Block of quantal end-plate currents of mouse muscle by physostigmine and procaine.
about
Presynaptic M(2) muscarinic receptors are involved in controlling the kinetics of ACh release at the frog neuromuscular junctionEvaluation of the number of agonist molecules needed to activate a ligand-gated channel from the current rising phase.Desensitization contributes to the synaptic response of gain-of-function mutants of the muscle nicotinic receptor.α-Conotoxin M1 (CTx) blocks αδ binding sites of adult nicotinic receptors while ACh binding at αε sites elicits only small and short quantal synaptic currents.
P2860
Block of quantal end-plate currents of mouse muscle by physostigmine and procaine.
description
1999 nî lūn-bûn
@nan
1999年の論文
@ja
1999年学术文章
@wuu
1999年学术文章
@zh
1999年学术文章
@zh-cn
1999年学术文章
@zh-hans
1999年学术文章
@zh-my
1999年学术文章
@zh-sg
1999年學術文章
@yue
1999年學術文章
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name
Block of quantal end-plate currents of mouse muscle by physostigmine and procaine.
@en
Block of quantal end-plate currents of mouse muscle by physostigmine and procaine.
@nl
type
label
Block of quantal end-plate currents of mouse muscle by physostigmine and procaine.
@en
Block of quantal end-plate currents of mouse muscle by physostigmine and procaine.
@nl
prefLabel
Block of quantal end-plate currents of mouse muscle by physostigmine and procaine.
@en
Block of quantal end-plate currents of mouse muscle by physostigmine and procaine.
@nl
P2093
P2860
P1476
Block of quantal end-plate currents of mouse muscle by physostigmine and procaine.
@en
P2093
P2860
P304
P356
10.1152/JN.1999.81.5.2386
P407
P577
1999-05-01T00:00:00Z