Role of motoneurons in the generation of muscle spasms after spinal cord injury.
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A Review on Locomotor Training after Spinal Cord Injury: Reorganization of Spinal Neuronal Circuits and Recovery of Motor FunctionRecovery of neuronal and network excitability after spinal cord injury and implications for spasticityTargeted neuroplasticity for rehabilitationIncreases in human motoneuron excitability after cervical spinal cord injury depend on the level of injuryMilestones in clinical neurophysiology.Recovery of motoneuron and locomotor function after spinal cord injury depends on constitutive activity in 5-HT2C receptors.Discharge behaviors of trapezius motor units during exposure to low and high levels of acute psychosocial stressManagement of spasticity after spinal cord injury: current techniques and future directions.Single motor unit firing rate after stroke is higher on the less-affected side during stable low-level voluntary contractions.Amphetamine increases persistent inward currents in human motoneurons estimated from paired motor-unit activityVibration-induced extra torque during electrically-evoked contractions of the human calf musclesEffects of serotonergic medications on locomotor performance in humans with incomplete spinal cord injury.The relation between neuromechanical parameters and Ashworth score in stroke patientsRestoring walking after spinal cord injury: operant conditioning of spinal reflexes can helpIdentification and classification of involuntary leg muscle contractions in electromyographic records from individuals with spinal cord injury.Motor neuron firing dysfunction in spastic patients with primary lateral sclerosisPersistent inward currents in spinal motoneurons: important for normal function but potentially harmful after spinal cord injury and in amyotrophic lateral sclerosis.Origins of spontaneous firing of motor units in the spastic-paretic biceps brachii muscle of stroke survivorsMotor unit behavior during clonus.Adrenergic receptors modulate motoneuron excitability, sensory synaptic transmission and muscle spasms after chronic spinal cord injury.Motor unit firing rates during spasms in thenar muscles of spinal cord injured subjects.Motoneuron excitability and muscle spasms are regulated by 5-HT2B and 5-HT2C receptor activity.Self-sustained motor activity triggered by interlimb reflexes in chronic spinal cord injury, evidence of functional ascending propriospinal pathways.Polysynaptic excitatory postsynaptic potentials that trigger spasms after spinal cord injury in rats are inhibited by 5-HT1B and 5-HT1F receptorsEffects of persistent inward currents, accommodation, and adaptation on motor unit behavior: a simulation studyModulation of inhibitory strength and kinetics facilitates regulation of persistent inward currents and motoneuron excitability following spinal cord injuryExcitability changes in intracortical neural circuits induced by differentially controlled walking patterns.Muscle spasticity associated with reduced whole-leg perfusion in persons with spinal cord injury.Hip-phase-dependent flexion reflex modulation and expression of spasms in patients with spinal cord injury.Decrease of mRNA Editing after Spinal Cord Injury is Caused by Down-regulation of ADAR2 that is Triggered by Inflammatory ResponseIntrinsic excitability of human motoneurons in biceps brachii versus triceps brachii.Contribution of intrinsic properties and synaptic inputs to motoneuron discharge patterns: a simulation study.Prolonged quadriceps activity following imposed hip extension: a neurophysiological mechanism for stiff-knee gait?Increased spinal reflex excitability is associated with enhanced central activation during voluntary lengthening contractions in human spinal cord injury.Persistent inward currents in spinal motoneurons and their influence on human motoneuron firing patterns.Differential contributions of somatic and dendritic calcium-dependent potassium currents to the control of motoneuron excitability following spinal cord injury.Beginning at the end: repetitive firing properties in the final common pathwayActivation of 5-HT2A receptors upregulates the function of the neuronal K-Cl cotransporter KCC2.Constitutively active 5-HT2/α1 receptors facilitate muscle spasms after human spinal cord injuryReduction of spinal sensory transmission by facilitation of 5-HT1B/D receptors in noninjured and spinal cord-injured humans.
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P2860
Role of motoneurons in the generation of muscle spasms after spinal cord injury.
description
2004 nî lūn-bûn
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2004年の論文
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2004年学术文章
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2004年学术文章
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2004年学术文章
@zh-cn
2004年学术文章
@zh-hans
2004年学术文章
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2004年学术文章
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2004年學術文章
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name
Role of motoneurons in the generation of muscle spasms after spinal cord injury.
@en
Role of motoneurons in the generation of muscle spasms after spinal cord injury.
@nl
type
label
Role of motoneurons in the generation of muscle spasms after spinal cord injury.
@en
Role of motoneurons in the generation of muscle spasms after spinal cord injury.
@nl
prefLabel
Role of motoneurons in the generation of muscle spasms after spinal cord injury.
@en
Role of motoneurons in the generation of muscle spasms after spinal cord injury.
@nl
P2093
P356
P1433
P1476
Role of motoneurons in the generation of muscle spasms after spinal cord injury.
@en
P2093
Dave J Bennett
Michael E Knash
Monica A Gorassini
Philip J Harvey
P304
P356
10.1093/BRAIN/AWH243
P407
P577
2004-09-01T00:00:00Z