TASK-3 two-pore domain potassium channels enable sustained high-frequency firing in cerebellar granule neurons.
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Pungent agents from Szechuan peppers excite sensory neurons by inhibiting two-pore potassium channelsTwo-pore domain potassium channels enable action potential generation in the absence of voltage-gated potassium channelsTASK Channel Deletion Reduces Sensitivity to Local Anesthetic-induced Seizures.Deletion of TASK1 and TASK3 channels disrupts intrinsic excitability but does not abolish glucose or pH responses of orexin/hypocretin neuronsMotoneuronal TASK channels contribute to immobilizing effects of inhalational general anestheticsExploring the significance of morphological diversity for cerebellar granule cell excitability.Pacemaker neuron and network oscillations depend on a neuromodulator-regulated linear current.Pacemaking in dopaminergic ventral tegmental area neurons: depolarizing drive from background and voltage-dependent sodium conductances.Two pore domain potassium channels in cerebral ischemia: a focus on K2P9.1 (TASK3, KCNK9).The role of linear and voltage-dependent ionic currents in the generation of slow wave oscillations.TWIK-1 contributes to the intrinsic excitability of dentate granule cells in mouse hippocampus.A new TASK for Dipeptidyl Peptidase-like Protein 6.Modulation of TRESK background K+ channel by membrane stretchAn unexpected role for TASK-3 potassium channels in network oscillations with implications for sleep mechanisms and anesthetic action.Leak K⁺ channel mRNAs in dorsal root ganglia: relation to inflammation and spontaneous pain behaviourIncreased excitability and altered action potential waveform in cerebellar granule neurons of the Ts65Dn mouse model of Down syndromeA Non-canonical Voltage-Sensing Mechanism Controls Gating in K2P K(+) Channels.Maternally inherited Birk Barel mental retardation dysmorphism syndrome caused by a mutation in the genomically imprinted potassium channel KCNK9Weaker control of the electrical properties of cerebellar granule cells by tonically active GABAA receptors in the Ts65Dn mouse model of Down's syndrome.New Perspectives on Genomic Imprinting, an Essential and Multifaceted Mode of Epigenetic Control in the Developing and Adult Brain.Gating of two pore domain potassium channels.Trafficking of neuronal two pore domain potassium channels.Molecular physiology of pH-sensitive background K(2P) channels.NALCN: a regulator of pacemaker activity.The role of K₂p channels in anaesthesia and sleep.Much more than a leak: structure and function of K₂p-channels.Diacylglycerol mediates regulation of TASK potassium channels by Gq-coupled receptors.Silencing the KCNK9 potassium channel (TASK-3) gene disturbs mitochondrial function, causes mitochondrial depolarization, and induces apoptosis of human melanoma cells.17β-estradiol downregulated the expression of TASK-1 channels in mouse neuroblastoma N2A cells.Enhancement of TWIK-related acid-sensitive potassium channel 3 (TASK3) two-pore domain potassium channel activity by tumor necrosis factor α.KV 10.1 opposes activity-dependent increase in Ca2+ influx into the presynaptic terminal of the parallel fibre-Purkinje cell synapse.Identification of a region in the TASK3 two pore domain potassium channel that is critical for its blockade by methanandamide.Neuromodulation to the Rescue: Compensation of Temperature-Induced Breakdown of Rhythmic Motor Patterns via Extrinsic Neuromodulatory Input.A role for TASK-1 (KCNK3) channels in the chemosensory control of breathing.The response of the tandem pore potassium channel TASK-3 (K(2P)9.1) to voltage: gating at the cytoplasmic mouth.Carotid body chemosensory responses in mice deficient of TASK channels.Probing the regulation of TASK potassium channels by PI4,5P₂ with switchable phosphoinositide phosphatases.The M1P1 loop of TASK3 K2P channels apposes the selectivity filter and influences channel function.Similar properties of transient, persistent, and resurgent Na currents in GABAergic and non-GABAergic vestibular nucleus neurons.Oxygen and mitochondrial inhibitors modulate both monomeric and heteromeric TASK-1 and TASK-3 channels in mouse carotid body type-1 cells.
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P2860
TASK-3 two-pore domain potassium channels enable sustained high-frequency firing in cerebellar granule neurons.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年学术文章
@wuu
2007年学术文章
@zh-cn
2007年学术文章
@zh-hans
2007年学术文章
@zh-my
2007年学术文章
@zh-sg
2007年學術文章
@yue
2007年學術文章
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2007年學術文章
@zh-hant
name
TASK-3 two-pore domain potassi ...... in cerebellar granule neurons.
@en
TASK-3 two-pore domain potassi ...... in cerebellar granule neurons.
@nl
type
label
TASK-3 two-pore domain potassi ...... in cerebellar granule neurons.
@en
TASK-3 two-pore domain potassi ...... in cerebellar granule neurons.
@nl
prefLabel
TASK-3 two-pore domain potassi ...... in cerebellar granule neurons.
@en
TASK-3 two-pore domain potassi ...... in cerebellar granule neurons.
@nl
P2093
P50
P1476
TASK-3 two-pore domain potassi ...... in cerebellar granule neurons.
@en
P2093
Cristina Sandu
Felicity G Alder
Harvinder Sambi
P304
P356
10.1523/JNEUROSCI.1427-07.2007
P407
P577
2007-08-01T00:00:00Z