Optimal AraC-Cytotoxicity to AML Cells Requires ERK5 Activity.

about

ATM meets ERK5.

P2860

Q41839972-B7603002-9AB7-4897-9DE5-78E5371BCC9E

P2860

Optimal AraC-Cytotoxicity to AML Cells Requires ERK5 Activity.

Item

http://www.wikidata.org/entity/Q51283531

description

2016 nî lūn-bûn

@nan

2016年の論文

@ja

2016年学术文章

@wuu

2016年学术文章

@zh

2016年学术文章

@zh-cn

2016年学术文章

@zh-hans

2016年学术文章

@zh-my

2016年学术文章

@zh-sg

2016年學術文章

@yue

2016年學術文章

@zh-hant

name

Optimal AraC-Cytotoxicity to AML Cells Requires ERK5 Activity.

@en

Optimal AraC-Cytotoxicity to AML Cells Requires ERK5 Activity.

@nl

type

Item

label

Optimal AraC-Cytotoxicity to AML Cells Requires ERK5 Activity.

@en

Optimal AraC-Cytotoxicity to AML Cells Requires ERK5 Activity.

@nl

prefLabel

Optimal AraC-Cytotoxicity to AML Cells Requires ERK5 Activity.

@en

Optimal AraC-Cytotoxicity to AML Cells Requires ERK5 Activity.

@nl

P1476

Optimal AraC-Cytotoxicity to AML Cells Requires ERK5 Activity.

@en

P2093

George P Studzinski

http://www.w3.org/2001/XMLSchema#string

Ruifang Zheng

http://www.w3.org/2001/XMLSchema#string

P2860

Erk5 null mice display multiple extraembryonic vascular and embryonic cardiovascular defects

The Bcl-2 apoptotic switch in cancer development and therapy

Activation and function of the MAPKs and their substrates, the MAPK-activated protein kinases

The comet assay for DNA damage and repair: principles, applications, and limitations

Bmk1/Erk5 is required for cell proliferation induced by epidermal growth factor

Canonical and kinase activity-independent mechanisms for extracellular signal-regulated kinase 5 (ERK5) nuclear translocation require dissociation of Hsp90 from the ERK5-Cdc37 complex

ERK and p38 MAPK-activated protein kinases: a family of protein kinases with diverse biological functions

Raf-1 signaling is required for the later stages of 1,25-dihydroxyvitamin D3-induced differentiation of HL60 cells but is not mediated by the MEK/ERK module

DNA damage-induced cell cycle checkpoints and DNA strand break repair in development and tumorigenesis.

Inhibition of Cot1/Tlp2 oncogene in AML cells reduces ERK5 activation and up-regulates p27Kip1 concomitant with enhancement of differentiation and cell cycle arrest induced by silibinin and 1,25-dihydroxyvitamin D(3).

P304

1583-1589

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1002/JCB.25820

http://www.w3.org/2001/XMLSchema#string

P433

http://www.w3.org/2001/XMLSchema#string

P478

118

http://www.w3.org/2001/XMLSchema#string

P577

2016-12-06T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

27922189

http://www.w3.org/2001/XMLSchema#string

Optimal AraC-Cytotoxicity to AML Cells Requires ERK5 Activity.

about

P2860

P2860

Optimal AraC-Cytotoxicity to AML Cells Requires ERK5 Activity.

description

name

type

label

prefLabel

P1433

P1476

P2093

P2860

P304

P31

P356

P433

P478

P577

P698

P356

P1433

P1476

P2093

P2860

P304

P31

P356

P433

P478

P577

P698