Overexpression of NaV 1.6 channels is associated with the invasion capacity of human cervical cancer. - Wikidata - wikimedia - TriplyDB

Overexpression of NaV 1.6 channels is associated with the invasion capacity of human cervical cancer.

Overexpression of NaV 1.6 channels is associated with the invasion capacity of human cervical cancer.

http://www.wikidata.org/entity/Q48258918

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Voltage-gated sodium channels and cancer: is excitability their primary role?Sodium channel β subunits: emerging targets in channelopathies Sodium channel-inhibiting drugs and cancer survival: protocol for a cohort study using the CPRD primary care database Is there a role for voltage-gated Na+ channels in the aggressiveness of breast cancer?Voltage-gated sodium channels were differentially expressed in human normal prostate, benign prostatic hyperplasia and prostate cancer cells.Ranolazine inhibits NaV1.5-mediated breast cancer cell invasiveness and lung colonization Therapeutic potential for phenytoin: targeting Na(v)1.5 sodium channels to reduce migration and invasion in metastatic breast cancer.Voltage-gated sodium channels and metastatic disease.Blockade of voltage-gated sodium channels inhibits invasion of endocrine-resistant breast cancer cells Nav1.5 regulates breast tumor growth and metastatic dissemination in vivo SCN4B acts as a metastasis-suppressor gene preventing hyperactivation of cell migration in breast cancer Ion channels or aquaporins as novel molecular targets in gastric cancer.Neurological perspectives on voltage-gated sodium channels.Regulation of voltage-gated sodium channel expression in cancer: hormones, growth factors and auto-regulation.Dual roles of voltage-gated sodium channels in development and cancer.Voltage-gated sodium channel Nav 1.7 promotes gastric cancer progression through MACC1-mediated upregulation of NHE1.Voltage-Gated Na+ Channels: Not Just for Conduction.The influence of sodium on pathophysiology of multiple sclerosis.Gender-specific profiling in SCN1A polymorphisms and time-to-recurrence in patients with stage II/III colorectal cancer treated with adjuvant 5-fluoruracil chemotherapy.Expression of voltage-gated sodium channel Nav1.5 in non-metastatic colon cancer and its associations with estrogen receptor (ER)-β expression and clinical outcomes.Voltage-Gated Sodium Channel β Subunits and Their Related Diseases.Preserved SCN4B expression is an independent indicator of favorable recurrence-free survival in classical papillary thyroid cancer.The invasiveness of human cervical cancer associated to the function of Na1.6 channels is mediated by MMP-2 activity

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Overexpression of NaV 1.6 channels is associated with the invasion capacity of human cervical cancer.

http://www.wikidata.org/entity/Q48258918

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2011 nî lūn-bûn

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2011年の論文

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2011年学术文章

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2011年学术文章

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2011年学术文章

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2011年学术文章

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2011年学术文章

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2011年學術文章

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2011年學術文章

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2011年學術文章

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name

Overexpression of NaV 1.6 chan ...... city of human cervical cancer.

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Overexpression of NaV 1.6 chan ...... city of human cervical cancer.

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type

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Overexpression of NaV 1.6 chan ...... city of human cervical cancer.

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Overexpression of NaV 1.6 chan ...... city of human cervical cancer.

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Overexpression of NaV 1.6 chan ...... city of human cervical cancer.

@en

Overexpression of NaV 1.6 chan ...... city of human cervical cancer.

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International Journal of Cancer

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Overexpression of NaV 1.6 chan ...... city of human cervical cancer.

@en

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Ana Alfaro

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Brenda Marquina-Castillo

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Cindy S Ortiz

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Everardo Hernandez-Plata

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Ingrid Medina-Martinez

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Jaime Berumen

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Juan C Gomora

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Manuel Rivera

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Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

Estimates of worldwide burden of cancer in 2008: GLOBOCAN 2008

From ionic currents to molecular mechanisms: the structure and function of voltage-gated sodium channels

Expression of alternatively spliced sodium channel alpha-subunit genes. Unique splicing patterns are observed in dorsal root ganglia

International Union of Pharmacology. XLVII. Nomenclature and structure-function relationships of voltage-gated sodium channels

Voltage-gated Na+ channel beta1 subunit-mediated neurite outgrowth requires Fyn kinase and contributes to postnatal CNS development in vivo

Functional reciprocity between Na+ channel Nav1.6 and beta1 subunits in the coordinated regulation of excitability and neurite outgrowth.

Voltage-gated Na+ channel SCN5A is a key regulator of a gene transcriptional network that controls colon cancer invasion

Molecular changes in neurons in multiple sclerosis: altered axonal expression of Nav1.2 and Nav1.6 sodium channels and Na+/Ca2+ exchanger

Ether a go-go potassium channels as human cervical cancer markers.

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2013-2023

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scholarly article

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10.1002/IJC.26210

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9

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130

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2011-08-12T00:00:00Z

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51182036

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21630263

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