Caenorhabditis elegans-based screen identifies Salmonella virulence factors required for conserved host-pathogen interactions.
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Moraxella osloensis gene expression in the slug host Deroceras reticulatumWorms and flies as genetically tractable animal models to study host-pathogen interactionsCe-Duox1/BLI-3 generates reactive oxygen species as a protective innate immune mechanism in Caenorhabditis elegansIdentification of Helicobacter pylori genes that contribute to stomach colonizationUnderstanding the complexities of Salmonella-host crosstalk as revealed by in vivo model organismsSalmonella enterica serovars Typhimurium and Typhi as model organisms: revealing paradigm of host-pathogen interactionsRecovery from an acute infection in C. elegans requires the GATA transcription factor ELT-2Transcriptional and functional analysis of the Neisseria gonorrhoeae Fur regulon.Transcriptional regulation of sdiA by cAMP-receptor protein, LeuO, and environmental signals in Salmonella enterica serovar Typhimurium.Pathogenicity of Salmonella enterica in Caenorhabditis elegans relies on disseminated oxidative stress in the infected hostStaphylococcus aureus virulence factors identified by using a high-throughput Caenorhabditis elegans-killing modelTasA-tasB, a new putative toxin-antitoxin (TA) system from Bacillus thuringiensis pGI1 plasmid is a widely distributed composite mazE-doc TA system.GATA transcription factor required for immunity to bacterial and fungal pathogens.The LysR-type transcriptional regulator LeuO controls expression of several genes in Salmonella enterica serovar TyphiVirulent synergistic effect between Enterococcus faecalis and Escherichia coli assayed by using the Caenorhabditis elegans model.Fate of the H-NS-repressed bgl operon in evolution of Escherichia coli.Multiple factors interact to produce responses resembling spectrum of human disease in Campylobacter jejuni infected C57BL/6 IL-10-/- mice.High-throughput screening and small animal models, where are we?Role of GATA transcription factor ELT-2 and p38 MAPK PMK-1 in recovery from acute P. aeruginosa infection in C. elegansRamA, a member of the AraC/XylS family, influences both virulence and efflux in Salmonella enterica serovar Typhimurium.The SKPO-1 peroxidase functions in the hypodermis to protect Caenorhabditis elegans from bacterial infection.Analysis of interactions of Salmonella type three secretion mutants with 3-D intestinal epithelial cellsThe pore-forming protein Cry5B elicits the pathogenicity of Bacillus sp. against Caenorhabditis elegans.Polyamines are required for virulence in Salmonella enterica serovar Typhimurium.Glycerol supplementation enhances L. reuteri's protective effect against S. Typhimurium colonization in a 3-D model of colonic epithelium.Salmonella enterica serovar Typhimurium ompS1 and ompS2 mutants are attenuated for virulence in mice.Yersinia pestis kills Caenorhabditis elegans by a biofilm-independent process that involves novel virulence factors.Ethanolamine utilization contributes to proliferation of Salmonella enterica serovar Typhimurium in food and in nematodes.The Caenorhabditis elegans ABL-1 tyrosine kinase is required for Shigella flexneri pathogenesis.Comparative genomics of Salmonella enterica serovars Derby and Mbandaka, two prevalent serovars associated with different livestock species in the UKNext generation sequencing analysis of nine Corynebacterium ulcerans isolates reveals zoonotic transmission and a novel putative diphtheria toxin-encoding pathogenicity island.LPS structure and PhoQ activity are important for Salmonella Typhimurium virulence in the Galleria mellonella infection model [corrected]A protocol to infect Caenorhabditis elegans with Salmonella typhimurium.Comparative proteogenomic analysis of the Leptospira interrogans virulence-attenuated strain IPAV against the pathogenic strain 56601.Association with pathogenic bacteria affects life-history traits and population growth in Caenorhabditis elegansCaenorhabditis elegans as a model to determine fitness of antibiotic-resistant Salmonella enterica serovar typhimurium.Oxidative stress enzymes are required for DAF-16-mediated immunity due to generation of reactive oxygen species by Caenorhabditis elegans.Disabling surveillance: bacterial type III secretion system effectors that suppress innate immunity.Specificity and complexity of the Caenorhabditis elegans innate immune response.Caenorhabditis elegans as a model for innate immunity to pathogens.
P2860
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P2860
Caenorhabditis elegans-based screen identifies Salmonella virulence factors required for conserved host-pathogen interactions.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年学术文章
@wuu
2004年学术文章
@zh
2004年学术文章
@zh-cn
2004年学术文章
@zh-hans
2004年学术文章
@zh-my
2004年学术文章
@zh-sg
2004年學術文章
@yue
2004年學術文章
@zh-hant
name
Caenorhabditis elegans-based s ...... ed host-pathogen interactions.
@en
Caenorhabditis elegans-based s ...... ed host-pathogen interactions.
@nl
type
label
Caenorhabditis elegans-based s ...... ed host-pathogen interactions.
@en
Caenorhabditis elegans-based s ...... ed host-pathogen interactions.
@nl
prefLabel
Caenorhabditis elegans-based s ...... ed host-pathogen interactions.
@en
Caenorhabditis elegans-based s ...... ed host-pathogen interactions.
@nl
P2093
P1433
P1476
Caenorhabditis elegans-based s ...... ed host-pathogen interactions.
@en
P2093
Alejandro Aballay
Beth A McCormick
Jennifer L Tenor
P304
P356
10.1016/J.CUB.2004.05.050
P407
P577
2004-06-01T00:00:00Z