Energy thresholds in brain mitochondria. Potential involvement in neurodegeneration.
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Loss of m-AAA protease in mitochondria causes complex I deficiency and increased sensitivity to oxidative stress in hereditary spastic paraplegiaOxidative damage to mitochondrial complex I due to peroxynitrite: identification of reactive tyrosines by mass spectrometryMitochondrial lipid abnormality and electron transport chain impairment in mice lacking alpha-synucleinThiamine triphosphate synthesis in rat brain occurs in mitochondria and is coupled to the respiratory chainMetabolic control analysis in a cellular model of elevated MAO-B: relevance to Parkinson's diseaseParkinson's Disease: The Mitochondria-Iron LinkMitochondrial biology and Parkinson's diseaseThe chicken or the egg: mitochondrial dysfunction as a cause or consequence of toxicity in Huntington's diseaseThe ketogenic diet component decanoic acid increases mitochondrial citrate synthase and complex I activity in neuronal cellsEffect of 'binary mitochondrial heteroplasmy' on respiration and ATP synthesis: implications for mitochondrial diseasesEnergy failure: does it contribute to neurodegeneration?Metabolic consequences of the cytochrome c oxidase deficiency in brain of copper-deficient Mo(vbr) miceSelective binding of nuclear alpha-synuclein to the PGC1alpha promoter under conditions of oxidative stress may contribute to losses in mitochondrial function: implications for Parkinson's diseaseProteomic and metabolomic analyses of mitochondrial complex I-deficient mouse model generated by spontaneous B2 short interspersed nuclear element (SINE) insertion into NADH dehydrogenase (ubiquinone) Fe-S protein 4 (Ndufs4) geneSocially responsive effects of brain oxidative metabolism on aggression.Mitochondrial dysfunction in bipolar disorder: evidence from magnetic resonance spectroscopy research.Sp1 expression is disrupted in schizophrenia; a possible mechanism for the abnormal expression of mitochondrial complex I genes, NDUFV1 and NDUFV2Interaction of cyanide and nitric oxide with cytochrome c oxidase: implications for acute cyanide toxicity.Neuroanatomical pattern of mitochondrial complex I pathology varies between schizophrenia, bipolar disorder and major depression.Quantitative proteomics of synaptic and nonsynaptic mitochondria: insights for synaptic mitochondrial vulnerability.Ginkgo biloba extract ameliorates oxidative phosphorylation performance and rescues abeta-induced failureAge-related changes in mitochondrial respiration and oxidative damage in the cerebral cortex of the Fischer 344 rat.Bioenergetics in Huntington's disease.Repositioning chlorpromazine for treating chemoresistant glioma through the inhibition of cytochrome c oxidase bearing the COX4-1 regulatory subunit.Mechanisms of pathogenesis in drug hepatotoxicity putting the stress on mitochondria.Differential effects of antiretroviral nucleoside analogs on mitochondrial function in HepG2 cells.A mild impairment of mitochondrial electron transport has sex-specific effects on lifespan and aging in mice.Long-lasting inhibition of presynaptic metabolism and neurotransmitter release by protein S-nitrosylation.Age-related mitochondrial changes after traumatic brain injuryCytochrome c oxidase loses catalytic activity and structural integrity during the aging process in Drosophila melanogaster.Expression profiles of mitochondrial genes in the frontal cortex and the caudate nucleus of developing humans and mice selectively bred for high and low fearNeuroprotective effect of EGb761® and low-dose whole-body γ-irradiation in a rat model of Parkinson's disease.The human postsynaptic density shares conserved elements with proteomes of unicellular eukaryotes and prokaryotes.Intrinsic bioenergetic properties and stress sensitivity of dopaminergic synaptosomes.Mitochondrial threshold effects.Normal brain ageing: models and mechanismsMechanisms of cell death pathway activation following drug-induced inhibition of mitochondrial complex I.Altered zinc transport disrupts mitochondrial protein processing/import in fragile X-associated tremor/ataxia syndrome.High-level inhibition of mitochondrial complexes III and IV is required to increase glutamate release from the nerve terminal.Tau oligomers impair memory and induce synaptic and mitochondrial dysfunction in wild-type mice.
P2860
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P2860
Energy thresholds in brain mitochondria. Potential involvement in neurodegeneration.
description
1998 nî lūn-bûn
@nan
1998年の論文
@ja
1998年学术文章
@wuu
1998年学术文章
@zh
1998年学术文章
@zh-cn
1998年学术文章
@zh-hans
1998年学术文章
@zh-my
1998年学术文章
@zh-sg
1998年學術文章
@yue
1998年學術文章
@zh-hant
name
Energy thresholds in brain mitochondria. Potential involvement in neurodegeneration.
@en
Energy thresholds in brain mitochondria. Potential involvement in neurodegeneration.
@nl
type
label
Energy thresholds in brain mitochondria. Potential involvement in neurodegeneration.
@en
Energy thresholds in brain mitochondria. Potential involvement in neurodegeneration.
@nl
prefLabel
Energy thresholds in brain mitochondria. Potential involvement in neurodegeneration.
@en
Energy thresholds in brain mitochondria. Potential involvement in neurodegeneration.
@nl
P356
P1476
Energy thresholds in brain mitochondria. Potential involvement in neurodegeneration
@en
P2093
P304
12753-12757
P356
10.1074/JBC.273.21.12753
P407
P50
P577
1998-05-01T00:00:00Z