A rapid and transient peripheral inflammatory response precedes brain inflammation after experimental stroke. - Wikidata - wikimedia - TriplyDB

A rapid and transient peripheral inflammatory response precedes brain inflammation after experimental stroke.

A rapid and transient peripheral inflammatory response precedes brain inflammation after experimental stroke.

http://www.wikidata.org/entity/Q48494462

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Ischemia, Immunosuppression and Infection--Tackling the Predicaments of Post-Stroke Complications Revisiting the timing hypothesis: biomarkers that define the therapeutic window of estrogen for stroke Interleukin-6 mediates enhanced thrombus development in cerebral arterioles following a brief period of focal brain ischemia.Obesity and stroke: Can we translate from rodents to patients?Lack of macrophage migration inhibitory factor in mice does not affect hallmarks of the inflammatory/immune response during the first week after stroke.Autoimmune responses to brain following stroke.Cardiopulmonary arrest and resuscitation disrupts cholinergic anti-inflammatory processes: a role for cholinergic α7 nicotinic receptors.Role of chemokines in CNS health and pathology: a focus on the CCL2/CCR2 and CXCL8/CXCR2 networks.Effects of the sigma-1 receptor agonist 1-(3,4-dimethoxyphenethyl)-4-(3-phenylpropyl)-piperazine dihydro-chloride on inflammation after stroke Toll-like receptors 2 and 4 in ischemic stroke: outcome and therapeutic values Knockdown of IL-1β improves hypoxia-ischemia brain associated with IL-6 up-regulation in cell and animal models AIM2 and NLRC4 inflammasomes contribute with ASC to acute brain injury independently of NLRP3.Vitamin D deficiency exacerbates experimental stroke injury and dysregulates ischemia-induced inflammation in adult rats.Neuroprotection by inhibiting the c-Jun N-terminal kinase pathway after cerebral ischemia occurs independently of interleukin-6 and keratinocyte-derived chemokine (KC/CXCL1) secretion Delayed administration of interleukin-1 receptor antagonist reduces ischemic brain damage and inflammation in comorbid rats.Hurdles to clear before clinical translation of ischemic postconditioning against stroke.Magnetic resonance imaging reveals therapeutic effects of interferon-beta on cytokine-induced reactivation of rat model of multiple sclerosis Intranasal Insulin and Insulin-Like Growth Factor 1 as Neuroprotectants in Acute Ischemic Stroke.Repeated, Intermittent Social Defeat across the Entire Juvenile Period Resulted in Behavioral, Physiological, Hormonal, Immunological, and Neurochemical Alterations in Young Adult Male Golden Hamsters.Fumarate modulates the immune/inflammatory response and rescues nerve cells and neurological function after stroke in rats.The acute-phase protein PTX3 is an essential mediator of glial scar formation and resolution of brain edema after ischemic injury.The complexity of atrial fibrillation newly diagnosed after ischemic stroke and transient ischemic attack: advances and uncertainties Inflammatory cytokines in experimental and human stroke.Systemic inflammation as a therapeutic target in acute ischemic stroke.Microglia and Monocyte-Derived Macrophages in Stroke Inflammation and Stroke: An Overview Rapid brain penetration of interleukin-1 receptor antagonist in rat cerebral ischaemia: pharmacokinetics, distribution, protection.Interleukin-1 mediates neuroinflammatory changes associated with diet-induced atherosclerosis.Surgical manipulation compromises leukocyte mobilization responses and inflammation after experimental cerebral ischemia in mice Experimental stroke-induced changes in the bone marrow reveal complex regulation of leukocyte responses.NLRP3-Inflammasome Activating DAMPs Stimulate an Inflammatory Response in Glia in the Absence of Priming Which Contributes to Brain Inflammation after Injury.Prolonged diet-induced obesity in mice modifies the inflammatory response and leads to worse outcome after stroke.Chronic systemic infection exacerbates ischemic brain damage via a CCL5 (regulated on activation, normal T-cell expressed and secreted)-mediated proinflammatory response in mice.Adipose tissue and metabolic and inflammatory responses to stroke are altered in obese mice.Acute high-fat feeding leads to disruptions in glucose homeostasis and worsens stroke outcome.Hepatic acute phase response protects the brain from focal inflammation during postnatal window of susceptibility.Ethanol Iris tenuifolia extract reduces brain damage in a mouse model of cerebral ischaemia.Piperine suppresses cerebral ischemia-reperfusion-induced inflammation through the repression of COX-2, NOS-2, and NF-κB in middle cerebral artery occlusion rat model.MicroRNA-124-loaded nanoparticles increase survival and neuronal differentiation of neural stem cells in vitro but do not contribute to stroke outcome in vivo.The role of dopaminergic immune cell signalling in poststroke inflammation.

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P2860

A rapid and transient peripheral inflammatory response precedes brain inflammation after experimental stroke.

http://www.wikidata.org/entity/Q48494462

description

2009 nî lūn-bûn

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2009年の論文

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2009年学术文章

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2009年学术文章

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2009年学术文章

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2009年学术文章

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2009年学术文章

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2009年学术文章

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2009年學術文章

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2009年學術文章

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name

A rapid and transient peripher ...... ion after experimental stroke.

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A rapid and transient peripher ...... ion after experimental stroke.

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type

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A rapid and transient peripher ...... ion after experimental stroke.

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A rapid and transient peripher ...... ion after experimental stroke.

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A rapid and transient peripher ...... ion after experimental stroke.

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A rapid and transient peripher ...... ion after experimental stroke.

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Journal of Cerebral Blood Flow & Metabolism

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A rapid and transient peripher ...... ion after experimental stroke.

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Adám Dénes

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Gavin Bennett

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Katie Z Chapman

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Nancy J Rothwell

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Verity Q Dale

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P2860

Peak plasma interleukin-6 and other peripheral markers of inflammation in the first week of ischaemic stroke correlate with brain infarct volume, stroke severity and long-term outcome

Inflammation and infection in clinical stroke

Differential effects of interleukin-1 alpha and beta on interleukin-6 and chemokine synthesis in neurones

Ratio of local to systemic chemokine concentrations regulates neutrophil recruitment

Central nervous system injury triggers hepatic CC and CXC chemokine expression that is associated with leukocyte mobilization and recruitment to both the central nervous system and the liver

Human cellular inflammation in the pathology of acute cerebral ischaemia

The interleukin-8 (IL-8/CXCL8) receptor inhibitor reparixin improves neurological deficits and reduces long-term inflammation in permanent and transient cerebral ischemia in rats.

Ambivalent aspects of interleukin-6 in cerebral ischemia: inflammatory versus neurotrophic aspects.

Monocyte function and plasma levels of interleukin-8 in acute ischemic stroke.

Systemic inflammatory stimulus potentiates the acute phase and CXC chemokine responses to experimental stroke and exacerbates brain damage via interleukin-1- and neutrophil-dependent mechanisms.

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1764-1768

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10.1038/JCBFM.2009.113

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19654587

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