EZH2 promotes neoplastic transformation through VAV interaction-dependent extranuclear mechanisms.

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EZH2 promotes neoplastic transformation through VAV interaction-dependent extranuclear mechanisms.

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2017 nî lūn-bûn

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2017年の論文

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年学术文章

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2017年學術文章

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2017年學術文章

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2017年學術文章

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EZH2 promotes neoplastic trans ...... ndent extranuclear mechanisms.

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EZH2 promotes neoplastic trans ...... ndent extranuclear mechanisms.

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EZH2 promotes neoplastic trans ...... ndent extranuclear mechanisms.

@en

EZH2 promotes neoplastic trans ...... ndent extranuclear mechanisms.

@nl

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EZH2 promotes neoplastic trans ...... ndent extranuclear mechanisms.

@en

EZH2 promotes neoplastic trans ...... ndent extranuclear mechanisms.

@nl

P1476

EZH2 promotes neoplastic trans ...... endent extranuclear mechanisms

@en

P2093

A Allahverdi

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E Cukuroglu

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H H Chung

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K P Tan

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L Nordenskiöld

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M S Madhusudhan

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N Venkatesan

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S Geifman-Shochat

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V C L Lin

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P2860

Phosphorylation of EZH2 activates STAT3 signaling via STAT3 methylation and promotes tumorigenicity of glioblastoma stem-like cells

Corepressor protein CDYL functions as a molecular bridge between polycomb repressor complex 2 and repressive chromatin mark trimethylated histone lysine 27

The Polycomb group protein EZH2 directly controls DNA methylation

Suz12 is essential for mouse development and for EZH2 histone methyltransferase activity

The genetic basis of early T-cell precursor acute lymphoblastic leukaemia.

EZH2 methyltransferase and H3K27 methylation in breast cancer

STAT3 serine phosphorylation by ERK-dependent and -independent pathways negatively modulates its tyrosine phosphorylation

Interaction of Vav with ENX-1, a putative transcriptional regulator of homeobox gene expression

EZH2 is a marker of aggressive breast cancer and promotes neoplastic transformation of breast epithelial cells

Targeting EZH2 in cancer

P2888

onc.2017.309

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461-477

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scholarly article

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10.1038/ONC.2017.309

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English

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P478

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P577

2017-10-02T00:00:00Z

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28967906

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