TNF and increased intracellular iron alter macrophage polarization to a detrimental M1 phenotype in the injured spinal cord.
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Regenerative Capacity of Macrophages for RemyelinationThe Reactive Oxygen Species in Macrophage Polarization: Reflecting Its Dual Role in Progression and Treatment of Human DiseasesMacrophage polarization in nerve injury: do Schwann cells play a role?Myelin damage and repair in pathologic CNS: challenges and prospectsMechanisms and potential therapeutic applications of microglial activation after brain injuryNeurotrauma and inflammation: CNS and PNS responsesAzithromycin drives alternative macrophage activation and improves recovery and tissue sparing in contusion spinal cord injuryIntravenous multipotent adult progenitor cell treatment decreases inflammation leading to functional recovery following spinal cord injury.Intercellular cross-talk in intracerebral hemorrhageImmune Surveillance of the CNS following Infection and InjuryRevisiting the Mechanisms of CNS Immune Privilege.Dealing with Danger in the CNS: The Response of the Immune System to Injury.Therapeutic inhibition of soluble brain TNF promotes remyelination by increasing myelin phagocytosis by microgliaBeneficial effects of early hemostasis on spinal cord injury in the ratLow-level laser facilitates alternatively activated macrophage/microglia polarization and promotes functional recovery after crush spinal cord injury in rats.Targeting iron metabolism in drug discovery and deliveryRelationship between brainstem neurodegeneration and clinical impairment in traumatic spinal cord injuryMicroglial Activation in the Pathogenesis of Huntington's Disease.Computational modeling of cytokine signaling in microglia.Macrophages in Tissue Repair, Regeneration, and Fibrosis.Alternative activation-skewed microglia/macrophages promote hematoma resolution in experimental intracerebral hemorrhage.Iron homeostasis: a new job for macrophages in adipose tissue?Macrophages in spinal cord injury: phenotypic and functional change from exposure to myelin debris.Neuroinflammation triggered by β-glucan/dectin-1 signaling enables CNS axon regeneration.Toll-Like Receptors and Dectin-1, a C-Type Lectin Receptor, Trigger Divergent Functions in CNS Macrophages.Expressing Constitutively Active Rheb in Adult Neurons after a Complete Spinal Cord Injury Enhances Axonal Regeneration beyond a Chondroitinase-Treated Glial Scar.Loss of MyD88 alters neuroinflammatory response and attenuates early Purkinje cell loss in a spinocerebellar ataxia type 6 mouse modelA Novel Strategy for TNF-Alpha Production by 2-APB Induced Downregulated SOCE and Upregulated HSP70 in O. tsutsugamushi-Infected Human MacrophagesMicroglia/Macrophage Polarization After Experimental Intracerebral Hemorrhage.Targeting acute inflammation to complement spinal cord repair.Microglia in the TBI brain: The good, the bad, and the dysregulated.Pioglitazone regulates myelin phagocytosis and multiple sclerosis monocytes.Cyclic AMP is a key regulator of M1 to M2a phenotypic conversion of microglia in the presence of Th2 cytokinesRole of Erythrocyte CD47 in Intracerebral Hematoma ClearanceReactive astrocytes undergo M1 microglia/macrohpages-induced necroptosis in spinal cord injury.Sustained TNF production by central nervous system infiltrating macrophages promotes progressive autoimmune encephalomyelitis.Adipose Natural Killer Cells Regulate Adipose Tissue Macrophages to Promote Insulin Resistance in ObesityAlternatively activated brain-infiltrating macrophages facilitate recovery from collagenase-induced intracerebral hemorrhageRescuing macrophage normal function in spinal cord injury with embryonic stem cell conditioned mediaTLR4 Deficiency Impairs Oligodendrocyte Formation in the Injured Spinal Cord.
P2860
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P2860
TNF and increased intracellular iron alter macrophage polarization to a detrimental M1 phenotype in the injured spinal cord.
description
2014 nî lūn-bûn
@nan
2014年の論文
@ja
2014年学术文章
@wuu
2014年学术文章
@zh
2014年学术文章
@zh-cn
2014年学术文章
@zh-hans
2014年学术文章
@zh-my
2014年学术文章
@zh-sg
2014年學術文章
@yue
2014年學術文章
@zh-hant
name
TNF and increased intracellula ...... pe in the injured spinal cord.
@en
TNF and increased intracellula ...... pe in the injured spinal cord.
@nl
type
label
TNF and increased intracellula ...... pe in the injured spinal cord.
@en
TNF and increased intracellula ...... pe in the injured spinal cord.
@nl
prefLabel
TNF and increased intracellula ...... pe in the injured spinal cord.
@en
TNF and increased intracellula ...... pe in the injured spinal cord.
@nl
P2093
P1433
P1476
TNF and increased intracellula ...... pe in the injured spinal cord.
@en
P2093
Andrew D Greenhalgh
Antje Kroner
Juan G Zarruk
Rosmarini Passos Dos Santos
Samuel David
P304
P356
10.1016/J.NEURON.2014.07.027
P407
P577
2014-08-14T00:00:00Z