Embryonic overexpression of receptors for advanced glycation end-products by alveolar epithelium induces an imbalance between proliferation and apoptosis.
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Plausible Roles for RAGE in Conditions Exacerbated by Direct and Indirect (Secondhand) Smoke Exposure.Antenatal exposure of maternal secondhand smoke (SHS) increases fetal lung expression of RAGE and induces RAGE-mediated pulmonary inflammation.Associations between maternal and infant morbidities and sRAGE within the first week of life in extremely preterm infants.The receptor for advanced glycation end products (RAGE) contributes to the progression of emphysema in miceThe Mouse-Specific Splice Variant mRAGE_v4 Encodes a Membrane-Bound RAGE That Is Resistant to Shedding and Does Not Contribute to the Production of Soluble RAGE.The Ser82 RAGE Variant Affects Lung Function and Serum RAGE in Smokers and sRAGE Production In Vitro.RAGE and tobacco smoke: insights into modeling chronic obstructive pulmonary disease.Effect of chronic hypoxia on RAGE and its soluble forms in lungs and plasma of mice.Molecular strategies to prevent, inhibit, and degrade advanced glycoxidation and advanced lipoxidation end products.Critical role of RAGE in lung physiology and tumorigenesis: a potential target of therapeutic intervention?Potential contribution of type I alveolar epithelial cells to chronic neonatal lung disease.Blockade of RAGE ameliorates elastase-induced emphysema development and progression via RAGE-DAMP signaling.Up-Regulation of Claudin-6 in the Distal Lung Impacts Secondhand Smoke-Induced InflammationConditional over-expression of RAGE by embryonic alveolar epithelium compromises the respiratory membrane and impairs endothelial cell differentiation.Conditionally induced RAGE expression by proximal airway epithelial cells in transgenic mice causes lung inflammation.Emphysema requires the receptor for advanced glycation end-products triggering on structural cells.Cardiomyocyte mitochondrial respiration is reduced by receptor for advanced glycation end-product signaling in a ceramide-dependent manner.Acute secondhand smoke-induced pulmonary inflammation is diminished in RAGE knockout mice.Conditional overexpression of receptors for advanced glycation end-products in the adult murine lung causes airspace enlargement and induces inflammation.Nitration and Glycation Turn Mature NGF into a Toxic Factor for Motor Neurons: A Role for p75NTR and RAGE Signaling in ALS.
P2860
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P2860
Embryonic overexpression of receptors for advanced glycation end-products by alveolar epithelium induces an imbalance between proliferation and apoptosis.
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2012 nî lūn-bûn
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2012年の論文
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2012年学术文章
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2012年学术文章
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2012年学术文章
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2012年学术文章
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2012年学术文章
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2012年學術文章
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name
Embryonic overexpression of re ...... n proliferation and apoptosis.
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Embryonic overexpression of re ...... n proliferation and apoptosis.
@nl
type
label
Embryonic overexpression of re ...... n proliferation and apoptosis.
@en
Embryonic overexpression of re ...... n proliferation and apoptosis.
@nl
prefLabel
Embryonic overexpression of re ...... n proliferation and apoptosis.
@en
Embryonic overexpression of re ...... n proliferation and apoptosis.
@nl
P2093
P2860
P356
P1476
Embryonic overexpression of re ...... en proliferation and apoptosis
@en
P2093
Adam B Robinson
Jason L Porter
Jeffrey A Stogsdill
Joshua M Hancock
Megan P Stogsdill
P2860
P356
10.1165/RCMB.2011-0385OC
P577
2012-02-16T00:00:00Z