Decrease in the numbers of dendritic cells and CD4+ T cells in cerebral perivascular spaces due to natalizumab.
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Exploring potential mechanisms of action of natalizumab in secondary progressive multiple sclerosisLeukocyte integrins: role in leukocyte recruitment and as therapeutic targets in inflammatory diseaseTreatment-related progressive multifocal leukoencephalopathy: current understanding and future stepsIntrathecal IgG synthesis: a resistant and valuable target for future multiple sclerosis treatmentsReview article: anti-adhesion therapies for inflammatory bowel diseaseVedolizumab for inflammatory bowel disease: Changing the game, or more of the same?Extracellular Vesicles in Multiple Sclerosis: What are They Telling Us?Role of the immunogenic and tolerogenic subsets of dendritic cells in multiple sclerosisMonoclonal antibodies and progressive multifocal leukoencephalopathyPlasma membrane profiling defines an expanded class of cell surface proteins selectively targeted for degradation by HCMV US2 in cooperation with UL141Long-term decrease in VLA-4 expression and functional impairment of dendritic cells during natalizumab therapy in patients with multiple sclerosisLymphocyte subsets show different response patterns to in vivo bound natalizumab--a flow cytometric study on patients with multiple sclerosisFunctional energetics of CD4+-cellular immunity in monoclonal antibody-associated progressive multifocal leukoencephalopathy in autoimmune disordersAlpha4beta1 integrin mediates the recruitment of immature dendritic cells across the blood-brain barrier during experimental autoimmune encephalomyelitis.Natalizumab: a review of its use in the management of relapsing-remitting multiple sclerosis.Rituximab therapy reduces organ-specific T cell responses and ameliorates experimental autoimmune encephalomyelitis.The Cache County Study on Memory in Aging: factors affecting risk of Alzheimer's disease and its progression after onsetEmerging viral infections of the central nervous system: part 2Effect of natalizumab treatment on circulating plasmacytoid dendritic cells: a cross-sectional observational study in patients with multiple sclerosis.Using genetics to enable studies on the prevention of Alzheimer's diseaseEstimating and disclosing the risk of developing Alzheimer's disease: challenges, controversies and future directionsPre-existing T- and B-cell defects in one progressive multifocal leukoencephalopathy patient.Targeting leukocyte migration and adhesion in Crohn's disease and ulcerative colitis.Natalizumab in the treatment of multiple sclerosisSafety of monoclonal antibodies for the treatment of multiple sclerosis.Immunological Markers for PML Prediction in MS Patients Treated with NatalizumabIntegrin targeted therapeutics.Peripherally triggered and GSK-3β-driven brain inflammation differentially skew adult hippocampal neurogenesis, behavioral pattern separation and microglial activation in response to ibuprofen.CD4 cell response to interval therapy with natalizumab.Th17 lymphocytes traffic to the central nervous system independently of α4 integrin expression during EAEAntibody to α4 integrin suppresses natural killer cells infiltration in central nervous system in experimental autoimmune encephalomyelitisH(1)R expression by CD11B(+) cells is not required for susceptibility to experimental allergic encephalomyelitisInflammatory Eicosanoids Increase Amyloid Precursor Protein Expression via Activation of Multiple Neuronal Receptors.Cellular sources of dysregulated cytokines in relapsing-remitting multiple sclerosis.Cutting Edge: CD99 Is a Novel Therapeutic Target for Control of T Cell-Mediated Central Nervous System Autoimmune Disease.Innate-adaptive crosstalk: how dendritic cells shape immune responses in the CNS.Inflammation and clinical presentation in neurodegenerative disease: a volatile relationship.Tanshinone IIA attenuates experimental autoimmune encephalomyelitis in rats.An update on the efficacy of non-steroidal anti-inflammatory drugs in Alzheimer's disease.Long-term safety and efficacy of natalizumab in relapsing-remitting multiple sclerosis: impact on quality of life.
P2860
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P2860
Decrease in the numbers of dendritic cells and CD4+ T cells in cerebral perivascular spaces due to natalizumab.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年学术文章
@wuu
2008年学术文章
@zh-cn
2008年学术文章
@zh-hans
2008年学术文章
@zh-my
2008年学术文章
@zh-sg
2008年學術文章
@yue
2008年學術文章
@zh
2008年學術文章
@zh-hant
name
Decrease in the numbers of den ...... lar spaces due to natalizumab.
@en
Decrease in the numbers of den ...... lar spaces due to natalizumab.
@nl
type
label
Decrease in the numbers of den ...... lar spaces due to natalizumab.
@en
Decrease in the numbers of den ...... lar spaces due to natalizumab.
@nl
prefLabel
Decrease in the numbers of den ...... lar spaces due to natalizumab.
@en
Decrease in the numbers of den ...... lar spaces due to natalizumab.
@nl
P2093
P1433
P1476
Decrease in the numbers of den ...... lar spaces due to natalizumab.
@en
P2093
B K Kleinschmidt-DeMasters
Bernhard Hemmer
Elliot M Frohman
Maria del Pilar Martin
Martin S Weber
Michael K Racke
Nitin J Karandikar
Petra D Cravens
Ryan Winger
Scott S Zamvil
P304
P356
10.1001/ARCHNEUR.65.12.NOC80051
P50
P577
2008-10-13T00:00:00Z