Vasodilator dysfunction in aged spontaneously hypertensive rats: changes in NO synthase III and soluble guanylyl cyclase expression, and in superoxide anion production. - Wikidata - wikimedia - TriplyDB

Vasodilator dysfunction in aged spontaneously hypertensive rats: changes in NO synthase III and soluble guanylyl cyclase expression, and in superoxide anion production.

Vasodilator dysfunction in aged spontaneously hypertensive rats: changes in NO synthase III and soluble guanylyl cyclase expression, and in superoxide anion production.

http://www.wikidata.org/entity/Q47863878

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Prolonged exposure of chromaffin cells to nitric oxide down-regulates the activity of soluble guanylyl cyclase and corresponding mRNA and protein levels Does the sympathetic nervous system contribute to the pathophysiology of metabolic syndrome?Chaperone-dependent E3 ligase CHIP ubiquitinates and mediates proteasomal degradation of soluble guanylyl cyclase AGAP1, a novel binding partner of nitric oxide-sensitive guanylyl cyclase Downregulation of vascular soluble guanylate cyclase induced by high salt intake in spontaneously hypertensive rats Hydrogen peroxide induces a greater contraction in mesenteric arteries of spontaneously hypertensive rats through thromboxane A(2) production Nitric oxide and coronary vascular endothelium adaptations in hypertension.Hypertension reduces soluble guanylyl cyclase expression in the mouse aorta via the Notch signaling pathway A critical look at cardiovascular translational research.Endothelium-dependent vasodilation in hypertension: a review.Alternative splicing impairs soluble guanylyl cyclase function in aortic aneurysm Targeting the heme-oxidized nitric oxide receptor for selective vasodilatation of diseased blood vessels Aging enhances pressure-induced arterial superoxide formation Regulation of the expression of soluble guanylyl cyclase by reactive oxygen species Vascular nitric oxide and oxidative stress: determinants of endothelial adaptations to cardiovascular disease and to physical activity.Dynamic micro- and macrovascular remodeling in coronary circulation of obese Ossabaw pigs with metabolic syndrome Chronic administration of the probiotic kefir improves the endothelial function in spontaneously hypertensive rats.Sex-specific differences in chromosome-dependent regulation of vascular reactivity in female consomic rat strains from a SSxBN cross.Effect of oral organic nitrates on expression and activity of vascular soluble guanylyl cyclase Constitutive nitric oxide synthases in the heart from hypertrophy to failure.Cyclic GMP signaling in cardiovascular pathophysiology and therapeutics.Nitric oxide is less effective at inhibiting neointimal hyperplasia in spontaneously hypertensive rats.The ubiquitin-proteasome pathway and endothelial (dys)function.Regulation of soluble guanylate cyclase by matricellular thrombospondins: implications for blood flow.Ageing and microvasculature.Antioxidant Therapy: Is it your Gateway to Improved Cardiovascular Health?A New Hypothesis for Insulin Resistance in Hypertension Due to Receptor Cleavage.Elevated pressure selectively blunts flow-evoked vasodilatation in rat mesenteric small arteries.Lack of nitric oxide- and guanosine 3':5'-cyclic monophosphate-dependent regulation of alpha-thrombin-induced calcium transient in endothelial cells of spontaneously hypertensive rat hearts.Exercise changes regional vascular control by commissural NTS in spontaneously hypertensive rats.Aldosterone increases oxidant stress to impair guanylyl cyclase activity by cysteinyl thiol oxidation in vascular smooth muscle cells.Influence of hypertension on nitric oxide synthase expression and vascular effects of lipopolysaccharide in rat mesenteric arteries.A cGMP-dependent protein kinase assay for high throughput screening based on time-resolved fluorescence resonance energy transfer.Zn deficiency aggravates hypertension in spontaneously hypertensive rats: possible role of Cu/Zn-superoxide dismutase.Effect of a methionine-supplemented diet on the blood pressure of Wistar-Kyoto and spontaneously hypertensive rats.Nitric oxide and the mechanism of rat vascular smooth muscle photorelaxation.Exercise training improves aortic endothelium-dependent vasorelaxation and determinants of nitric oxide bioavailability in spontaneously hypertensive rats.Endothelial dysfunction in congestive heart failure: ACE inhibition vs. angiotensin II antagonism.Chronic limb-threatening ischemia could benefit from growth hormone therapy for wound healing and limb salvage.

P2860

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P2860

Vasodilator dysfunction in aged spontaneously hypertensive rats: changes in NO synthase III and soluble guanylyl cyclase expression, and in superoxide anion production.

http://www.wikidata.org/entity/Q47863878

description

1998 nî lūn-bûn

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1998年の論文

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1998年学术文章

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1998年学术文章

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1998年学术文章

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1998年学术文章

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1998年学术文章

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1998年学术文章

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1998年學術文章

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1998年學術文章

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name

Vasodilator dysfunction in age ...... n superoxide anion production.

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Vasodilator dysfunction in age ...... n superoxide anion production.

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type

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Vasodilator dysfunction in age ...... n superoxide anion production.

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Vasodilator dysfunction in age ...... n superoxide anion production.

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Vasodilator dysfunction in age ...... n superoxide anion production.

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Cardiovascular Research

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Vasodilator dysfunction in age ...... n superoxide anion production.

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Bauersachs J

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Bouloumié A

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Busse R

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Mülsch A

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Wiemer G

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P2860

Does superoxide underlie the pathogenesis of hypertension?

Single-Step Method of RNA Isolation by Acid Guanidinium Thiocyanate–Phenol–Chloroform Extraction

Molecular cloning and characterization of human endothelial nitric oxide synthase

Role of endothelium-derived nitric oxide in the regulation of blood pressure

Isolation and characterization of rat and human glyceraldehyde-3-phosphate dehydrogenase cDNAs: genomic complexity and molecular evolution of the gene

Angiotensin II-mediated hypertension in the rat increases vascular superoxide production via membrane NADH/NADPH oxidase activation. Contribution to alterations of vasomotor tone.

Display of the characteristics of endothelium-derived hyperpolarizing factor by a cytochrome P450-derived arachidonic acid metabolite in the coronary microcirculation.

Nitric oxide and its putative role in hypertension.

Regulation and functional consequences of endothelial nitric oxide formation.

Endothelium-dependent responses in hypertension.

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