Influence of complete ischemia on glycolytic metabolites, citric acid cycle intermediates, and associated amino acids in the rat cerebral cortex.
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Circulating anions usually associated with the Krebs cycle in patients with metabolic acidosisGPR91: expanding the frontiers of Krebs cycle intermediatesComplement component c1q mediates mitochondria-driven oxidative stress in neonatal hypoxic-ischemic brain injury.Calcium and mitochondrial reactive oxygen species generation: how to read the facts.Succinate causes pathological cardiomyocyte hypertrophy through GPR91 activation.Inhibitors of ROS production by the ubiquinone-binding site of mitochondrial complex I identified by chemical screeningIsoflurane anesthesia initiated at the onset of reperfusion attenuates oxidative and hypoxic-ischemic brain injuryThe oxygen free radicals originating from mitochondrial complex I contribute to oxidative brain injury following hypoxia-ischemia in neonatal mice.Detection of 31P nuclear magnetic resonance signals in brain by in vivo and freeze-trapped assays.AIF, reactive oxygen species, and neurodegeneration: a "complex" problemMetabolomic profiling reveals a role for caspase-2 in lipoapoptosisInhibitors of succinate: quinone reductase/Complex II regulate production of mitochondrial reactive oxygen species and protect normal cells from ischemic damage but induce specific cancer cell death.The role of HIFs in ischemia-reperfusion injury.Oxidative metabolism of the cerebral cortex of the rat in severe insulin-induced hypoglycaemia.The effect of hyperthermia upon oxygen consumption and upon organic phosphates, glycolytic metabolites, citric and cycle intermediates and associated amino acids in rat cerebral cortes.Cerebral metabolic changes during prolonged epileptic seizures in rats.Oxidized and reduced glutathione in the rat brain under normoxic and hypoxic conditions.Differential susceptibility of mitochondrial complex II to inhibition by oxaloacetate in brain and heart.Effect of vitamin B12 deficiency on energy-rich phosphates, glycolytic and citric acid cycle metabolites and associated amino acids in rat cerebral cortex.The importance of the measurement of ATP depletion and subsequent cell damage with an estimate of size and nature of the market for a practicable method: a review designed for technology transfer.Kinetic Modeling of the Mitochondrial Energy Metabolism of Neuronal Cells: The Impact of Reduced α-Ketoglutarate Dehydrogenase Activities on ATP Production and Generation of Reactive Oxygen Species.Blood-brain amino acid transport and content during anoxia and reoxygenation.Hypoxic-ischemic injury in the developing brain: the role of reactive oxygen species originating in mitochondria.Pyruvate carboxylation as an anaplerotic mechanism in the isolated perfused rat heart.The state of systemic circulation, collapsed or preserved defines the need for hyperoxic or normoxic resuscitation in neonatal mice with hypoxia-ischemia.Substrate-dependent modulation of oxidative phosphorylation in isolated mitochondria following in vitro hypoxia and reoxygenation injury.Cerebral dicarboxylate transport and metabolism studied with isotopically labelled fumarate, malate and malonate.Krebs cycle metabolites and preferential succinate oxidation following neonatal hypoxic-ischemic brain injury in mice.Reverse electron transfer results in a loss of flavin from mitochondrial complex I: Potential mechanism for brain ischemia reperfusion injury.Mapping of lactate and N-acetyl-L-aspartate predicts infarction during acute focal ischemia: in vivo 1H magnetic resonance spectroscopy in rats.Implications of enzyme deficiencies on mitochondrial energy metabolism and reactive oxygen species formation of neurons involved in rotenone-induced Parkinson's disease: a model-based analysis.Cerebral energy metabolism during induced mitochondrial dysfunction.Cerebral metabolic changes in profound, insulin-induced hypoglycemia, and in the recovery period following glucose administration.Restitution of cerebral energy state, as well as of glycolytic metabolites, citric acid cycle intermediates and associated amino acids after 30 minutes of complete ischemia in rats anaesthetized with nitrous oxide or phenobarbital.Changes in the cerebral metabolism induced by hyperventilation at different blood glucose levels.Carbohydrate and amino acid metabolism in rat cerebral cortex in moderate and extreme hypercapnia.Ammonia intoxication: effects on cerebral cortex and spinal cord.Focal and perifocal changes in tissue energy state during middle cerebral artery occlusion in normo- and hyperglycemic rats.Cerebral extracellular calcium activity in severe hypoglycemia: relation to extracellular potassium and energy state.Regional assessment of energy-producing metabolism following prolonged complete ischemia of cat brain.
P2860
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P2860
Influence of complete ischemia on glycolytic metabolites, citric acid cycle intermediates, and associated amino acids in the rat cerebral cortex.
description
1974 nî lūn-bûn
@nan
1974年の論文
@ja
1974年学术文章
@wuu
1974年学术文章
@zh
1974年学术文章
@zh-cn
1974年学术文章
@zh-hans
1974年学术文章
@zh-my
1974年学术文章
@zh-sg
1974年學術文章
@yue
1974年學術文章
@zh-hant
name
Influence of complete ischemia ...... ds in the rat cerebral cortex.
@en
Influence of complete ischemia ...... ds in the rat cerebral cortex.
@nl
type
label
Influence of complete ischemia ...... ds in the rat cerebral cortex.
@en
Influence of complete ischemia ...... ds in the rat cerebral cortex.
@nl
prefLabel
Influence of complete ischemia ...... ds in the rat cerebral cortex.
@en
Influence of complete ischemia ...... ds in the rat cerebral cortex.
@nl
P2093
P1433
P1476
Influence of complete ischemia ...... ds in the rat cerebral cortex.
@en
P2093
Folbergrová J
Ljunggren B
P304
P356
10.1016/0006-8993(74)90690-8
P407
P577
1974-11-01T00:00:00Z