Oral insulin stimulates intestinal epithelial cell turnover following massive small bowel resection in a rat and a cell culture model.
about
Insulin-like growth factor 2 and its enterocyte receptor are not required for adaptation in response to massive small bowel resection.Deletion of intestinal epithelial insulin receptor attenuates high-fat diet-induced elevations in cholesterol and stem, enteroendocrine, and Paneth cell mRNAs.On the horizon: trophic peptide growth factors as therapy for neonatal short bowel syndrome.MEK/ERK pathway activation by insulin receptor isoform alteration is associated with the abnormal proliferation and differentiation of intestinal epithelial cells in diabetic mice.A Novel Role of Spred2 in the Colonic Epithelial Cell Homeostasis and Inflammation.Epithelial IGF1R is dispensable for IGF2 mediated enhanced intestinal adaptation in retinoblastoma-deficient mice.
P2860
Oral insulin stimulates intestinal epithelial cell turnover following massive small bowel resection in a rat and a cell culture model.
description
2012 nî lūn-bûn
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2012年の論文
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2012年学术文章
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2012年学术文章
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2012年学术文章
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2012年学术文章
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2012年学术文章
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2012年學術文章
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2012年學術文章
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name
Oral insulin stimulates intest ...... rat and a cell culture model.
@en
Oral insulin stimulates intest ...... rat and a cell culture model.
@nl
type
label
Oral insulin stimulates intest ...... rat and a cell culture model.
@en
Oral insulin stimulates intest ...... rat and a cell culture model.
@nl
prefLabel
Oral insulin stimulates intest ...... rat and a cell culture model.
@en
Oral insulin stimulates intest ...... rat and a cell culture model.
@nl
P2093
P2860
P1476
Oral insulin stimulates intest ...... rat and a cell culture model.
@en
P2093
Arnold G Coran
Igor Sukhotnik
Jorge G Mogilner
Naim Shehadeh
Raanan Shamir
Shani Ben Lulu
P2860
P2888
P304
P356
10.1007/S00383-011-2991-4
P577
2012-02-01T00:00:00Z