virulence and Disease Pathogenesis by Acting As a Tri-Perditious Toxin Potassium channel-blockers as therapeutic agents to interfere with bone resorption of periodontal disease RANK/RANKL/OPG Signalization Implication in Periodontitis: New Evidence from a RANK Transgenic Mouse Model.Saliva as a diagnostic fluid.Role of periodontal pathogenic bacteria in RANKL-mediated bone destruction in periodontal disease.Effect of periodontal surgery on osteoprotegerin levels in gingival crevicular fluid, saliva, and gingival tissues of chronic periodontitis patients.B and T lymphocytes are the primary sources of RANKL in the bone resorptive lesion of periodontal disease Enhancement of periodontal tissue regeneration by transplantation of osteoprotegerin-engineered periodontal ligament stem cells Receptor activator of NF-kappaB ligand (RANKL) and CD 31 expressions in chronic periodontitis patients before and after surgery.MAPK usage in periodontal disease progression.RANKL inhibitors induce osteonecrosis of the jaw in mice with periapical disease.Are cementoblasts a subpopulation of osteoblasts or a unique phenotype?Oral Mucosal Lamina Propria-Progenitor Cells Exert Antibacterial Properties via the Secretion of Osteoprotegerin and Haptoglobin.Inflammation-induced bone remodeling in periodontal disease and the influence of post-menopausal osteoporosis.Novel host response therapeutic approaches to treat periodontal diseases.Gingival crevicularfluid osteoprotegerin levels in Indian population.Serum, saliva, and GCF concentration of RANKL and osteoprotegerin in smokers versus nonsmokers with chronic periodontitis RANKL inhibition through osteoprotegerin blocks bone loss in experimental periodontitis.Mechanisms of tooth eruption and orthodontic tooth movement.N-3 polyunsaturated fatty acid effect in periodontal disease: state of art and possible mechanisms involved.Salivary soluble receptor activator of nuclear factor kappa B ligand/osteoprotegerin ratio in periodontal disease and health Polymorphisms of Il-10 (-1082) and RANKL (-438) Genes and the Failure of Dental Implants.Host-derived diagnostic markers related to soft tissue destruction and bone degradation in periodontitis.Role of osteoclasts and interleukin-17 in the pathogenesis of rheumatoid arthritis: crucial 'human osteoclastology'.The RANKL-OPG system in clinical periodontology.The receptor activator of NF-κB ligand-osteoprotegerin system in pulpal and periapical disease.Expression of Toll-like receptors 2 and 4 and the OPG-RANKL-RANK system in inflammatory external root resorption and external cervical resorption.A preliminary investigation of short-term cytokine expression in gingival crevicular fluid secondary to high-level orthodontic forces and the associated root resorption: case series analytical study The osteoimmunology of alveolar bone loss.Biomarkers and Bacteria Around Implants and Natural Teeth in the Same Individuals.Tumor necrosis factor-α enhances RANKL expression in gingival epithelial cells via protein kinase A signaling.The presence of RANKL-OPG complex in human osteosarcoma U2OS.Transient receptor potential vanilloid-1 regulates osteoprotegerin/RANKL homeostasis in human periodontal ligament cells.Receptor activator of nuclear factor kappa B ligand and osteoprotegerin levels in gingival crevicular fluid.The presence of TRAIL-OPG complex in human osteosarcoma and human salivary gland adenocarcinoma.A dominant function of p38 mitogen-activated protein kinase signaling in receptor activator of nuclear factor-kappaB ligand expression and osteoclastogenesis induction by Aggregatibacter actinomycetemcomitans and Escherichia coli lipopolysaccharide.Clinical, radiographic, microbiological, and immunological outcomes of flapped vs. flapless dental implants: a prospective randomized controlled clinical trial.Tumor necrosis factor-alpha-converting enzyme (TACE) levels in periodontal diseases.

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P2860

Differential expression of RANKL and osteoprotegerin in gingival crevicular fluid of patients with periodontitis.

http://www.wikidata.org/entity/Q47243804

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2004 nî lūn-bûn

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Differential expression of RAN ...... f patients with periodontitis.

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Differential expression of RAN ...... f patients with periodontitis.

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Differential expression of RAN ...... f patients with periodontitis.

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Differential expression of RAN ...... f patients with periodontitis.

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Journal of Dental Research

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Differential expression of RAN ...... f patients with periodontitis.

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P2860

Osteoprotegerin ligand is a cytokine that regulates osteoclast differentiation and activation

Osteoprotegerin: a novel secreted protein involved in the regulation of bone density

Osteoclast differentiation factor is a ligand for osteoprotegerin/osteoclastogenesis-inhibitory factor and is identical to TRANCE/RANKL

Activated T cells regulate bone loss and joint destruction in adjuvant arthritis through osteoprotegerin ligand

The roles of osteoprotegerin and osteoprotegerin ligand in the paracrine regulation of bone resorption.

Functional human T-cell immunity and osteoprotegerin ligand control alveolar bone destruction in periodontal infection.

Host responses in periodontal diseases: current concepts.

TRANCE (tumor necrosis factor [TNF]-related activation-induced cytokine), a new TNF family member predominantly expressed in T cells, is a dendritic cell-specific survival factor.

Osteoclast differentiation factor in human osteosarcoma cell line.

The use of crevicular fluid prostaglandin E2 levels as a predictor of periodontal attachment loss.

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