Anandamide administration alone and after inhibition of fatty acid amide hydrolase (FAAH) increases dopamine levels in the nucleus accumbens shell in rats.
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High Times for Painful Blues: The Endocannabinoid System in Pain-Depression ComorbidityIndividual differences in response to positive and negative stimuli: endocannabinoid-based insight on approach and avoidance behaviorsΔ9-tetrahydrocannabinol and endocannabinoid degradative enzyme inhibitors attenuate intracranial self-stimulation in miceInhibition of FAAH and activation of PPAR: new approaches to the treatment of cognitive dysfunction and drug addictionAlterations of monetary reward and punishment processing in chronic cannabis users: an FMRI studyDopaminergic augmentation of delta-9-tetrahydrocannabinol (THC) discrimination: possible involvement of D2-induced formation of anandamideEndocannabinoid signalling in reward and addiction.Individual and additive effects of the CNR1 and FAAH genes on brain response to marijuana cues.In vivo pharmacology of endocannabinoids and their metabolic inhibitors: therapeutic implications in Parkinson's disease and abuse liabilitySelective alterations of the CB1 receptors and the fatty acid amide hydrolase in the ventral striatum of alcoholics and suicidesEnvironmental enrichment during early stages of life reduces the behavioral, neurochemical, and molecular effects of cocaine.The endocannabinoid system as a target for the treatment of neurodegenerative disease.Animal models of cannabinoid reward.The endogenous cannabinoid, anandamide, inhibits dopamine transporter function by a receptor-independent mechanism.Interactions between dopamine transporter and cannabinoid receptor ligands in rhesus monkeysEndocannabinoid 2-Arachidonoylglycerol Self-Administration by Sprague-Dawley Rats and Stimulation of in vivo Dopamine Transmission in the Nucleus Accumbens Shell.Regulation of monoamine transporters: Role of transporter phosphorylation.Drug addiction.Acetaminophen modulation of hydrocodone reward in rats.Innate difference in the endocannabinoid signaling and its modulation by alcohol consumption in alcohol-preferring sP rats.Endogenous fatty acid ethanolamides suppress nicotine-induced activation of mesolimbic dopamine neurons through nuclear receptorsChronic stimulation of the tone of endogenous anandamide reduces cue- and stress-induced relapse in rats.Chronic unpredictable stress enhances cocaine-conditioned place preference in type 1 cannabinoid receptor knockout mice.The fatty acid amide hydrolase inhibitor URB 597: interactions with anandamide in rhesus monkeysPromising cannabinoid-based therapies for Parkinson's disease: motor symptoms to neuroprotectionEndocannabinoid influence in drug reinforcement, dependence and addiction-related behaviors.New insights on endocannabinoid transmission in psychomotor disorders.Effects of Fatty Acid Amide Hydrolase (FAAH) Inhibitors in Non-Human Primate Models of Nicotine Reward and RelapseModulation of the endocannabinoid system: therapeutic potential against cocaine dependenceEndocannabinoid system involvement in brain reward processes related to drug abuseSupraspinal modulation of pain by cannabinoids: the role of GABA and glutamateThe endogenous cannabinoid anandamide has effects on motivation and anxiety that are revealed by fatty acid amide hydrolase (FAAH) inhibitionAcute effects of THC on time perception in frequent and infrequent cannabis usersThe endocannabinoid system in brain reward processesCannabis cue-elicited craving and the reward neurocircuitry.Examination of the Addictive and Behavioral Properties of Fatty Acid-Binding Protein Inhibitor SBFI26.The FAAH inhibitor URB597 efficiently reduces tyrosine hydroxylase expression through CB₁- and FAAH-independent mechanisms.Pharmacological modulation of the endocannabinoid signalling alters binge-type eating behaviour in female rats.Adaptations in endocannabinoid signaling in response to repeated homotypic stress: a novel mechanism for stress habituationPrior stimulation of the endocannabinoid system prevents methamphetamine-induced dopaminergic neurotoxicity in the striatum through activation of CB2 receptors.
P2860
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P2860
Anandamide administration alone and after inhibition of fatty acid amide hydrolase (FAAH) increases dopamine levels in the nucleus accumbens shell in rats.
description
2006 nî lūn-bûn
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2006年の論文
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2006年学术文章
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2006年学术文章
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name
Anandamide administration alon ...... cleus accumbens shell in rats.
@en
Anandamide administration alone and after inhibition of fatty acid amide hydrolase
@nl
type
label
Anandamide administration alon ...... cleus accumbens shell in rats.
@en
Anandamide administration alone and after inhibition of fatty acid amide hydrolase
@nl
prefLabel
Anandamide administration alon ...... cleus accumbens shell in rats.
@en
Anandamide administration alone and after inhibition of fatty acid amide hydrolase
@nl
P2860
P50
P921
P1476
Anandamide administration alon ...... cleus accumbens shell in rats.
@en
P2093
Steven R Goldberg
P2860
P304
P356
10.1111/J.1471-4159.2006.03880.X
P407
P577
2006-07-01T00:00:00Z