Expression of Na+/I- symporter and Pendred syndrome genes in trophoblast cells.
about
Loss of KCNJ10 protein expression abolishes endocochlear potential and causes deafness in Pendred syndrome mouse model.Genetic factors that might lead to different responses in individuals exposed to perchlorate.Transcriptional regulation of the pendrin gene.Localization and functional studies of pendrin in the mouse inner ear provide insight about the etiology of deafness in pendred syndrome.Loss of cochlear HCO3- secretion causes deafness via endolymphatic acidification and inhibition of Ca2+ reabsorption in a Pendred syndrome mouse model.Expression of the Na+/I- symporter (NIS) is markedly decreased or absent in gastric cancer and intestinal metaplastic mucosa of Barrett esophagusSodium iodide symporter (NIS) in extrathyroidal malignancies: focus on breast and urological cancerIn utero exposure to iodine-131 from Chernobyl fallout and anthropometric characteristics in adolescence.Decrease in thyroid adenoma associated (THADA) expression is a marker of dedifferentiation of thyroid tissue.Methylation of the human pendrin promoter.Origins and frequencies of SLC26A4 (PDS) mutations in east and south Asians: global implications for the epidemiology of deafnessMouse models for pendrin-associated loss of cochlear and vestibular functionLack of pendrin HCO3- transport elevates vestibular endolymphatic [Ca2+] by inhibition of acid-sensitive TRPV5 and TRPV6 channels.A screening study of thyroid cancer and other thyroid diseases among individuals exposed in utero to iodine-131 from Chernobyl fallout.The Na+/I- symporter (NIS): mechanism and medical impact.Regulation of iodide uptake in placental primary cultures.Molecular and functional characterization of human pendrin and its allelic variants.Pendrin function in airway epithelia.Pendrin is a novel in vivo downstream target gene of the TTF-1/Nkx-2.1 homeodomain transcription factor in differentiated thyroid cells.Regulation of pendrin by pH: dependence on glycosylation.Na+/I- symporter and type 3 iodothyronine deiodinase gene expression in amniotic membrane and placenta and its relationship to maternal thyroid hormones.The ESF meeting on "The proteomics, epigenetics and pharmacogenetics of pendrin".The sodium-iodide symporter expression in placental tissue at different gestational age: an immunohistochemical study.Most bicarbonate secretion by Calu-3 cells is mediated by CFTR and independent of pendrin.Is placental iodine content related to dietary iodine intake?
P2860
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P2860
Expression of Na+/I- symporter and Pendred syndrome genes in trophoblast cells.
description
2000 nî lūn-bûn
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2000年の論文
@ja
2000年学术文章
@wuu
2000年学术文章
@zh
2000年学术文章
@zh-cn
2000年学术文章
@zh-hans
2000年学术文章
@zh-my
2000年学术文章
@zh-sg
2000年學術文章
@yue
2000年學術文章
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name
Expression of Na+/I- symporter and Pendred syndrome genes in trophoblast cells.
@en
Expression of Na+/I- symporter and Pendred syndrome genes in trophoblast cells.
@nl
type
label
Expression of Na+/I- symporter and Pendred syndrome genes in trophoblast cells.
@en
Expression of Na+/I- symporter and Pendred syndrome genes in trophoblast cells.
@nl
prefLabel
Expression of Na+/I- symporter and Pendred syndrome genes in trophoblast cells.
@en
Expression of Na+/I- symporter and Pendred syndrome genes in trophoblast cells.
@nl
P2093
P356
P1476
Expression of Na+/I- symporter and Pendred syndrome genes in trophoblast cells.
@en
P2093
Evain-Brion D
Schlumberger M
P304
P356
10.1210/JC.85.11.4367
P407
P577
2000-11-01T00:00:00Z