Macrophage interactions with neutrophils regulate Leishmania major infection. - Wikidata - wikimedia - TriplyDB

Macrophage interactions with neutrophils regulate Leishmania major infection.

Macrophage interactions with neutrophils regulate Leishmania major infection.

http://www.wikidata.org/entity/Q47914783

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Peroxisome Proliferator-Activated Receptor-γ-Mediated Polarization of Macrophages in Leishmania Infection IL-17 and IL-22 are associated with protection against human kala azar caused by Leishmania donovani Escaping Deleterious Immune Response in Their Hosts: Lessons from Trypanosomatids The site of the bite: Leishmania interaction with macrophages, neutrophils and the extracellular matrix in the dermis The influence of early neutrophil-Leishmania interactions on the host immune response to infection Immunoregulation in human American leishmaniasis: balancing pathology and protection Are Neutrophil Extracellular Traps Playing a Role in the Parasite Control in Active American Tegumentary Leishmaniasis Lesions?Rapid Sequestration of Leishmania mexicana by Neutrophils Contributes to the Development of Chronic Lesion Crosstalk between purinergic receptors and lipid mediators in leishmaniasis Neutrophils increase or reduce parasite burden in Trypanosoma cruzi-infected macrophages, depending on host strain: role of neutrophil elastase Enhanced Leishmania braziliensis infection following pre-exposure to sandfly saliva.Neutrophil-derived CCL3 is essential for the rapid recruitment of dendritic cells to the site of Leishmania major inoculation in resistant mice.B-1 cells modulate the murine macrophage response to Leishmania major infection.Neutrophils reduce the parasite burden in Leishmania (Leishmania) amazonensis-infected macrophages Dependency of B-1 Cells in the Maintenance of Splenic Interleukin-10 Producing Cells and Impairment of Macrophage Resistance in Visceral Leishmaniasis A secondary wave of neutrophil infiltration causes necrosis and ulceration in lesions of experimental American cutaneous leishmaniasis Molecular epidemiology for vector research on leishmaniasis.Innate immunity to Leishmania infection: within phagocytes.Toward a novel experimental model of infection to study American cutaneous leishmaniasis caused by Leishmania braziliensis Type I IFN receptor regulates neutrophil functions and innate immunity to Leishmania parasites.Efficient capture of infected neutrophils by dendritic cells in the skin inhibits the early anti-leishmania response.Insulin-like growth factor-I induces arginase activity in Leishmania amazonensis amastigote-infected macrophages through a cytokine-independent mechanism.The macrophage and the apoptotic cell: an innate immune interaction viewed simplistically?Impact of protozoan cell death on parasite-host interactions and pathogenesis Blocking junctional adhesion molecule C enhances dendritic cell migration and boosts the immune responses against Leishmania major.Dual effect of Lutzomyia longipalpis saliva on Leishmania braziliensis infection is mediated by distinct saliva-induced cellular recruitment into BALB/c mice ear Transgenic expression of CXCR3 on T cells enhances susceptibility to cutaneous Leishmania major infection by inhibiting monocyte maturation and promoting a Th2 response.Leishmania amazonensis promastigotes induce and are killed by neutrophil extracellular traps Impact of neutrophil-secreted myeloid related proteins 8 and 14 (MRP 8/14) on leishmaniasis progression.Neutrophils have a protective role during early stages of Leishmania amazonensis infection in BALB/c mice.Leishmania disease development depends on the presence of apoptotic promastigotes in the virulent inoculum.Neutrophils in oral paracoccidioidomycosis and the involvement of Nrf2 Impairment of T cell function in parasitic infections.Exposure to Leishmania braziliensis triggers neutrophil activation and apoptosis.Phlebotomine salivas inhibit immune inflammation-induced neutrophil migration via an autocrine DC-derived PGE2/IL-10 sequential pathway Interactions between Neutrophils and Leishmania braziliensis Amastigotes Facilitate Cell Activation and Parasite Clearance Distinct Macrophage Fates after in vitro Infection with Different Species of Leishmania: Induction of Apoptosis by Leishmania (Leishmania) amazonensis, but Not by Leishmania (Viannia) guyanensis.New Insights on the Inflammatory Role of Lutzomyia longipalpis Saliva in Leishmaniasis Reactive oxygen species and nitric oxide in cutaneous leishmaniasis.The central role of Fas-ligand cell signaling in inflammatory lung diseases.

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Macrophage interactions with neutrophils regulate Leishmania major infection.

http://www.wikidata.org/entity/Q47914783

description

2004 nî lūn-bûn

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2004年の論文

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年学术文章

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2004年學術文章

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2004年學術文章

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name

Macrophage interactions with neutrophils regulate Leishmania major infection.

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Macrophage interactions with neutrophils regulate Leishmania major infection.

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type

label

Macrophage interactions with neutrophils regulate Leishmania major infection.

@en

Macrophage interactions with neutrophils regulate Leishmania major infection.

@nl

prefLabel

Macrophage interactions with neutrophils regulate Leishmania major infection.

@en

Macrophage interactions with neutrophils regulate Leishmania major infection.

@nl

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JIMMUNOL.172.7.4454

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Journal of Immunology

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Macrophage interactions with neutrophils regulate Leishmania major infection

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Ana C Otero

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Andrea C Arnholdt

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Flávia L Ribeiro-Gomes

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George A DosReis

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Lea Cysne-Finkelstein

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Maria Carolina A Moniz-De-Souza

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Nitza A Gomes

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Sergio G Coutinho

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Vera L Calich

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4454-4462

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scholarly article

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10.4049/JIMMUNOL.172.7.4454

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7

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172

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Marcela F Lopes

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2004-04-01T00:00:00Z

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8667630

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