Homozygous L-SIGN (CLEC4M) plays a protective role in SARS coronavirus infection. - Wikidata - wikimedia - TriplyDB

Homozygous L-SIGN (CLEC4M) plays a protective role in SARS coronavirus infection.

Homozygous L-SIGN (CLEC4M) plays a protective role in SARS coronavirus infection.

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Lack of association between polymorphisms of MASP2 and susceptibility to SARS coronavirus infection Severe acute respiratory syndrome coronavirus as an agent of emerging and reemerging infection The role of host genetic factors in respiratory tract infectious diseases: systematic review, meta-analyses and field synopsis The origin and evolution of variable number tandem repeat of CLEC4M gene in the global human population The interferon gamma gene polymorphism +874 A/T is associated with severe acute respiratory syndrome Identifying epitopes responsible for neutralizing antibody and DC-SIGN binding on the spike glycoprotein of the severe acute respiratory syndrome coronavirus.Sex- and age-dependent association of SLC11A1 polymorphisms with tuberculosis in Chinese: a case control study.The association of RANTES polymorphism with severe acute respiratory syndrome in Hong Kong and Beijing Chinese Roles of TNF-alpha gene polymorphisms in the occurrence and progress of SARS-Cov infection: a case-control study.Functional genetic variants in DC-SIGNR are associated with mother-to-child transmission of HIV-1 The neck-region polymorphism of DC-SIGNR in peri-centenarian from Han Chinese population.HIV-1 transmission by dendritic cell-specific ICAM-3-grabbing nonintegrin (DC-SIGN) is regulated by determinants in the carbohydrate recognition domain that are absent in liver/lymph node-SIGN (L-SIGN)Respiratory viral infections in infants: causes, clinical symptoms, virology, and immunology.DC-SIGN (CD209) Promoter -336 A/G polymorphism is associated with dengue hemorrhagic fever and correlated to DC-SIGN expression and immune augmentation Pathology and pathogenesis of severe acute respiratory syndrome Genetic variation of the human α-2-Heremans-Schmid glycoprotein (AHSG) gene associated with the risk of SARS-CoV infection.The Effects of Temperature and Relative Humidity on the Viability of the SARS Coronavirus.The VNTR polymorphism of the DC-SIGNR gene and susceptibility to HIV-1 infection: a meta-analysis N-linked glycosylation facilitates sialic acid-independent attachment and entry of influenza A viruses into cells expressing DC-SIGN or L-SIGN Lectin switching during dengue virus infection Extremely low exposure of a community to severe acute respiratory syndrome coronavirus: false seropositivity due to use of bacterially derived antigens A novel subset of putative stem/progenitor CD34+Oct-4+ cells is the major target for SARS coronavirus in human lung.Specific asparagine-linked glycosylation sites are critical for DC-SIGN- and L-SIGN-mediated severe acute respiratory syndrome coronavirus entry.Surface vimentin is critical for the cell entry of SARS-CoV.Overexpression of NANOG in gestational trophoblastic diseases: effect on apoptosis, cell invasion, and clinical outcome.Priming with rAAV encoding RBD of SARS-CoV S protein and boosting with RBD-specific peptides for T cell epitopes elevated humoral and cellular immune responses against SARS-CoV infection.The C-type lectin receptor CLEC4M binds, internalizes, and clears von Willebrand factor and contributes to the variation in plasma von Willebrand factor levels.Novel roles of DC-SIGNR in colon cancer cell adhesion, migration, invasion, and liver metastasis.The highly cited SARS research literature.DC-SIGN, DC-SIGNR and LSECtin: C-type lectins for infection.Solution NMR analyses of the C-type carbohydrate recognition domain of DC-SIGNR protein reveal different binding modes for HIV-derived oligosaccharides and smaller glycan fragments Low expression of dendritic cell-specific intercellular adhesion molecule-grabbing nonintegrin-related protein in lung cancer and significant correlations with brain metastasis and natural killer cells.C-type lectin receptors in tuberculosis: what we know.Haplotypic diversity in human CEACAM genes: effects on susceptibility to meningococcal disease.CLEC4M-positive and CD81-negative Huh7 cells are not susceptible to JFH-1 HCVcc infection but mediate transinfection.Human lung ex vivo infection models.Aberrant activation of hedgehog signaling pathway in ovarian cancers: effect on prognosis, cell invasion and differentiation.Highly conserved regions within the spike proteins of human coronaviruses 229E and NL63 determine recognition of their respective cellular receptors.Geometry and adhesion of extracellular domains of DC-SIGNR neck length variants analyzed by force-distance measurements.What is the evidence of a role for host genetics in susceptibility to influenza A/H5N1?

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Homozygous L-SIGN (CLEC4M) plays a protective role in SARS coronavirus infection.

http://www.wikidata.org/entity/Q47665076

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Homozygous L-SIGN (CLEC4M) plays a protective role in SARS coronavirus infection.

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Homozygous L-SIGN

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type

label

Homozygous L-SIGN (CLEC4M) plays a protective role in SARS coronavirus infection.

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Homozygous L-SIGN

@nl

prefLabel

Homozygous L-SIGN (CLEC4M) plays a protective role in SARS coronavirus infection.

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Homozygous L-SIGN

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Nature Genetics

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Homozygous L-SIGN (CLEC4M) plays a protective role in SARS coronavirus infection.

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Annie N Y Cheung

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Bojian Zheng

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Chen-Lung S Lin

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Jonathan M Austyn

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Kelvin Y K Chan

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Kwok-Hung Chan

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Li-Chong Chan

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Malik Peiris

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Paul K H Tam

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Vera S F Chan

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P2860

Severe acute respiratory syndrome (SARS) and healthcare workers.

DC-SIGN; a related gene, DC-SIGNR; and CD23 form a cluster on 19p13.

DC-SIGN (dendritic cell-specific ICAM-grabbing non-integrin) and DC-SIGN-related (DC-SIGNR): friend or foe?

Cell line A549 as a model of the type II pneumocyte. Phospholipid biosynthesis from native and organometallic precursors.

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10.1038/NG1698

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1

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38

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Gavin R Screaton

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Leo Lit Man Poon

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2005-12-20T00:00:00Z

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7400488

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16369534

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severe acute respiratory syndrome-related coronavirus

severe acute respiratory syndrome coronavirus