Temporal and spatial patterns of Kv1.1 and Kv1.2 protein and gene expression in spinal cord white matter after acute and chronic spinal cord injury in rats: implications for axonal pathophysiology after neurotrauma.
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Human neural stem cells differentiate and promote locomotor recovery in an early chronic spinal cord injury NOD-scid mouse modelMyelin damage and repair in pathologic CNS: challenges and prospectsDelayed nerve stimulation promotes axon-protective neurofilament phosphorylation, accelerates immune cell clearance and enhances remyelination in vivo in focally demyelinated nervesCombination of electroacupuncture and grafted mesenchymal stem cells overexpressing TrkC improves remyelination and function in demyelinated spinal cord of rats.Delayed administration of a bio-engineered zinc-finger VEGF-A gene therapy is neuroprotective and attenuates allodynia following traumatic spinal cord injury.Chondroitinase and growth factors enhance activation and oligodendrocyte differentiation of endogenous neural precursor cells after spinal cord injury.Examination of the combined effects of chondroitinase ABC, growth factors and locomotor training following compressive spinal cord injury on neuroanatomical plasticity and kinematics.Transplantation of oligodendrocyte precursor cells improves locomotion deficits in rats with spinal cord irradiation injuryChanges in gap junction expression and function following ischemic injury of spinal cord white matterGenome-wide gene expression profiling of stress response in a spinal cord clip compression injury modelSensory afferents regenerated into dorsal columns after spinal cord injury remain in a chronic pathophysiological state.Sodium channel expression in the ventral posterolateral nucleus of the thalamus after peripheral nerve injuryAge-related molecular reorganization at the node of RanvierParanodal myelin damage after acute stretch in Guinea pig spinal cordAxonal thinning and extensive remyelination without chronic demyelination in spinal injured rats.Association between chronic stress-induced structural abnormalities in Ranvier nodes and reduced oligodendrocyte activity in major depression.No evidence for chronic demyelination in spared axons after spinal cord injury in a mouse.Voltage-gated potassium channels as therapeutic targets.Pharmacological modulation of voltage-gated potassium channels as a therapeutic strategy.Potassium channel blockers as an effective treatment to restore impulse conduction in injured axons.Voltage-gated potassium channels and the diversity of electrical signalling.Pharmacology and clinical efficacy of dalfampridine for treating multiple sclerosis.From demyelination to remyelination: the road toward therapies for spinal cord injury.Specific ion channels contribute to key elements of pathology during secondary degeneration following neurotrauma.Targeting ion channels for the treatment of autoimmune neuroinflammation.Potassium channel blocker, 4-aminopyridine-3-methanol, restores axonal conduction in spinal cord of an animal model of multiple sclerosis.Novel potassium channel blocker, 4-AP-3-MeOH, inhibits fast potassium channels and restores axonal conduction in injured guinea pig spinal cord white matter.Dysregulation of the neuregulin-1-ErbB network modulates endogenous oligodendrocyte differentiation and preservation after spinal cord injury.Functional changes in genetically dysmyelinated spinal cord axons of shiverer mice: role of juxtaparanodal Kv1 family K+ channels.Identification of temporal genes involved in the mechanisms of spinal cord injury.Platelet-derived growth factor-responsive neural precursors give rise to myelinating oligodendrocytes after transplantation into the spinal cords of contused rats and dysmyelinated mice.Alterations of juxtaparanodal domains in two rodent models of CNS demyelination.Perturbing chondroitin sulfate proteoglycan signaling through LAR and PTPσ receptors promotes a beneficial inflammatory response following spinal cord injury.
P2860
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P2860
Temporal and spatial patterns of Kv1.1 and Kv1.2 protein and gene expression in spinal cord white matter after acute and chronic spinal cord injury in rats: implications for axonal pathophysiology after neurotrauma.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年学术文章
@wuu
2004年学术文章
@zh
2004年学术文章
@zh-cn
2004年学术文章
@zh-hans
2004年学术文章
@zh-my
2004年学术文章
@zh-sg
2004年學術文章
@yue
2004年學術文章
@zh-hant
name
Temporal and spatial patterns ...... ophysiology after neurotrauma.
@en
Temporal and spatial patterns ...... ophysiology after neurotrauma.
@nl
type
label
Temporal and spatial patterns ...... ophysiology after neurotrauma.
@en
Temporal and spatial patterns ...... ophysiology after neurotrauma.
@nl
prefLabel
Temporal and spatial patterns ...... ophysiology after neurotrauma.
@en
Temporal and spatial patterns ...... ophysiology after neurotrauma.
@nl
P2860
P50
P1476
Temporal and spatial patterns ...... ophysiology after neurotrauma.
@en
P2860
P304
P356
10.1111/J.0953-816X.2004.03164.X
P407
P577
2004-02-01T00:00:00Z