Regulatory interactions of N1303K-CFTR and ENaC in Xenopus oocytes: evidence that chloride transport is not necessary for inhibition of ENaC.
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Functional interaction between CFTR and the sodium-phosphate co-transport type 2a in Xenopus laevis oocytesDeficiency of the two-pore-domain potassium channel TREK-1 promotes hyperoxia-induced lung injury.Regulation of endogenous ENaC functional expression by CFTR and ΔF508-CFTR in airway epithelial cells.Liquid movement across the surface epithelium of large airwaysRegulation of the epithelial Na+ channel and airway surface liquid volume by serine proteases.Does epithelial sodium channel hyperactivity contribute to cystic fibrosis lung disease?Repairing the basic defect in cystic fibrosis - one approach is not enough.Hyperoxia treatment of TREK-1/TREK-2/TRAAK-deficient mice is associated with a reduction in surfactant proteins.
P2860
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P2860
Regulatory interactions of N1303K-CFTR and ENaC in Xenopus oocytes: evidence that chloride transport is not necessary for inhibition of ENaC.
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2006 nî lūn-bûn
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2006年の論文
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2006年学术文章
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2006年学术文章
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name
Regulatory interactions of N13 ...... essary for inhibition of ENaC.
@en
Regulatory interactions of N13 ...... essary for inhibition of ENaC.
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type
label
Regulatory interactions of N13 ...... essary for inhibition of ENaC.
@en
Regulatory interactions of N13 ...... essary for inhibition of ENaC.
@nl
prefLabel
Regulatory interactions of N13 ...... essary for inhibition of ENaC.
@en
Regulatory interactions of N13 ...... essary for inhibition of ENaC.
@nl
P2093
P2860
P1476
Regulatory interactions of N13 ...... essary for inhibition of ENaC.
@en
P2093
Amal Robay
Laurence Suaud
Marcelo D Carattino
Thomas R Kleyman
Wusheng Yan
P2860
P304
P356
10.1152/AJPCELL.00064.2006
P577
2006-12-20T00:00:00Z