IL-1 and TNF-alpha play a pivotal role in the host immune response in a mouse model of Staphylococcus aureus-induced experimental brain abscess. - Wikidata - wikimedia - TriplyDB

IL-1 and TNF-alpha play a pivotal role in the host immune response in a mouse model of Staphylococcus aureus-induced experimental brain abscess.

IL-1 and TNF-alpha play a pivotal role in the host immune response in a mouse model of Staphylococcus aureus-induced experimental brain abscess.

http://www.wikidata.org/entity/Q47730836

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Cytokines and chemokines at the crossroads of neuroinflammation, neurodegeneration, and neuropathic pain Immunopathogenesis of brain abscess Bacterial Exotoxins and the Inflammasome Pathogenesis of Staphylococcus aureus abscesses Toll-like receptor 2 modulates the proinflammatory milieu in Staphylococcus aureus-induced brain abscess Inflammasome activation and IL-1β/IL-18 processing are influenced by distinct pathways in microglia Neuroinflammation leads to region-dependent alterations in astrocyte gap junction communication and hemichannel activity.Correlation of DTI metrics in the wall and cavity of brain abscess with histology and immunohistochemistry.Staphylococcus aureus evades lysozyme-based peptidoglycan digestion that links phagocytosis, inflammasome activation, and IL-1beta secretion Microglia in infectious diseases of the central nervous system.Brain abscess: Current management.Neutrophil-derived IL-1β is sufficient for abscess formation in immunity against Staphylococcus aureus in mice Roles of Toll-like receptor 2 (TLR2) and superantigens on adaptive immune responses during CNS staphylococcal infection.Toll-like receptors in health and disease in the brain: mechanisms and therapeutic potential.Overview of toll-like receptors in the CNS.Toll-like receptors in brain abscess.Evaluation of capsular and acapsular strains of S. aureus in an experimental brain abscess model The interleukin-1β/CXCL1/2/neutrophil axis mediates host protection against group B streptococcal infection.A narrow repertoire of transcriptional modules responsive to pyogenic bacteria is impaired in patients carrying loss-of-function mutations in MYD88 or IRAK4 Fetal alcohol spectrum disorders and neuroimmune changes IL-1RI (interleukin-1 receptor type I) signalling is essential for host defence and hemichannel activity during acute central nervous system bacterial infection.NLRC4 inflammasome-mediated production of IL-1β modulates mucosal immunity in the lung against gram-negative bacterial infection Minocycline modulates neuroinflammation independently of its antimicrobial activity in staphylococcus aureus-induced brain abscess.Toll-like receptor (TLR) and inflammasome actions in the central nervous system Contribution of Staphylococcus aureus Coagulases and Clumping Factor A to Abscess Formation in a Rabbit Model of Skin and Soft Tissue Infection.Biofilm-infected intracerebroventricular shunts elicit inflammation within the central nervous system.MyD88-dependent signals are essential for the host immune response in experimental brain abscess.The role of Toll-like receptors in CNS response to microbial challenge.S. aureus-dependent microglial activation is selectively attenuated by the cyclopentenone prostaglandin 15-deoxy-Delta12,14- prostaglandin J2 (15d-PGJ2)Toll-like receptor 2 (TLR2) is pivotal for recognition of S. aureus peptidoglycan but not intact bacteria by microglia.The synthetic peroxisome proliferator-activated receptor-gamma agonist ciglitazone attenuates neuroinflammation and accelerates encapsulation in bacterial brain abscesses.15-deoxy-Delta12,14-prostaglandin J2 (15d-PGJ2) and ciglitazone modulate Staphylococcus aureus-dependent astrocyte activation primarily through a PPAR-gamma-independent pathway.Tumor necrosis factor-alpha (TNF-alpha) regulates Toll-like receptor 2 (TLR2) expression in microglia.Modulation of connexin expression and gap junction communication in astrocytes by the gram-positive bacterium S. aureus.A role for tumor necrosis factor-alpha in experimental Bacillus cereus endophthalmitis pathogenesis.Downregulation of CD40 ligand response in monocytes from sepsis patients.Staphylococcal Protein A Promotes Colonization and Immune Evasion of the Epidemic Healthcare-Associated MRSA ST239 Meprin A metalloproteases enhance renal damage and bladder inflammation after LPS challenge.TLR2 expression in astrocytes is induced by TNF-alpha- and NF-kappa B-dependent pathways.MyD88 expression by CNS-resident cells is pivotal for eliciting protective immunity in brain abscesses.

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IL-1 and TNF-alpha play a pivotal role in the host immune response in a mouse model of Staphylococcus aureus-induced experimental brain abscess.

http://www.wikidata.org/entity/Q47730836

description

2004 nî lūn-bûn

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2004年の論文

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年學術文章

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2004年學術文章

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name

IL-1 and TNF-alpha play a pivo ...... ed experimental brain abscess.

@en

IL-1 and TNF-alpha play a pivo ...... ed experimental brain abscess.

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type

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IL-1 and TNF-alpha play a pivo ...... ed experimental brain abscess.

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IL-1 and TNF-alpha play a pivo ...... ed experimental brain abscess.

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prefLabel

IL-1 and TNF-alpha play a pivo ...... ed experimental brain abscess.

@en

IL-1 and TNF-alpha play a pivo ...... ed experimental brain abscess.

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Journal of Neuropathology & Experimental Neurology

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IL-1 and TNF-alpha play a pivo ...... ed experimental brain abscess.

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P2093

Aaron C Baldwin

http://www.w3.org/2001/XMLSchema#string

Edward D Bearden

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Nilufer Esen

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Tammy Kielian

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P2860

A chemokine-driven positive feedback loop organizes lymphoid follicles

CXCL13 is required for B1 cell homing, natural antibody production, and body cavity immunity

Recombinant human interleukin-1 induces meningitis and blood-brain barrier injury in the rat. Characterization and comparison with tumor necrosis factor

Patterns of chemokine expression in models of Schistosoma mansoni inflammation and infection reveal relationships between type 1 and type 2 responses and chemokines in vivo.

Effect of deficiency of tumor necrosis factor alpha or both of its receptors on Streptococcus pneumoniae central nervous system infection and peritonitis.

Diminished virulence of an alpha-toxin mutant of Staphylococcus aureus in experimental brain abscesses

Interleukin-6 expression and regulation in astrocytes.

Immune function of microglia.

Immune function of astrocytes.

Pathogenesis and pathophysiology of pneumococcal meningitis.

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381-396

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10.1093/JNEN/63.4.381

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4

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63

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2004-04-01T00:00:00Z

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8605752

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15099027

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