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Development of a tonic form of synaptic inhibition in rat cerebellar granule cells resulting from persistent activation of GABAA receptorsTwo-pore domain potassium channels enable action potential generation in the absence of voltage-gated potassium channelsSignaling across the synapse: a role for Wnt and Dishevelled in presynaptic assembly and neurotransmitter releaseNR2B and NR2D subunits coassemble in cerebellar Golgi cells to form a distinct NMDA receptor subtype restricted to extrasynaptic sitesTectal-derived interneurons contribute to phasic and tonic inhibition in the visual thalamus.Altered activity in the central medial thalamus precedes changes in the neocortex during transitions into both sleep and propofol anesthesia.Profound desensitization by ambient GABA limits activation of δ-containing GABAA receptors during spilloverAn unexpected role for TASK-3 potassium channels in network oscillations with implications for sleep mechanisms and anesthetic action.Are extrasynaptic GABAA receptors important targets for sedative/hypnotic drugs?GABAergic inhibition of histaminergic neurons regulates active waking but not the sleep-wake switch or propofol-induced loss of consciousnessThe contribution of δ subunit-containing GABAA receptors to phasic and tonic conductance changes in cerebellum, thalamus and neocortexBottom-Up versus Top-Down Induction of Sleep by Zolpidem Acting on Histaminergic and Neocortex Neurons.The role of K₂p channels in anaesthesia and sleep.Neuronal ensembles sufficient for recovery sleep and the sedative actions of α2 adrenergic agonists.Circadian factor BMAL1 in histaminergic neurons regulates sleep architecture.Properties of GABA(A) receptor-mediated transmission at newly formed Golgi-granule cell synapses in the cerebellum.Maturation of EPSCs and intrinsic membrane properties enhances precision at a cerebellar synapse.Acting locally but sensing globally: impact of GABAergic synaptic plasticity on phasic and tonic inhibition in the thalamus.The involvement of hypothalamic sleep pathways in general anesthesia: testing the hypothesis using the GABAA receptor beta3N265M knock-in mouse.GABA transporter deficiency causes tremor, ataxia, nervousness, and increased GABA-induced tonic conductance in cerebellum.The Maturation of the Superior Collicular Map of Auditory Space in the Guinea Pig is Disrupted by Developmental Auditory Deprivation.TASK-3 two-pore domain potassium channels enable sustained high-frequency firing in cerebellar granule neurons.Synchronizing retinal activity in both eyes disrupts binocular map development in the optic tectum.Synaptic release generates a tonic GABA(A) receptor-mediated conductance that modulates burst precision in thalamic relay neurons.NMDA receptor diversity in the cerebellum: identification of subunits contributing to functional receptors.Cerebellar Golgi cell inhibition gets slowly more complicated.Modifying the subunit composition of TASK channels alters the modulation of a leak conductance in cerebellar granule neurons.Slow deactivation kinetics of NMDA receptors containing NR1 and NR2D subunits in rat cerebellar Purkinje cellsExperience-dependent mechanism of binocular map plasticity in Xenopus: incongruent connections are masked by retinal inputDeveloping more effective seizure therapies requires more selective drugs
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description
hulumtues
@sq
researcher
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wetenschapper
@nl
հետազոտող
@hy
name
Stephen G Brickley
@ast
Stephen G Brickley
@en
Stephen G Brickley
@es
Stephen G Brickley
@nl
Stephen G Brickley
@sl
type
label
Stephen G Brickley
@ast
Stephen G Brickley
@en
Stephen G Brickley
@es
Stephen G Brickley
@nl
Stephen G Brickley
@sl
prefLabel
Stephen G Brickley
@ast
Stephen G Brickley
@en
Stephen G Brickley
@es
Stephen G Brickley
@nl
Stephen G Brickley
@sl
P106
P21
P31
P496
0000-0002-0930-7346