Spontaneous DNA damage in Saccharomyces cerevisiae elicits phenotypic properties similar to cancer cells.
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The current state of eukaryotic DNA base damage and repairDynamic compartmentalization of base excision repair proteins in response to nuclear and mitochondrial oxidative stressAdaptation of organisms by resonance of RNA transcription with the cellular redox cycleA two-tiered compensatory response to loss of DNA repair modulates aging and stress response pathways.Regulation of base excision repair: Ntg1 nuclear and mitochondrial dynamic localization in response to genotoxic stressA novel bisindole-PBD conjugate causes DNA damage induced apoptosis via inhibition of DNA repair pathway.Targeted detection of in vivo endogenous DNA base damage reveals preferential base excision repair in the transcribed strandDNA damage induces reactive oxygen species generation through the H2AX-Nox1/Rac1 pathway.Saccharomyces cerevisiae Apn1 mutation affecting stable protein expression mimics catalytic activity impairment: implications for assessing DNA repair capacity in humansChronic oxidative DNA damage due to DNA repair defects causes chromosomal instability in Saccharomyces cerevisiae.Oxidative stress-induced mutagenesis in single-strand DNA occurs primarily at cytosines and is DNA polymerase zeta-dependent only for adenines and guanines.Checkpoint kinase phosphorylation in response to endogenous oxidative DNA damage in repair-deficient stationary-phase Saccharomyces cerevisiaeUnraveling new functions of superoxide dismutase using yeast model system: Beyond its conventional role in superoxide radical scavenging.Biological consequences of oxidative stress-induced DNA damage in Saccharomyces cerevisiae.Abrogation of the Chk1-Pds1 checkpoint leads to tolerance of persistent single-strand breaks in Saccharomyces cerevisiae.Yap1: a DNA damage responder in Saccharomyces cerevisiae.Screening and identification of critical transcription factors involved in the protection of cardiomyocytes against hydrogen peroxide-induced damage by Yixin-shu.Oxidative DNA damage causes mitochondrial genomic instability in Saccharomyces cerevisiae.DNA damage-induced reactive oxygen species (ROS) stress response in Saccharomyces cerevisiae.Genome-wide map of Apn1 binding sites under oxidative stress in Saccharomyces cerevisiae.DNA base excision repair and nucleotide excision repair synergistically contribute to survival of stationary-phase cells of the fission yeast Schizosaccharomyces pombe.Identification of SUMO modification sites in the base excision repair protein, Ntg1.Nitrogen-Doped Graphene Quantum Dot-Combined Sodium 10-Amino-2-methoxyundecanoate: Studies of Proinflammatory Gene Expression and Live Cell Imagingi-BLESS is an ultra-sensitive method for detection of DNA double-strand breaks
P2860
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P2860
Spontaneous DNA damage in Saccharomyces cerevisiae elicits phenotypic properties similar to cancer cells.
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2004 nî lūn-bûn
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2004年の論文
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2004年学术文章
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2004年学术文章
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2004年学术文章
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Spontaneous DNA damage in Sacc ...... rties similar to cancer cells.
@en
Spontaneous DNA damage in Sacc ...... rties similar to cancer cells.
@nl
type
label
Spontaneous DNA damage in Sacc ...... rties similar to cancer cells.
@en
Spontaneous DNA damage in Sacc ...... rties similar to cancer cells.
@nl
prefLabel
Spontaneous DNA damage in Sacc ...... rties similar to cancer cells.
@en
Spontaneous DNA damage in Sacc ...... rties similar to cancer cells.
@nl
P2093
P2860
P356
P1476
Spontaneous DNA damage in Sacc ...... rties similar to cancer cells.
@en
P2093
Barbara A Evert
Binwei Song
Liu Jingjing
Paul W Doetsch
Tiffany B Salmon
Wolfram Siede
P2860
P304
22585-22594
P356
10.1074/JBC.M400468200
P407
P577
2004-03-12T00:00:00Z