Neuroprotection by mefenamic acid against D-serine: involvement of oxidative stress, inflammation and apoptosis.

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Radioprotective effect of mefenamic acid against radiation-induced genotoxicity in human lymphocytes.Mefenamic acid decreases inflammation but not joint lesions in experimental osteoarthritis.Putative Inflammatory Sensitive Mechanisms Underlying Risk or Resilience to Social Stress

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Neuroprotection by mefenamic acid against D-serine: involvement of oxidative stress, inflammation and apoptosis.

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2012 nî lūn-bûn

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2012年の論文

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2012年学术文章

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2012年学术文章

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2012年学术文章

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2012年学术文章

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2012年学术文章

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2012年学术文章

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2012年學術文章

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Neuroprotection by mefenamic a ...... s, inflammation and apoptosis.

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Neuroprotection by mefenamic a ...... s, inflammation and apoptosis.

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Item

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Neuroprotection by mefenamic a ...... s, inflammation and apoptosis.

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Neuroprotection by mefenamic a ...... s, inflammation and apoptosis.

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prefLabel

Neuroprotection by mefenamic a ...... s, inflammation and apoptosis.

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Neuroprotection by mefenamic a ...... s, inflammation and apoptosis.

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P1476

Neuroprotection by mefenamic a ...... ss, inflammation and apoptosis

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P2093

Ayfer Yalcin

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Guliz Armagan

http://www.w3.org/2001/XMLSchema#string

Lutfiye Kanit

http://www.w3.org/2001/XMLSchema#string

P2860

D-serine is an endogenous ligand for the glycine site of the N-methyl-D-aspartate receptor

Induction of serine racemase expression and D-serine release from microglia by amyloid beta-peptide

Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

Neuroprotective effects of non-steroidal anti-inflammatory drugs by direct scavenging of nitric oxide radicals

Spreading depression and focal brain ischemia induce cyclooxygenase-2 in cortical neurons through N-methyl-D-aspartic acid-receptors and phospholipase A2

Mitochondrial Ca2+ overload underlies Abeta oligomers neurotoxicity providing an unexpected mechanism of neuroprotection by NSAIDs

Apoptosis and necrosis: two distinct events induced, respectively, by mild and intense insults with N-methyl-D-aspartate or nitric oxide/superoxide in cortical cell cultures.

Excitotoxic cell death.

Glutamate-induced neuronal death: a succession of necrosis or apoptosis depending on mitochondrial function.

Excitatory amino acids as a final common pathway for neurologic disorders.

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scholarly article

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10.3109/10715762.2012.669836

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Ezgi Turunc

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2012-03-16T00:00:00Z

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22369458

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P921

D-Serine

mefenamic acid

Neuroprotection by mefenamic acid against D-serine: involvement of oxidative stress, inflammation and apoptosis.

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P2860

P2860

Neuroprotection by mefenamic acid against D-serine: involvement of oxidative stress, inflammation and apoptosis.

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P1476

P2093

P2860

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P698

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P921