about
IL-22BP is regulated by the inflammasome and modulates tumorigenesis in the intestineNLRP10 is a NOD-like receptor essential to initiate adaptive immunity by dendritic cellsIL-22 deficiency alters colonic microbiota to be transmissible and colitogenic.Bacterial flagellin stimulates Toll-like receptor 5-dependent defense against vancomycin-resistant Enterococcus infection.Listeria monocytogenes phosphatidylinositol-specific phospholipase C has evolved for virulence by greatly reduced activity on GPI anchorsCharacterization of Listeria monocytogenes expressing anthrolysin O and phosphatidylinositol-specific phospholipase C from Bacillus anthracis.RasGRP1 regulates antigen-induced developmental programming by naive CD8 T cells.IL-22 signaling contributes to West Nile encephalitis pathogenesis.Memory/effector (CD45RB(lo)) CD4 T cells are controlled directly by IL-10 and cause IL-22-dependent intestinal pathology.Interleukin 23 production by intestinal CD103(+)CD11b(+) dendritic cells in response to bacterial flagellin enhances mucosal innate immune defense.Defective Intestinal Mucin-Type O-Glycosylation Causes Spontaneous Colitis-Associated Cancer in MiceSugar-coated regulation of T cells.Interleukin-22 but not interleukin-17 provides protection to hepatocytes during acute liver inflammationUnraveling the genetics of autoimmunityExcessive Th1 responses due to the absence of TGF-β signaling cause autoimmune diabetes and dysregulated Treg cell homeostasisInnate and adaptive immune responses to Listeria monocytogenes: a short overview.Oxygen Levels and Immunological StudiesRecent advances in IL-22 biology.Changes in availability of oxygen accentuate differences in capsular polysaccharide expression by phenotypic variants and clinical isolates of Streptococcus pneumoniae.Innate and adaptive interleukin-22 protects mice from inflammatory bowel diseaseNonsecreted bacterial proteins induce recall CD8 T cell responses but do not serve as protective antigens.Bacillus anthracis lethal toxin negatively modulates ILC3 function through perturbation of IL-23-mediated MAPK signaling.Transcription Factor HIF-1α Controls Expression of the Cytokine IL-22 in CD4 T Cells.Listeria monocytogenes virulence proteins induce surface expression of Fas ligand on T lymphocytes.Hypoxic modulation of hepatocyte responses to the cytokine interleukin-22.Activation of antigen-specific CD8 T cells results in minimal killing of bystander bacteria.The dual nature of TH17 cells: shifting the focus to function5'-Methoxyhydnocarpin-D and pheophorbide A: Berberis species components that potentiate berberine growth inhibition of resistant Staphylococcus aureusCutting edge: CD4 and CD8 T cells are intrinsically different in their proliferative responsesCorrigendum: NLRP10 is a NOD-like receptor essential to initiate adaptive immunity by dendritic cellsGlucocorticoids Inhibit Group 3 Innate Lymphocyte IL-22 ProductionIL-22: There Is a Gap in Our Knowledge
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P50
description
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Lauren A Zenewicz
@ast
Lauren A Zenewicz
@en
Lauren A Zenewicz
@es
Lauren A Zenewicz
@nl
Lauren A Zenewicz
@sl
type
label
Lauren A Zenewicz
@ast
Lauren A Zenewicz
@en
Lauren A Zenewicz
@es
Lauren A Zenewicz
@nl
Lauren A Zenewicz
@sl
prefLabel
Lauren A Zenewicz
@ast
Lauren A Zenewicz
@en
Lauren A Zenewicz
@es
Lauren A Zenewicz
@nl
Lauren A Zenewicz
@sl
P106
P31
P496
0000-0002-8787-5955